e-learning
resources
Munich 2006
Monday 04.09.2006
Mechanisms of acute lung injury and of fibrosis
Login
Search all ERS
e-learning
resources
Disease Areas
Airways Diseases
Interstitial Lung Diseases
Respiratory Critical Care
Respiratory Infections
Paediatric Respiratory Diseases
Pulmonary Vascular Diseases
Sleep and Breathing Disorders
Thoracic Oncology
Events
International Congress
Courses
Webinars
Conferences
Research Seminars
Journal Clubs
Publications
Breathe
Monograph
ERJ
ERJ Open Research
ERR
European Lung White Book
Handbook Series
Guidelines
All ERS guidelines
e-learning
CME Online
Case reports
Short Videos
SpirXpert
Procedure Videos
CME tests
Reference Database of Respiratory Sounds
Radiology Image Challenge
Brief tobacco interventions
EU Projects
VALUE-Dx
ERN-LUNG
ECRAID
UNITE4TB
Disease Areas
Events
Publications
Guidelines
e-learning
EU Projects
Login
Search
Hyperoxia-induced gene expression and apoptosis of human pulmonary artery endothelial cells
A. Motozuka, K. Kokubo, N. Tanaka, T. Shinbo, M. Hirose, N. Masuda, H. Kobayashi (Sagamihara, Japan)
Source:
Annual Congress 2006 - Mechanisms of acute lung injury and of fibrosis
Session:
Mechanisms of acute lung injury and of fibrosis
Session type:
Thematic Poster Session
Number:
2346
Disease area:
Pulmonary vascular diseases
Rating:
You must
login
to grade this presentation.
Share or cite this content
Citations should be made in the following way:
A. Motozuka, K. Kokubo, N. Tanaka, T. Shinbo, M. Hirose, N. Masuda, H. Kobayashi (Sagamihara, Japan). Hyperoxia-induced gene expression and apoptosis of human pulmonary artery endothelial cells. Eur Respir J 2006; 28: Suppl. 50, 2346
You must
login
to share this Presentation/Article on Twitter, Facebook, LinkedIn or by email.
Member's Comments
No comment yet.
You must
Login
to comment this presentation.
Related content which might interest you:
Plasma surfactant protein D (SP-D) in patients (pts) with stable (S) COPD and during COPD acute exacerbation (AE)
Severe respiratory infection: when is ICU mandatory?
Comment on: Mesenchymal stromal (stem) cell therapy modulates miR-193b-5p expression to attenuate sepsis-induced acute lung injury
Related content which might interest you:
Hypoxia-induced increase in NO production and eNOS expression by murine pulmonary microvascular endothelial cells
Source: Annual Congress 2006 - Pulmonary circulation: inflammation and remodelling
Year: 2006
Alkalosis induce eNOS activation in human pulmonary arterial endothelial cells
Source: Eur Respir J 2001; 18: Suppl. 33, 73s
Year: 2001
Kaempferol-induced HO-1 expression attenuated inflammation in human pulmonary alveolar epithelial cells
Source: International Congress 2017 – Therapeutic approaches in lung disease
Year: 2017
Temporal changes in gene expressions and cytokine productions caused by stretching of normal human pulmonary artery endothelial cells
Source: Annual Congress 2010 - Determinants of acute lung injury and fibrosis
Year: 2010
microRNA-34a and microRNA 199a-5p in COPD and their control of HIF-1α expression in pulmonary vasucular endothelial cells
Source: Annual Congress 2011 - Molecular: pathology of infectious and inflammatory lung disease
Year: 2011
Hypoxia-induced proliferation of human pulmonary artery smooth muscle cells: possible mechanisms
Source: Eur Respir J 2004; 24: Suppl. 48, 561s
Year: 2004
Implication of aquaporin-1 in the stress response of human pulmonary microvascular endothelial cells
Source: International Congress 2019 – Biomarkers, laboratory findings and pre-clinical data in acute respiratory failure
Year: 2019
Overexpression of pro-apoptotic factors and endothelial cell death in chronically obstructed and reperfused pulmonary artery
Source: Eur Respir J 2007; 30: Suppl. 51, 488s
Year: 2007
Effects of hypoxia on VEGF and VEGF-R expression by human alveolar epithelial (ATII) and human pulmonary microvascular endothelial (HMVEC-l) cells
Source: Annual Congress 2008 - Border patrol: the alveolar barrier in lung injury
Year: 2008
miR 16 modulates human pulmonary artery smooth muscle cell phenotype
Source: Annual Congress 2012 - Pulmonary circulation: basic mechanisms, animal models and cell biology
Year: 2012
The gene expression of the endothelial to mesenchymal transition-related transcription factor
slug
is increased in remodelled pulmonary arteries of COPD
Source: Annual Congress 2010 - Pathobiology of pulmonary hypertension
Year: 2010
The pro-proliferative phenotype of pulmonary microvascular endothelium
Source: Research Seminar 2005 - Pulmonary Circulation: Molecular Determinants of Pulmonary Vascular Remodelling
Year: 2005
Wnt5a down-regulated hypoxia-induced pulmonary arterial smooth muscle cell proliferation by inhibition of β-catenin
Source: Annual Congress 2010 - Pathobiology of pulmonary hypertension
Year: 2010
Inhibition of miR-34a protects human umbilical vein endothelial cells against intermittent hypoxia-induced endothelial injury by targeting ATG7
Source: Virtual Congress 2020 – Pathophysiology and treatment of obstructive sleep apnoea
Year: 2020
Notch1 signalling regulates endothelial proliferation and apoptosis in pulmonary arterial hypertension
Source: Eur Respir J 2016; 48: 1137-1149
Year: 2016
Cigarette smoke- and hypoxia-induced imbalanced vasoactive gene expression in human pulmonary artery smooth muscle cells
Source: International Congress 2017 – Experimental PH
Year: 2017
Mir-17 modulates smooth muscle cell markers, apoptosis and BMPR-II levels in human pulmonary artery smooth muscle cells
Source: Annual Congress 2012 - Pulmonary circulation: basic mechanisms, animal models and experimental treatments
Year: 2012
Role of c-Abelson in the loss of genome integrity in endothelial cells in pulmonary arterial hypertension
Source: Virtual Congress 2020 – Pathophysiology of pulmonary hypertension
Year: 2020
Bosentan stimulates the expression of cell cycle inhibitor p27(kip1) in pulmonary artery smooth muscle cells (PASMC)
Source: Annual Congress 2007 - Animal and
ex vivo
models of airway inflammation
Year: 2007
Effect of eNOS gene overexpression on the vascular reactivity of isolated pulmonary arteries of chronic hypoxic rats
Source: Annual Congress 2006 - Taormina 4th ERS Lung Science Conference "hypoxia in lung biology and disease"
Year: 2006
We use cookies on our website to give you the most relevant experience by remembering your preferences and repeat visits. By clicking "Accept", you consent to the use of the cookies.
Accept