e-learning
resources
Berlin 2008
Tuesday, 07.10.2008
COPD and other environment-related lung diseases
Login
Search all ERS
e-learning
resources
Disease Areas
Airways Diseases
Interstitial Lung Diseases
Respiratory Critical Care
Respiratory Infections
Paediatric Respiratory Diseases
Pulmonary Vascular Diseases
Sleep and Breathing Disorders
Thoracic Oncology
Events
International Congress
Courses
Webinars
Conferences
Research Seminars
Journal Clubs
Publications
Breathe
Monograph
ERJ
ERJ Open Research
ERR
European Lung White Book
Handbook Series
Guidelines
All ERS guidelines
e-learning
CME Online
Case reports
Short Videos
SpirXpert
Procedure Videos
CME tests
Reference Database of Respiratory Sounds
Radiology Image Challenge
Brief tobacco interventions
EU Projects
VALUE-Dx
ERN-LUNG
ECRAID
UNITE4TB
Disease Areas
Events
Publications
Guidelines
e-learning
EU Projects
Login
Search
DNA damage in macrophages caused by exposure nanoparticles at an air/tissue interface
A. Tellabati, P. Howes, D. Jones, J. Grigg (Leicester, London, United Kingdom)
Source:
Annual Congress 2008 - COPD and other environment-related lung diseases
Session:
COPD and other environment-related lung diseases
Session type:
Thematic Poster Session
Number:
3832
Disease area:
Airway diseases
Rating:
You must
login
to grade this presentation.
Share or cite this content
Citations should be made in the following way:
A. Tellabati, P. Howes, D. Jones, J. Grigg (Leicester, London, United Kingdom). DNA damage in macrophages caused by exposure nanoparticles at an air/tissue interface. Eur Respir J 2008; 32: Suppl. 52, 3832
You must
login
to share this Presentation/Article on Twitter, Facebook, LinkedIn or by email.
Member's Comments
No comment yet.
You must
Login
to comment this presentation.
Related content which might interest you:
Panel discussion: Lung function in epidemiological studies from infancy to childhood with focus on urban environment
Panel discussion on ERS Statement – A core outcome set for clinical trials evaluating the management of COPD exacerbations
ERS statement: a core outcome set for clinical trials evaluating the management of COPD exacerbations
Related content which might interest you:
LATE-BREAKING ABSTRACT: Induction of inflammation, oxidative stress and autophagy in human alveolar type I epithelial cells following exposure to silver nanoparticles
Source: Annual Congress 2012 - Molecular biology of pro- and anti-inflammatory responses in the lung
Year: 2012
Nanoparticle-driven DNA damage mimics irradiation-related carcinogenesis pathways
Source: Eur Respir J 2008; 31: 241-251
Year: 2008
Nanoparticle induction of oxidative stress in human alveolar epithelium reflects surface chemistry
Source: Annual Congress 2010 - Epithelial cells: cell biology and role in lung disease
Year: 2010
Exposure of alveolar epithelial cells to zinc oxide particles at the air-liquid interface
Source: Eur Respir J 2006; 28: Suppl. 50, 617s
Year: 2006
Lung-lining fluid proteins bind to ultrafine insoluble particles: a potential way for particles to pass air-blood barrier of the lung?
Source: Eur Respir J 2004; 24: Suppl. 48, 100s
Year: 2004
Pulmonary toxicity induced by electric charged soot particles in mice
Source: Virtual Congress 2020 – Occupational and environmental causes of respiratory disease
Year: 2020
Autophagic and apoptotic changes in macrophages in response to graphene oxides with or without chemical modification
Source: International Congress 2019 – Involvement of the environment and inflammation in pulmonary remodelling
Year: 2019
Adhesive properties of human endothelial cells exposed to amorphous silica nanoparticles
Source: Annual Congress 2010 - Causal factors and mechanisms of outdoor air pollution and nanoparticulates
Year: 2010
Metal-handling protein and antioxidant homeostasis at the air-lung interface in response to bronchial instillation of ambient particles
Source: Eur Respir J 2007; 30: Suppl. 51, 144s
Year: 2007
Imaging of the interaction of fine particles and nanoparticles with lung cells
Source: Annual Congress 2009 - WS1 - New imaging methods for the researcher: believe what you see!
Year: 2009
Junctional modulation by lipopolysaccharide on epithelial damage caused by diesel exhaust particles
Source: International Congress 2018 – COPD: novel pathological mechanisms in the airway epithelium
Year: 2018
No evidence electric charge increases inhaled ultrafine particle deposition in human lungs
Source: Virtual Congress 2020 – Occupational and environmental causes of respiratory disease
Year: 2020
Asian dust particles and titanium dioxide-induced pulmonary inflammation, oxidative stress and DNA damage
Source: Annual Congress 2009 - Old and new topics in environmental and occupational lung disease
Year: 2009
Pro-inflammatory effects of exposure of the combination of silicon- and iron-containing particles upon human lung fibroblasts
Source: International Congress 2019 – Effects of environmental exposures in lung pathology and the underlying mechanisms
Year: 2019
Elevated markers of lipid oxidative damage among workers exposed to engineered TiO2 nanoparticles
Source: International Congress 2016 – Human responses to exposure, including physiological and biomarker outcomes
Year: 2016
The compatible solute ectoine prevents neutrophilic lung inflammation induced by environmental model nanoparticles in vivo
Source: Annual Congress 2010 - Cell biology and immunology of lung disease
Year: 2010
Nanoparticles driven DNA damage cell signalling mimicks irradiation model of cancerogenesis
Source: Eur Respir J 2005; 26: Suppl. 49, 151s
Year: 2005
Effect of surface functionalisation on the interaction of carbon nanotubes with primary human alveolar cells in vitro.
Source: International Congress 2017 – New insights into pathogenesis of lung disease
Year: 2017
DNA damage of murine peripheral blood monocytes and histopathologic changes of murine lung induced by Asian dust particles
Source: Annual Congress 2008 - COPD and other environment-related lung diseases
Year: 2008
Toxicological and inflammatory effects of engineered nanoparticles and diesel exhaust particles on human mesothelial cells
Source: International Congress 2017 – Translational studies of lung disease
Year: 2017
We use cookies on our website to give you the most relevant experience by remembering your preferences and repeat visits. By clicking "Accept", you consent to the use of the cookies.
Accept