Source: Eur Respir J, 52 (4) 1800437; 10.1183/13993003.00437-2018
Disease area: Airway diseases



Rating:

Share or cite this content

Citations should be made in the following way:
Lisa Giovannini-Chami, Agnès Paquet, Céline Sanfiorenzo, Nicolas Pons, Julie Cazareth, Virginie Magnone, Kévin Lebrigand, Benoit Chevalier, Ambre Vallauri, Valérie Julia, Charles-Hugo Marquette, Brice Marcet, Sylvie Leroy, Pascal Barbry. The “one airway, one disease” concept in light of Th2 inflammation. Eur Respir J, 52 (4) 1800437; 10.1183/13993003.00437-2018

Member's Comments

No comment yet.

Related content which might interest you: Late Breaking Abstract - The “one airway, one disease” concept at the light of Th2 inflammation Source: International Congress 2017 – Gene signatures in bronchial diseases Year: 2017 Interferon-γ-pretreated dendritic cells induced dual Th1 and Th2 responses and failed to prevent development of allergic airway inflammation Source: Eur Respir J 2006; 28: Suppl. 50, 230s Year: 2006 Local application of FTY720 to the lung abrogates experimental asthma by altering myeloid dendritic cell function Source: Annual Congress 2006 - Towards understanding the pathogenesis of asthma Year: 2006 Resolution of inflammation in airway diseases Source: Annual Congress 2005 - Mechanisms of resolution of inflammation Year: 2005 IL-33 blockade impacts mediators of persistence and exacerbation of chronic airway inflammation. Source: International Congress 2018 – Novel drug targets for asthma and COPD Year: 2018 Treatment: how does inflammatory phenotype influence response to treatment in airway disease? Source: Annual Congress 2007 - Inflammatory phenotypes in airway disease Year: 2007 Different Th1, Th2, Th17 and Treg associated chemokine local pattern expression in lung in a murine model of allergic inflammation Source: Annual Congress 2011 - T-cell subsets Year: 2011 Airway epithelial inflammatory responses and clinical parameters in COPD Source: Eur Respir J 2003; 22: 94-99 Year: 2003 γδ T cells contribute to local and systemic B cell reactivity in allergic eosinophilic airway inflammation but without a major influence on the TH1/TH2 balance Source: Eur Respir J 2002; 20: Suppl. 38, 568s Year: 2002 Th2 immune response and respiratory bronchiolitis-associated interstitial lung disease in p66Shc KO smoking mice Source: Annual Congress 2008 - Inflammatory trigger mechanisms in asthma and COPD Year: 2008 Chymase-positive mast cells represent a major source for IL-17A in airway inflammation Source: International Congress 2015 – Mucosal immunology: what’s new in 2015 Year: 2015 IL-17A from innate and adaptive lymphocytes contributes to inflammation and damage in cystic fibrosis lung disease Source: Eur Respir J, 57 (6) 1900716; 10.1183/13993003.00716-2019 Year: 2021 Farm dust decreases Th2 driven allergic airway inflammation in mice: A role for airway TLR2 and TLR4? Source: Annual Congress 2011 - T-cell subsets Year: 2011 Aerobic training decreases epithelial expression of Th2 cytokines in a murine model of allergic lung inflammation Source: Annual Congress 2007 - Towards an understanding of asthma pathogenesis - novel in vitro assays and results from animal models Year: 2007 ST2 gene knockout attenuates allergic airways disease Source: Annual Congress 2006 - Immunological mechanisms in cell recruitment to the lungs Year: 2006 Airway inflammation in COPD: physiological outcome measures and induced sputum Source: Eur Respir J 2003; 21: 19S-28S Year: 2003 ILC2s in allergic airway inflammation Source: International Congress 2014 – Highlights from the Lung Science Conference 2014 Year: 2014 CCL6 produced by eosinophils drives allergic airway inflammation in mice Source: International Congress 2019 – Modelling and monitoring of airway diseases Year: 2019