Source: Annual Congress 2009 - Pulmonary rehabilitation: extending the scope and benefit of rehabilitation for patients with chronic respiratory disease
Disease area: Airway diseases

Abstract

Background Pulmonary rehabilitation has become a recommended standard of care for chronic obstructive pulmonary disease(COPD).Exercise training is the centerpiece of the whole rehabilitation program.Some studies showed that higher intensity exercise produced greater improvements in exercise capacity compared with lower intensity exercise.But exercise may be a trigger for myocardial ischaemia or hypoxemia and higher intensity exercise increases the risk more.The aim of this study is to assess the risk during exercise in patients with COPD.
Methods Cardiopulmonary exercise testing was performed in 51 patients(47 men,4 women) with COPD.Oxygen uptake(VO2) and carbon dioxide output(VCO2) were measured breath-by-breath .Arterial oxygen saturation was measured by pulse osimetry and ten-lead ECG was monitored continuously throughout testing.Blood pressure was measured with the cuff technique.Arterial blood samples were drawn both at rest and maximal exercise.
Results There were 13 patients with stageI,23 patients with stageII and 15 patients with stageIII.The work rate,VO2 and heart rate at peak exercise were 93.51±34.32 watts,15.51±4.08 ml/kg/min, 129.74±19.61 beats/min.The blood pressure rised from 145.47±22.83/79.66±8.56 mmHg at rest to 183.92/95.75±12.57 mmHg at peak exercise. There was a little decrease in PaO₂ from 95.91±17.91 mmHg at rest to 91.30±16.04 mmHg at peak exercise,but a little increase in PaCO₂ during exercise. We did not find ischaemic ST-segment changes and frequent ectopic beats on ECG.
Conclusions Exercise training is safe for most patients with mild-to-moderate COPD if the resting arterial oxygen are more than 80 mmHg and there are no signs of myocardial ischaemia at rest.

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Citations should be made in the following way:
Q. Xu, H. Wang (Beijing, China). Risk of exercise training in patients with chronic obstructive pulmonary disease. Eur Respir J 2009; 34: Suppl. 53, 528

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