e-learning
resources
Amsterdam 2015
Monday, 28.09.2015
New targets and modalities for the treatment of asthma and COPD
Login
Search all ERS
e-learning
resources
Disease Areas
Airways Diseases
Interstitial Lung Diseases
Respiratory Critical Care
Respiratory Infections
Paediatric Respiratory Diseases
Pulmonary Vascular Diseases
Sleep and Breathing Disorders
Thoracic Oncology
Events
International Congress
Courses
Webinars
Conferences
Research Seminars
Journal Clubs
Publications
Breathe
Monograph
ERJ
ERJ Open Research
ERR
European Lung White Book
Handbook Series
Guidelines
All ERS guidelines
e-learning
CME Online
Case reports
Short Videos
SpirXpert
Procedure Videos
CME tests
Reference Database of Respiratory Sounds
Radiology Image Challenge
Brief tobacco interventions
EU Projects
VALUE-Dx
ERN-LUNG
ECRAID
UNITE4TB
Disease Areas
Events
Publications
Guidelines
e-learning
EU Projects
Login
Search
Serelaxin is a potent inhibitor of airway contraction and enhances β-adrenoceptor-mediated relaxation
Jane Bourke (Clayton, Australia), Jane Bourke, Maggie Lam, Maria Jelinic, Laura Parry, Simon Royce, Chrishan Samuel
Source:
International Congress 2015 – New targets and modalities for the treatment of asthma and COPD
Session:
New targets and modalities for the treatment of asthma and COPD
Session type:
Poster Discussion
Number:
2121
Disease area:
Airway diseases
Rating:
You must
login
to grade this presentation.
Share or cite this content
Citations should be made in the following way:
Jane Bourke (Clayton, Australia), Jane Bourke, Maggie Lam, Maria Jelinic, Laura Parry, Simon Royce, Chrishan Samuel. Serelaxin is a potent inhibitor of airway contraction and enhances β-adrenoceptor-mediated relaxation. Eur Respir J 2015; 46: Suppl. 59, 2121
You must
login
to share this Presentation/Article on Twitter, Facebook, LinkedIn or by email.
Member's Comments
No comment yet.
You must
Login
to comment this presentation.
Related content which might interest you:
Late Breaking Abstract - Implications of treatable traits and treatment choices on exacerbation risk in moderate-severe asthma
Management of Severe Asthma in Pediatric Patients by an Interdisciplinary Team in a Public Hospital Setting.
Impact of Dexamethasone on pathogen profile of COVID-19 patients requiring intensive care: a multicentre retrospective study
Related content which might interest you:
The effect of adenosine on VEGF release from airway smooth muscle from subjects with asthma
Source: Annual Congress 2013 –Airway smooth muscle and cell biology
Year: 2013
Central role of AC6 in β2 agonist induced relaxation of human airway smooth muscle
Source: International Congress 2016 – Novel insights into the treatment of COPD
Year: 2016
The effect of JQ1/SGCBD01 upon c-Myc in human airway smooth muscle in asthma
Source: International Congress 2014 – New treatments for cough, asthma, COPD and ILDs
Year: 2014
The anti-proliferative effect of hydrogen sulfide upon human airway smooth muscle in COPD
Source: International Congress 2014 – New drugs for asthma, COPD and pulmonary fibrosis
Year: 2014
Involvement of the BK channels/G proteins processes in the synergistic effects between anticholinergic agents and beta
2
-adrenoceptor agonists in airway smooth muscle
Source: Annual Congress 2013 –Novel mechanisms for established drugs for asthma and COPD management
Year: 2013
Impaired inhibitory action of corticosteroids on chemokine expression induced by TNFalpha in airway smooth muscle (ASM) cells from patients with severe asthma
Source: Annual Congress 2013 –Airway smooth muscle and cell biology
Year: 2013
Endothelin-1-mediated contraction of mouse small airways is resistant to salbutamol, but sensitive to rosiglitazone
Source: Annual Congress 2013 –Preclinical models for the development of new drugs for respiratory diseases
Year: 2013
Distribution of increased airway smooth muscle thickness and airway inflammation in asthma
Source: Annual Congress 2013 –Airway smooth muscle and cell biology
Year: 2013
Compound A (CpdA) suppressed production of corticosteroid-resistant chemokines via GR-independent mechanisms in airway smooth muscle (ASM) cells
Source: Annual Congress 2013 –Preclinical models for the development of new drugs for respiratory diseases
Year: 2013
Prostacyclin IP receptor activation blocks hyperosmolar-induced bronchoconstriction in isolated human small airways
Source: International Congress 2016 – Airway pharmacology: drug targets and mode of action
Year: 2016
Compound A modulates steroid insensitive chemokines in airway smooth muscle cells via IRF-1 dependent and independent pathways
Source: International Congress 2015 – New targets and modalities for the treatment of asthma and COPD
Year: 2015
A functional role for WNT-5A in driving airway myocyte proliferation
Source: Annual Congress 2013 –Growth factors at the crossroads of acute and chronic inflammation
Year: 2013
Long-term effects of bronchial thermoplasty on airway smooth muscle and collagen deposition in severe asthma
Source: International Congress 2016 – Asthma: from basic mechanisms to novel therapeutic perspectives
Year: 2016
MNK-1 inhibition reduces proliferation and CXCL10 in airway smooth muscle cells
Source: Annual Congress 2013 –Airway smooth muscle and cell biology
Year: 2013
The critical role of bronchial smooth muscle remodeling in non-severe asthma
Source: International Congress 2015 – Airway remodelling: recent developments
Year: 2015
Eosinophil-induced airway smooth muscle cells remodeling is attenuated by blocking of RGD-binding integrins in asthma
Source: International Congress 2016 – New findings in mucosal immunology
Year: 2016
Potent bronchodilation and reduced stiffness by relaxant stimuli under dynamic conditions
Source: Eur Respir J 2009; 33: 844-851
Year: 2009
Targeting ASK1 in preventing airway smooth muscle growth: Implications for airway remodeling in COPD
Source: International Congress 2016 – New findings in mucosal immunology
Year: 2016
Relaxation of tracheal smooth muscle is impaired in innate airway hyperresponsiveness
Source: Eur Respir J 2009; 33: 417-424
Year: 2009
Exaggerated airway narrowing is related to increased airway smooth muscle mass in bronchial segments from subjects with a history of asthma
Source: Annual Congress 2013 –Airway smooth muscle and cell biology
Year: 2013
We use cookies on our website to give you the most relevant experience by remembering your preferences and repeat visits. By clicking "Accept", you consent to the use of the cookies.
Accept