e-learning
resources
Vienna 2009
Wednesday, 16.09.2009
What is new in molecular pathology and functional genomics of neoplastic and non-neoplastic lung disease?
Login
Search all ERS
e-learning
resources
Disease Areas
Airways Diseases
Interstitial Lung Diseases
Respiratory Critical Care
Respiratory Infections
Paediatric Respiratory Diseases
Pulmonary Vascular Diseases
Sleep and Breathing Disorders
Thoracic Oncology
Events
International Congress
Courses
Webinars
Conferences
Research Seminars
Journal Clubs
Publications
Breathe
Monograph
ERJ
ERJ Open Research
ERR
European Lung White Book
Handbook Series
Guidelines
All ERS guidelines
e-learning
CME Online
Case reports
Short Videos
SpirXpert
Procedure Videos
CME tests
Reference Database of Respiratory Sounds
Radiology Image Challenge
Brief tobacco interventions
EU Projects
VALUE-Dx
ERN-LUNG
ECRAID
UNITE4TB
Disease Areas
Events
Publications
Guidelines
e-learning
EU Projects
Login
Search
TLR-4 downregulation in COPD impairs cytokine release from monocytes and Th1-lymphocytes in response to bacterial endotoxin
J. Knobloch, K. Schild, D. Jungck, S. Chikosi, L. Njue, K. Urban, A. Koch (Cologne, Germany)
Source:
Annual Congress 2009 - What is new in molecular pathology and functional genomics of neoplastic and non-neoplastic lung disease?
Session:
What is new in molecular pathology and functional genomics of neoplastic and non-neoplastic lung disease?
Session type:
E-Communication Session
Number:
4770
Disease area:
Airway diseases
Rating:
You must
login
to grade this presentation.
Share or cite this content
Citations should be made in the following way:
J. Knobloch, K. Schild, D. Jungck, S. Chikosi, L. Njue, K. Urban, A. Koch (Cologne, Germany). TLR-4 downregulation in COPD impairs cytokine release from monocytes and Th1-lymphocytes in response to bacterial endotoxin. Eur Respir J 2009; 34: Suppl. 53, 4770
You must
login
to share this Presentation/Article on Twitter, Facebook, LinkedIn or by email.
Member's Comments
No comment yet.
You must
Login
to comment this presentation.
Related content which might interest you:
Panel discussion on ERS Statement – A core outcome set for clinical trials evaluating the management of COPD exacerbations
ERS statement: a core outcome set for clinical trials evaluating the management of COPD exacerbations
Evidence-based eHealth for COPD
Related content which might interest you:
LPS-induced TLR4 expression and cytokine release is impaired in monocytes and T-lymphocytes of COPD patients
Source: Annual Congress 2008 - Novel targets in the molecular pathology of lung cancer and COPD
Year: 2008
TLR2 and TLR4 induced tolerance in alveolar macrophages; differential effect on TNFα and IL-8 release
Source: Annual Congress 2012 - Cellular signalling mechanisms in the airways
Year: 2012
Exaggerated IL-6 responses to LPS after viral airway infection in mice is mediated by endogenous TNF-a production
Source: Annual Congress 2007 - Immunological function in viral and nonviral pathogens
Year: 2007
In COPD moxifloxacin enhances th1-immune response in the presence of bacterial endotoxin by inducing IL-2 expression
Source: Annual Congress 2009 - Inflammatory cells and cytokines
Year: 2009
Rhinovirus-induced cytokine production of circulating leukocytes is reduced in severe asthma
Source: Virtual Congress 2020 – New insight into the immunology of asthma and COPD
Year: 2020
Overexpression of CBP augments cigarette smoke-induced pro-inflammatory cytokine release in macrophages
Source: Eur Respir J 2005; 26: Suppl. 49, 105s
Year: 2005
Differential gene expression and cytokine production from neutrophils in asthma phenotypes
Source: Eur Respir J 2010; 35: 522-531
Year: 2010
The relationship between PBMC secretion of IFN-alpha following TLR7 and TLR9 activation and lung function in COPD
Source: Annual Congress 2011 - Role of infection in exacerbations of COPD
Year: 2011
IL-33 augments rhinovirus-induced type 2 immune responses in asthma via selective upregulation of one chain of the IL-33 receptor.
Source: International Congress 2017 – "Alarming" immunological patterns in asthma
Year: 2017
Upregulated response to chemokines in oxidative metabolism of eosinophils in asthma and allergic rhinitis
Source: Eur Respir J 2003; 21: 925-931
Year: 2003
Late-breaking abstract: Activation of the macrophage inflammasome by TLR3 ligation potentiates release of IL-8 and IP-10 from alveolar type II epithelial cells via a paracrine cytokine network
Source: Annual Congress 2011 - Epithelial cells: role in health and disease
Year: 2011
Role of endogenous interleukin-17 in early neutrophil recruitment after endotoxin exposure in mouse airways
in vivo
Source: Eur Respir J 2003; 22: Suppl. 45, 566s
Year: 2003
Chemokinome characterization in human lung macrophages following stimulation with Th2-type cytokines IL-4 or IL-13
Source: Annual Congress 2012 - Update on neutrophil and macrophage function in COPD
Year: 2012
Aerobic training modulates parenquimal inflammation
via
decreases of IL-4 and IL-5 release by inflammatory cells
Source: Annual Congress 2007 - Towards an understanding of asthma pathogenesis - novel
in vitro
assays and results from animal models
Year: 2007
NOD1 and TLR4 synergise to elevate release of the pro-inflammatory cytokine CXCL8 in human pulmonary artery smooth muscle cells
Source: Annual Congress 2010 - Sepsis and multiorgan failure
Year: 2010
CD40 ligation modulates inflammatory cytokines production by human bronchial epithelial cells
Source: Eur Respir J 2001; 18: Suppl. 33, 246s
Year: 2001
Statins suppress LPS-induced proinflammatory cytokine production from human bronchial epithelial cells
Source: Annual Congress 2009 - Epithelial cell biology
Year: 2009
Ionomycin stimulates a synergistic effect of IL-8 and induced sputum on PMN chemotaxis during COPD
Source: Annual Congress 2010 - COPD and asthma: models and human studies
Year: 2010
Endotoxin-induced IL-17 release by murine T-lymphocytes: interactions with airway macrophages
Source: Eur Respir J 2001; 18: Suppl. 33, 408s
Year: 2001
The PBMC of severe asthmatics showed enhanced candida antigen induced IL-5, IL-13 and TARC production and this enhancement was inhibited by dendric cells deprition
Source: Annual Congress 2009 - Inflammatory cells and cytokines
Year: 2009
We use cookies on our website to give you the most relevant experience by remembering your preferences and repeat visits. By clicking "Accept", you consent to the use of the cookies.
Accept