e-learning
resources
Paris 2018
Monday, 17.09.2018
Novel drug targets for asthma and COPD
Login
Search all ERS
e-learning
resources
Disease Areas
Airways Diseases
Interstitial Lung Diseases
Respiratory Critical Care
Respiratory Infections
Paediatric Respiratory Diseases
Pulmonary Vascular Diseases
Sleep and Breathing Disorders
Thoracic Oncology
Events
International Congress
Courses
Webinars
Conferences
Research Seminars
Journal Clubs
Publications
Breathe
Monograph
ERJ
ERJ Open Research
ERR
European Lung White Book
Handbook Series
Guidelines
All ERS guidelines
e-learning
CME Online
Case reports
Short Videos
SpirXpert
Procedure Videos
CME tests
Reference Database of Respiratory Sounds
Radiology Image Challenge
Brief tobacco interventions
EU Projects
VALUE-Dx
ERN-LUNG
ECRAID
UNITE4TB
Disease Areas
Events
Publications
Guidelines
e-learning
EU Projects
Login
Search
Targeting NADPH oxidase-2 reduces cigarette smoke-induced lung inflammation and skeletal muscle wasting in mice
I. Bernardo (Bundoora, Australia), S. Passey (Bundoora, Australia), H. Seow (Bundoora, Australia), S. Bozinovski (Bundoora, Australia), R. Vlahos (Bundoora, Australia)
Source:
International Congress 2018 – Novel drug targets for asthma and COPD
Session:
Novel drug targets for asthma and COPD
Session type:
Oral Presentation
Number:
1945
Disease area:
Airway diseases
Rating:
You must
login
to grade this presentation.
Share or cite this content
Citations should be made in the following way:
I. Bernardo (Bundoora, Australia), S. Passey (Bundoora, Australia), H. Seow (Bundoora, Australia), S. Bozinovski (Bundoora, Australia), R. Vlahos (Bundoora, Australia). Targeting NADPH oxidase-2 reduces cigarette smoke-induced lung inflammation and skeletal muscle wasting in mice. 1945
You must
login
to share this Presentation/Article on Twitter, Facebook, LinkedIn or by email.
Member's Comments
No comment yet.
You must
Login
to comment this presentation.
Related content which might interest you:
Late Breaking Abstract - Implications of treatable traits and treatment choices on exacerbation risk in moderate-severe asthma
Management of Severe Asthma in Pediatric Patients by an Interdisciplinary Team in a Public Hospital Setting.
Respiratory sequelae of preterm birth across the life span
Related content which might interest you:
Inhibition of NOX2 oxidase activity ameliorates cigarette smoke-induced lung inflammation and wasting in mice
Source: International Congress 2016 – Novel insights into the treatment of COPD
Year: 2016
Ninjin-yoeito ameliorates cigarette smoke-induced skeletal muscle complications in mice
Source: Virtual Congress 2020 – Clinical and basic pharmacology for COPD
Year: 2020
Irisin secreted from muscle by exercise ameliorates emphysema of cigarette smoke-induced COPD in mice.
Source: International Congress 2019 – New molecular signatures in COPD, lung cancer and a1-antitrypsin deficiency
Year: 2019
Disruption of pulmonary lipid homeostasis drives cigarette smoke-induced lung inflammation in mice
Source: Eur Respir J 2015; 46: 1451-1460
Year: 2015
Transgenic expression of human glutathione peroxidase-1 prevents cigarette smoke-induced inflammation and emphysema formation in mice
Source: Eur Respir J 2007; 30: Suppl. 51, 133s
Year: 2007
Mitochondria-targeted H2S suppresses and reverses cigarette smoke-induced inflammasome activity and lung injury in experimental COPD
Source: ERS Lung Science Conference 2020
Year: 2020
P2RX4 signalling enhances acute cigarette smoke-induced lung inflammation and chronic lung emphysema development
Source: International Congress 2019 – Airway inflammation: pharmacological mechanisms and novel targets
Year: 2019
LPS accelerates cigarette smoke-induced deterioration in respiratory function and airway inflammation in mice
Source: Annual Congress 2007 - New treatments for asthma and COPD - experimental evidence
Year: 2007
Increased autophagy and reduced protein synthesis signaling are involved in pulmonary inflammation-induced skeletal muscle atrophy
Source: International Congress 2016 – How the understanding og molecular and genomic crosstalk is helping to diagnose lung disease
Year: 2016
Ablation of ADAM17 protects against cigarette smoke-induced emphysema in mice
Source: International Congress 2019 – Effects of environmental exposures in lung pathology and the underlying mechanisms
Year: 2019
Deficiency in neutrophil serine proteases protects mice from the development of cigarette smoke-induced emphysema
Source: Annual Congress 2010 - Proteases and the matrix
Year: 2010
PDE4 inhibition prevents corticosteroid-resistant lung inflammation induced by cigarette smoke exposure in mice
Source: Annual Congress 2009 - Highlighting the epithelial cell in lung disease: from COPD to cancer to acute lung injury
Year: 2009
Characterisation of the pathways in cigarette smoke induced skeletal muscle atrophy and regeneration after cessation in mice
Source: Annual Congress 2008 - COPD
Year: 2008
Ceramide-1-phosphate inhibits cigarette smoke-induced airway inflammation
Source: Eur Respir J 2015; 45: 1669-1680
Year: 2015
Quercetin attenuates inflammation in cigarette smoke stimulated airway epithelial cells: Possible involvement of autophagy
Source: Annual Congress 2013 –New drugs in respiratory medicine
Year: 2013
Effects of inflammation on skeletal muscle
Source: ISSN=1025-448x, ISBN=1-904097-27-8, page=68
Year: 2003
Neutrophil elastase contributes to activated human neutrophil-induced rat small airway dysfunction
Source: International Congress 2015 – Translational studies in airway cell biology
Year: 2015
Caspase-7 knockout mice are not protected against cigarette smoke-induced inflammation
Source: Annual Congress 2010 - Novel mechanisms in COPD
Year: 2010
Lack of RTP801, a suppressor of the mTOR pathway, attenuates cigarette smoke-induced pulmonary injuries in mice
Source: Eur Respir J 2007; 30: Suppl. 51, 140s
Year: 2007
Attenuation of lung inflammation and emphysema induced by cigarette smoke exposure in CXCR3 knockout mice
Source: Annual Congress 2008 - Cell biology of asthma and COPD
Year: 2008
We use cookies on our website to give you the most relevant experience by remembering your preferences and repeat visits. By clicking "Accept", you consent to the use of the cookies.
Accept