Effects of TGF-beta and budesonide on MAPK phosphorylation, IL-6/IL-11 secretion, and cell proliferation in human lung fibroblasts Source: Eur Respir J 2006; 28: Suppl. 50, 662s Year: 2006
17-beclomethasone monopropionate or inhibition of MAPK p38 signalling abolish IL-8 release stimulated by cigarette smoke extract or acrolein in normal human airway smooth muscle cells Source: Annual Congress 2010 - Airway smooth muscle cells Year: 2010
Modulation of bradykinin (BK)-induced lung fibroblast proliferation and differentiation by budesonide (Bud) and formoterol (Form) involves the inhibition of ERK1/2 phosphorylation and Ca2+ mobilization Source: Annual Congress 2009 - Translational models of lung disease Year: 2009
Proteoglycan production in cultured human lung fibroblasts is synergistcally inhibited by combination of budesonide and formoterol Source: Eur Respir J 2003; 22: Suppl. 45, 286s Year: 2003
Rapid inhibitory effects of budesonide and formoterol on bradykinin-induced Ca2+ mobilization in human lung fibroblasts Source: Annual Congress 2010 - Regulation of airway pharmacology Year: 2010
TGFβ-induced proteoglycan production and α-smooth muscle actin expression in cultured human lung fibroblasts is inhibited by combination of budesonide and formoterol Source: Eur Respir J 2004; 24: Suppl. 48, 251s Year: 2004
N-Acetylcysteine inhibits eotaxin and MCP-1 release in human airway smooth muscle cells in vitro through the inhibition of ROS production, resulting in a decrease of p38 MAP kinase activation Source: Eur Respir J 2002; 20: Suppl. 38, 170s Year: 2002
Effects of budesonide on p38 MAPK activation, apoptosis and IL-8 secretion, induced by TNF-alpha and Haemophilus influenzae in human bronchial epithelial cells Source: Annual Congress 2010 - Regulation of airway pharmacology Year: 2010
Inhibition of P38 kinase does not reduce production of TNF-alpha from airway cells in COPD Source: Annual Congress 2007 - Epithelial networks in the airways Year: 2007
Protective effect of MAPK inhibitors and budesonide against apoptosis induced by TGF-β in primary cultures of human bronchial epithelial cells Source: Eur Respir J 2003; 22: Suppl. 45, 394s Year: 2003
LATE-BREAKING ABSTRACT: Roflumilast and its active metabolite inhibit LPS-induced cytokines production from human parenchymal and bronchial explants: interaction with formoterol Source: Annual Congress 2009 - Airway cell biology Year: 2009
Theophylline at the therapeutic concentrations abrogate decreased smad7 expression in cultured smooth muscle cells stimulated with LTD4 and EGF Source: Eur Respir J 2005; 26: Suppl. 49, 25s Year: 2005
Budesonide prevents cytokine-induced decrease of the relaxant response to formoterol but not to salmeterol in mouse trachea Source: Annual Congress 2009 - Translational research/biomarkers Year: 2009
Dexamethasone and formoterol modulate human rhinovirus induced MMP-9 expression Source: Annual Congress 2010 - Lung cell biology and immunology Year: 2010
Formoterol is more potent and more effective than salmeterol in the inhibition of oxidative burst in human eosinophils Source: Eur Respir J 2001; 18: Suppl. 33, 49s Year: 2001
Synergistic inhibition of oxidative burst in human eosinophils by combination treatment with formoterol and budesonide Source: Eur Respir J 2001; 18: Suppl. 33, 48s Year: 2001
Muscarinic receptors mediate stimulation of collagen synthesis in human lung fibroblasts Source: Eur Respir J 2008; 32: 555-562 Year: 2008
Combination of budesonide and formoterol is more effective in inhibiting GM-CSF production by bronchial epithelial cells than either drug alone Source: Eur Respir J 2003; 22: Suppl. 45, 457s Year: 2003
Differential anti-inflammatory effects of budesonide and a p38 MAPK inhibitor AZD7624 on COPD pulmonary cells Source: International Congress 2018 – Preclinical pharmacology: novel targets in airway inflammation, narrowing and neural control Year: 2018
Ceramide-1-phosphate decreases allergic airway inflammation, remodeling and hyperresponsiveness by inhibition of phosphorilation of NF-kappa B, Akt, MAPK p-38 in dendritic cells Source: Annual Congress 2010 - Models of airways disease Year: 2010