Source: Eur Respir J 2001; 18: 139-145
Disease area: Airway diseases



Rating:

Share or cite this content

Citations should be made in the following way:
Silvestri M., Fregonese L., Sabatini F., Dasic G., Rossi G.A.. Fluticasone and salmeterol downregulate in vitro, fibroblast proliferation and ICAM-1 or H-CAM expression. Eur Respir J 2001; 18: 139-145

Member's Comments

No comment yet.

Related content which might interest you: Proliferation is not increased in airway myofibroblasts isolated from asthmatics Source: Eur Respir J 2008; 32: 362-371 Year: 2008 Inhibitory activities of fluticasone propionate and salmeterol on airway epithelial cell functions (FLIC24): adhesion molecule expression and cytokine release Source: Eur Respir J 2001; 18: Suppl. 33, 246s Year: 2001 Effect of beclomethasone dipropionate, formoterol and their combination on TNF-α-induced ICAM-1 and IL-8 expression in human bronchial epithelial cells Source: Annual Congress 2012 - Molecular biology of pro- and anti-inflammatory responses in the lung Year: 2012 Allergen-induced airway decorin production is inhibited by budesonide and formoterol in a mouse model of asthma Source: Eur Respir J 2004; 24: Suppl. 48, 690s Year: 2004 Tiotropium and fluticasone inhibit rhinovirus-induced mucin production via multiple mechanisms in differentiated airway epithelial cells Source: Virtual Congress 2020 – Host-microbe interactions and their influence on lung disease onset and severity Year: 2020 Nasal fibroblast functions involved in airway inflammation and remodelling are downregulated by fluticasone propionate (FP) Source: Eur Respir J 2001; 18: Suppl. 33, 147s Year: 2001 Fluticasone induces apoptosis in peripheral T-lymphocytes: a comparison between asthmatic and normal subjects Source: Eur Respir J 2002; 19: 257-266 Year: 2002 Fluticasone propionate decreases MUC5AC expression on airway epithelial cells induced by rhinovirus infection in the airway Source: Annual Congress 2011 - Experimental modulation of airway inflammation Year: 2011 Synergistic anti-inflammatory effects of salmeterol and fluticasone on the release of IL-8 and MCP-1 by human bronchial epithelial cells Source: Eur Respir J 2003; 22: Suppl. 45, 39s Year: 2003 Inhibition of IL-8 and RANTES release from cultured human airway smooth muscle cells by fluticasone and budesonide Source: Eur Respir J 2002; 20: Suppl. 38, 384s Year: 2002 Formoterol and salmeterol enhance cytokine release in primary bronchial epithelial cells Source: Eur Respir J 2004; 24: Suppl. 48, 101s Year: 2004 IL-8 and TSLP production as markers of bronchial epithelial cell activation in IL-17A mediated airway inflammation in COPD: Role of tiotropium Source: Annual Congress 2013 –New insights into epigenetic mechanisms in the lung Year: 2013 Effects of formoterol and salmeterol on the production of Th1- and Th2-related chemokines by monocytes and bronchial epithelial cells Source: Eur Respir J 2008; 31: 1313-1321 Year: 2008 Inhibition of allergen-induced airway remodelling by tiotropium and budesonide: a comparison Source: Eur Respir J 2007; 30: 653-661 Year: 2007 Inhaled budesonide induces corticosteroid-dependent gene expression in asthmatics: Validation in primary epithelial and airways smooth muscle cells Source: Annual Congress 2011 - Models of disease and drug actions Year: 2011 The inhibitory effect of formoterol and salmeterol on the IP-10 (CXCL10) in bronchial epithelial cells Source: Annual Congress 2009 - Models of chronic airways disease and therapy Year: 2009 Downregulation of TNF-α induced E-selectin expression by beclomethasone dipropionate and formoterol in human endothelial cells Source: Annual Congress 2010 - Lung cell biology and immunology Year: 2010 E-cadherin gene polymorphisms in asthma patients using inhaled corticosteroids Source: Eur Respir J 2011; 38: 1044-1052 Year: 2011 Inhibitory activity of fluticasone propionate (FP), on the constitutive or cytokine-induced bronchial epithelial cell (HBECs) functions in vitro Source: Eur Respir J 2003; 22: Suppl. 45, 76s Year: 2003 Formoterol reduces asthmatic airway smooth muscle cell proliferation through p27 (Kip) which is supported by steroids Source: Annual Congress 2012 - The best of pharmacology treatment for asthma and COPD Year: 2012