e-learning
resources
Virtual 2020
Pre-Congress Content
Translational therapeutic advances
Login
Search all ERS
e-learning
resources
Disease Areas
Airways Diseases
Interstitial Lung Diseases
Respiratory Critical Care
Respiratory Infections
Paediatric Respiratory Diseases
Pulmonary Vascular Diseases
Sleep and Breathing Disorders
Thoracic Oncology
Events
International Congress
Courses
Webinars
Conferences
Research Seminars
Journal Clubs
Publications
Breathe
Monograph
ERJ
ERJ Open Research
ERR
European Lung White Book
Handbook Series
Guidelines
All ERS guidelines
e-learning
CME Online
Case reports
Short Videos
SpirXpert
Procedure Videos
CME tests
Reference Database of Respiratory Sounds
Radiology Image Challenge
Brief tobacco interventions
EU Projects
VALUE-Dx
ERN-LUNG
ECRAID
UNITE4TB
Disease Areas
Events
Publications
Guidelines
e-learning
EU Projects
Login
Search
Transforming growth factor beta (TGF-ß) pathway activation by IPF fibroblast-derived soluble factors is mediated by IL-6 trans-signaling
G. Epstein Shochet (Kfar-Saba, Israel), B. Bardenstein-Wald (Tel Aviv, Israel), E. Brook (Tel Aviv, Israel), D. Shitrit (Tel Aviv, Israel)
Source:
Virtual Congress 2020 – Translational therapeutic advances
Session:
Translational therapeutic advances
Session type:
E-poster session
Number:
3352
Disease area:
Interstitial lung diseases
Rating:
You must
login
to grade this presentation.
Share or cite this content
Citations should be made in the following way:
G. Epstein Shochet (Kfar-Saba, Israel), B. Bardenstein-Wald (Tel Aviv, Israel), E. Brook (Tel Aviv, Israel), D. Shitrit (Tel Aviv, Israel). Transforming growth factor beta (TGF-ß) pathway activation by IPF fibroblast-derived soluble factors is mediated by IL-6 trans-signaling. 3352
You must
login
to share this Presentation/Article on Twitter, Facebook, LinkedIn or by email.
Member's Comments
No comment yet.
You must
Login
to comment this presentation.
Related content which might interest you:
Panel discussion: Diagnostic tools for Obstructive Sleep Apnoea in adults and children
Expert interview: Physiological classification of lung function impairment
Occupational exposures to respiratory diseases: A case-based discussion
Related content which might interest you:
Dimethylfumarate reduces growth factor- and cytokine-induced IL-6 release from human lung fibroblasts: role of (NF)-κB
Source: Annual Congress 2008 - Airway cell biology and immunology
Year: 2008
Transforming growth factor-β induces collagen synthesis without inducing IL-8 production in mesothelial cells
Source: Eur Respir J 2003; 22: 197-202
Year: 2003
Insulin-like growth factor binding protein-3 induction by transforming growth factor-β1 mediates differentiation of a myofibroblast phenotype in human lung fibroblasts
Source: Eur Respir J 2003; 22: Suppl. 45, 7s
Year: 2003
Pharmacological inhibition of epidermal growth factor receptor modulates cigarette smoke-induced CXCL8 release but not VEGF release
Source: Annual Congress 2012 - Basic and translational studies in COPD
Year: 2012
Transforming growth factor beta (TGFβ)-2 and -3 gene polymorphisms in different phenotypes of sarcoidosis
Source: Annual Congress 2009 - What is new in molecular pathology and functional genomics of neoplastic and non-neoplastic lung disease?
Year: 2009
Transforming growth factor-beta 1 (TGF-beta1) gene polymorphism in coincidence with some other host and environmental factors is associated with irreversible bronchoconstriction in asthmatics
Source: Eur Respir J 2006; 28: Suppl. 50, 191s
Year: 2006
The hypoxia-inducible factor 1a protein stabilizers inhibit transforming growth factor ß-induced extracellular matrix proteins in epithelial cells and fibroblasts.
Source: International Congress 2018 – Pulmonary fibrosis: from models to patients
Year: 2018
Transforming growth factor-α (TGF-α)-induced interleukin-8 (IL-8) release in human bronchial epithelial cells in vitro: role of mitogen-activated protein kinases (MAPK)
Source: Eur Respir J 2002; 20: Suppl. 38, 573s
Year: 2002
Intermittent hypoxia activates the Interleukin (IL)-1ß and nuclear factor (NF)?B signaling pathway in primary murine macrophages
Source: International Congress 2019 – Insights into the pathophysiology of sleep-disordered breathing
Year: 2019
Transforming growth factor-β inhibition of lung fibroblast proliferation is mediated through prostaglandin E
2
,
via
activation of p38 mitogen-activated protein kinase
Source: Annual Congress 2005 - Pulmonary hypertension and fibrosis: novel mechanisms
Year: 2005
The natural anticoagulant activated protein C inhibits the expression of platelet-derived growth factor in the lung
Source: Eur Respir J 2002; 20: Suppl. 38, 571s
Year: 2002
Transforming growth factor beta induces Beclin-1 in pulmonary arterial endothelial cells
Source: Virtual Congress 2020 – Chronic thromboembolic pulmonary hypertension
Year: 2020
Expression of transforming growth factor (TGF) in bronchial epithelium: association with COPD
Source: Eur Respir J 2001; 18: Suppl. 33, 92s
Year: 2001
Inhibition of VEGF blocks TGF-β1 production through a PI3K/Akt signalling pathway
Source: Eur Respir J 2008; 31: 523-531
Year: 2008
The role of integrins in pulmonary fibrosis
Source: Eur Respir Rev 2008; 17: 157-162
Year: 2008
Overexpression of fibroblast growth factor and down-regulation of platelet-derived growth factor correlate with advanced-stage lung fibrosis
Source: Annual Congress 2010 - Cell biology and immunology of lung disease
Year: 2010
Jun kinase regulates the expression of TGF-beta induced connective tissue growth factor in human lung fibroblast
Source: Annual Congress 2004 - Inflammation and remodelling in human lungs
Year: 2004
The contribution of transforming growth factor-β and epidermal growth factor signalling to airway remodelling in chronic asthma
Source: Eur Respir J 2006; 27: 208-229
Year: 2006
Influence of tobacco-smoking on serum level of transforming growth factor (TGF) beta 1 in COPD patients
Source: International Congress 2017 – COPD biomarkers
Year: 2017
We use cookies on our website to give you the most relevant experience by remembering your preferences and repeat visits. By clicking "Accept", you consent to the use of the cookies.
Accept