Oxidised IL-33 signals via RAGE/EGFR to drive a COPD-associated phenotype

S. Strickson (Cambridge, United Kingdom), K. Houslay (Cambridge, United Kingdom), V. Negri (Cambridge, United Kingdom), Y. Ohne (Gaithersburg, United States), T. Ottosson (Gothenburg, Sweden), R. Dodd (Cambridge, United Kingdom), C. Chaillan Huntington (Cambridge, United Kingdom), J. Li (Cambridge, United Kingdom), H. Killick (Cambridge, United Kingdom), D. Rees (Cambridge, United Kingdom), S. Koch (Cambridge, United Kingdom), G. Sims (Gaithersburg, United States), I. Scott (Cambridge, United Kingdom), X. Romero Ros (Cambridge, United Kingdom), E. Cohen (Cambridge, United Kingdom)

Source: International Congress 2022 – It is all about communication: novel interactions between lung structural cells and the immune system

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Citations should be made in the following way:
S. Strickson (Cambridge, United Kingdom), K. Houslay (Cambridge, United Kingdom), V. Negri (Cambridge, United Kingdom), Y. Ohne (Gaithersburg, United States), T. Ottosson (Gothenburg, Sweden), R. Dodd (Cambridge, United Kingdom), C. Chaillan Huntington (Cambridge, United Kingdom), J. Li (Cambridge, United Kingdom), H. Killick (Cambridge, United Kingdom), D. Rees (Cambridge, United Kingdom), S. Koch (Cambridge, United Kingdom), G. Sims (Gaithersburg, United States), I. Scott (Cambridge, United Kingdom), X. Romero Ros (Cambridge, United Kingdom), E. Cohen (Cambridge, United Kingdom). Oxidised IL-33 signals via RAGE/EGFR to drive a COPD-associated phenotype. 2940

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