e-learning
resources
Berlin 2008
Monday, 06.10.2008
Understanding allergic airway inflammation with the help of animal models
Login
Search all ERS
e-learning
resources
Disease Areas
Airways Diseases
Interstitial Lung Diseases
Respiratory Critical Care
Respiratory Infections
Paediatric Respiratory Diseases
Pulmonary Vascular Diseases
Sleep and Breathing Disorders
Thoracic Oncology
Events
International Congress
Courses
Webinars
Conferences
Research Seminars
Journal Clubs
Publications
Breathe
Monograph
ERJ
ERJ Open Research
ERR
European Lung White Book
Handbook Series
Guidelines
All ERS guidelines
e-learning
CME Online
Case reports
Short Videos
SpirXpert
Procedure Videos
CME tests
Reference Database of Respiratory Sounds
Radiology Image Challenge
Brief tobacco interventions
EU Projects
VALUE-Dx
ERN-LUNG
ECRAID
UNITE4TB
Disease Areas
Events
Publications
Guidelines
e-learning
EU Projects
Login
Search
Role of Clara cell protein (CC)- 10 in the regulation of allergic airway inflammation and tolerance induction in mice
M. Raes, C. Van Hove, G. Joos, T. Kurt (Ghent, Belgium)
Source:
Annual Congress 2008 - Understanding allergic airway inflammation with the help of animal models
Session:
Understanding allergic airway inflammation with the help of animal models
Session type:
Thematic Poster Session
Number:
2021
Disease area:
Airway diseases
Rating:
You must
login
to grade this presentation.
Share or cite this content
Citations should be made in the following way:
M. Raes, C. Van Hove, G. Joos, T. Kurt (Ghent, Belgium). Role of Clara cell protein (CC)- 10 in the regulation of allergic airway inflammation and tolerance induction in mice. Eur Respir J 2008; 32: Suppl. 52, 2021
You must
login
to share this Presentation/Article on Twitter, Facebook, LinkedIn or by email.
Member's Comments
No comment yet.
You must
Login
to comment this presentation.
Related content which might interest you:
Late Breaking Abstract - Implications of treatable traits and treatment choices on exacerbation risk in moderate-severe asthma
Management of Severe Asthma in Pediatric Patients by an Interdisciplinary Team in a Public Hospital Setting.
Respiratory sequelae of preterm birth across the life span
Related content which might interest you:
Modulation of inflammatory cells apoptosis induced by oral tolerance in a model of chronic allergic airway inflammation in guinea pigs
Source: Annual Congress 2005 - Regulation of the allergic and asthmatic response
Year: 2005
Airway epithelial cell P2Y6R-Expression is up-regulated during acute and chronic allergic inflammation and contributes via modulating of CXC8 and IL-6 secretion to development of airway remodelling
Source: Annual Congress 2010 - Pre-clinical models of airways disease
Year: 2010
Neutrophil predominance phenotype in bronchial asthma. Therapeutic implication and the role of Clara cell 16-kD protein
Source: Annual Congress 2007 - Invasive and noninvasive methods to assess airway inflammation
Year: 2007
Inhibition of ER stress suppresses IL-13 induced airway epithelial remodeling
Source: International Congress 2019 – COPD, asthma and rhinitis: from cellular mechanisms to patients’ clinical responses
Year: 2019
A role for Th1 cells in airway remodeling and hyperreactivity in mice subjected to chronic allergen exposure
Source: Eur Respir J 2007; 30: Suppl. 51, 734s
Year: 2007
Increased expression of Lipocalin2 suppresses allergen-induced airway disease in mice
Source: Annual Congress 2008 - Novel mechanisms and targets of molecular pulmonary pathology
Year: 2008
Interactions between zinc deficiency and murine airway inflammation on apoptosis in airway epithelium
Source: Eur Respir J 2003; 22: Suppl. 45, 267s
Year: 2003
Attenuation of airway mucosal dendritic cell function in the induction of T cell hyporesponsiveness following chronic aeroallergen exposure
Source: Annual Congress 2010 - Animal models of asthma and lung inflammation
Year: 2010
Soluble biglycan promotes ovalbumin induced airway inflammation by modulating dendritic cell function in mice
Source: International Congress 2019 – Novel mechanisms elucidated by translational studies of asthma
Year: 2019
Transgenic mice overexpressing IL-18 display increased IL-13 in the airways but no changes of the airway hyperresponsiveness in a murine model of late-phase asthma: evidence for a dual role of IL-18 on mucosal Th1/Th2 responses
Source: Eur Respir J 2002; 20: Suppl. 38, 1s
Year: 2002
Clara cells drive eosinophil accumulation in allergic asthma
Source: Eur Respir J 2012; 39: 429-438
Year: 2012
Increased susceptibility to airway allergic responses in CD40 deficient mice
Source: Eur Respir J 2002; 20: Suppl. 38, 567s
Year: 2002
γδ T cells contribute to local and systemic B cell reactivity in allergic eosinophilic airway inflammation but without a major influence on the TH1/TH2 balance
Source: Eur Respir J 2002; 20: Suppl. 38, 568s
Year: 2002
CCL6 produced by eosinophils drives allergic airway inflammation in mice
Source: International Congress 2019 – Modelling and monitoring of airway diseases
Year: 2019
Endotoxin interferes with allergy-induced airway inflammation in mice
Source: Eur Respir J 2004; 24: Suppl. 48, 348s
Year: 2004
A dual role of IL-6 signalling and STAT-3 activation on airway inflammation versus airway hyperresponsiveness in a murine model of asthma
Source: Eur Respir J 2002; 20: Suppl. 38, 568s
Year: 2002
Interleukin-17A induces glucocorticoid insensitivity in human bronchial epithelial cells
Source: Eur Respir J 2012; 39: 439-445
Year: 2012
Evidence for secretion of IL-18 in asthmatic airway inflammation assessed by endobronchial allergen challenge and its relationship to accumulation of inflammatory cells
Source: Eur Respir J 2001; 18: Suppl. 33, 531s
Year: 2001
We use cookies on our website to give you the most relevant experience by remembering your preferences and repeat visits. By clicking "Accept", you consent to the use of the cookies.
Accept