e-learning
resources
Vienna 2009
Wednesday, 16.09.2009
From emphysema to fibrosis: mechanisms of cellular remodelling in the diseased lung
Login
Search all ERS
e-learning
resources
Disease Areas
Airways Diseases
Interstitial Lung Diseases
Respiratory Critical Care
Respiratory Infections
Paediatric Respiratory Diseases
Pulmonary Vascular Diseases
Sleep and Breathing Disorders
Thoracic Oncology
Events
International Congress
Courses
Webinars
Conferences
Research Seminars
Journal Clubs
Publications
Breathe
Monograph
ERJ
ERJ Open Research
ERR
European Lung White Book
Handbook Series
Guidelines
All ERS guidelines
e-learning
CME Online
Case reports
Short Videos
SpirXpert
Procedure Videos
CME tests
Reference Database of Respiratory Sounds
Radiology Image Challenge
Brief tobacco interventions
EU Projects
VALUE-Dx
ERN-LUNG
ECRAID
UNITE4TB
Disease Areas
Events
Publications
Guidelines
e-learning
EU Projects
Login
Search
Structural impairment, disturbed cellular composition and increased apoptosis in experimental emphysema
L. Farkas, J. Gauldie, W. Shi, M. Kolb (Hamilton, Canada; Los Angeles, United States Of America)
Source:
Annual Congress 2009 - From emphysema to fibrosis: mechanisms of cellular remodelling in the diseased lung
Session:
From emphysema to fibrosis: mechanisms of cellular remodelling in the diseased lung
Session type:
Oral Presentation
Number:
4455
Rating:
You must
login
to grade this presentation.
Share or cite this content
Citations should be made in the following way:
L. Farkas, J. Gauldie, W. Shi, M. Kolb (Hamilton, Canada; Los Angeles, United States Of America). Structural impairment, disturbed cellular composition and increased apoptosis in experimental emphysema. Eur Respir J 2009; 34: Suppl. 53, 4455
You must
login
to share this Presentation/Article on Twitter, Facebook, LinkedIn or by email.
Member's Comments
No comment yet.
You must
Login
to comment this presentation.
Related content which might interest you:
Panel discussion on ERS Statement – A core outcome set for clinical trials evaluating the management of COPD exacerbations
ERS statement: a core outcome set for clinical trials evaluating the management of COPD exacerbations
Evidence-based eHealth for COPD
Related content which might interest you:
PTEN deficiency in alveolar type 2 cells accelerates cellular senescence, disrupts epithelial–mesenchymal and mitochondrial homeostasis, and induces age-related emphysema
Source: International Congress 2019 – Cellular metabolism and immune senescence in lung pathology
Year: 2019
The expression of antioxidants in small airways in COPD is increased, heterogeneous and correlates with local histopathological alterations
Source: Annual Congress 2009 - COPD: basic science and clinical studies
Year: 2009
Cigarette-decreased NAD/SIRT1 aggravates pulmonary fibrosis by promoting senescence of alveolar type 2 cells through autophagy inhibition and mitochondrial oxidative stress induction
Source: Virtual Congress 2020 – Idiopathic pulmonary fibrosis and interstitial lung disease: progression, genes and more
Year: 2020
Air space distension precedes spontaneous fibrotic remodeling and impaired cholesterol metabolism in the absence of surfactant protein C
Source: ERS Lung Science Conference 2020
Year: 2020
Altered iron metabolism and elevated cellular senescence in COPD small airway epithelial cells
Source: Virtual Congress 2020 – Metabolic alterations and ageing in lung disease
Year: 2020
Gut microbial modulation attenuates emphysema development by suppressing inflammation and apoptosis
Source: Virtual Congress 2020 – New insight into the immunology of asthma and COPD
Year: 2020
Alterations in mitochondrial permeability transition pore contribute to apoptosis resistance in IPF fibroblasts.
Source: International Congress 2019 – Extracellular matrix formation and remodelling in physiology and disease
Year: 2019
Physical stress modulates distal lung tissue mechanics, inflammation and remodelling associated to iNOS and oxidative stress activation
Source: Annual Congress 2008 - Pathophysiology of respiratory diseases
Year: 2008
High dimensional single cell analysis reveals unexpected immune cell types, and loss of motility and chemokine receptor expression of alveolar macrophages in chronic obstructive respiratory disease
Source: International Congress 2018 – Advanced profiling of immune cells in chronic lung diseases
Year: 2018
Dysregulated mitochondrial integrity associated with lamin B1 reduction is involved in cellular senescence progression in COPD pathogenesis.
Source: International Congress 2018 – New kids on the block: novel molecular pathways and therapeutic targets in chronic lung diseases
Year: 2018
Impaired lung function is associated with systemic inflammation and macrophage activation
Source: Eur Respir J 2015; 45: 557-559
Year: 2015
MiRNA-mRNA-protein network integration reveals dysregulation of oxidative stress and apoptosis in smoking-induced COPD in women
Source: Virtual Congress 2020 – Inflammatory pathways in chronic airway diseases
Year: 2020
Increased autophagy and reduced protein synthesis signaling are involved in pulmonary inflammation-induced skeletal muscle atrophy
Source: International Congress 2016 – How the understanding og molecular and genomic crosstalk is helping to diagnose lung disease
Year: 2016
Inflammation induced conformational changes in Z alpha-1 antirypsin promotes lung Z-AT cell damage
Source: Annual Congress 2013 –The multiple faces of stress in lung injury
Year: 2013
Relationship between markers of systemic inflammation and microstructural brain damage in COPD.
Source: International Congress 2019 – From the health effects of tobacco to the epidemiology of tobacco/electronic nicotine delivery system use
Year: 2019
Repeated stress modulates lung tissue mechanics and induces pulmonary inflammation, remodelling and oxidative stress pathway activation in guinea pigs
Source: Annual Congress 2010 - Emerging mechanisms in lung injury
Year: 2010
Low doses of PM2.5 induces inflammation and oxidative stress with lung functional changes in mice
Source: Annual Congress 2008 - Lung, traffic and volcanoes
Year: 2008
Fas activation impairs the alveolar epithelial function in mice by mechanisms involving apoptosis
Source: Annual Congress 2011 - Anti-inflammatory strategies in acute lung injury
Year: 2011
Global gene expression analyses highlight the role of plasma membrane remodeling as a molecular functions of airway epithelium in response to interleukin-13 treatment
Source: International Congress 2015 – Promising novel findings in translational pulmonary research
Year: 2015
High proteasome activity and absence of NF-kB activation in cystic fibrosis cell culture and CF KO mouse models in response to oxidative stress
Source: Eur Respir J 2005; 26: Suppl. 49, 403s
Year: 2005
We use cookies on our website to give you the most relevant experience by remembering your preferences and repeat visits. By clicking "Accept", you consent to the use of the cookies.
Accept