Different Th1, Th2, Th17 and Treg associated chemokine local pattern expression in lung in a murine model of allergic inflammation Source: Annual Congress 2011 - T-cell subsets Year: 2011
Aerobic training decreases perivascular inflammation and remodeling in a murine model of chronic allergic lung inflammation Source: Annual Congress 2007 - Towards an understanding of asthma pathogenesis - novel in vitro assays and results from animal models Year: 2007
CCL6 produced by eosinophils drives allergic airway inflammation in mice Source: International Congress 2019 – Modelling and monitoring of airway diseases Year: 2019
Aerobic training modulates parenquimal inflammation via decreases of IL-4 and IL-5 release by inflammatory cells Source: Annual Congress 2007 - Towards an understanding of asthma pathogenesis - novel in vitro assays and results from animal models Year: 2007
Transgenic mice overexpressing IL-18 display increased IL-13 in the airways but no changes of the airway hyperresponsiveness in a murine model of late-phase asthma: evidence for a dual role of IL-18 on mucosal Th1/Th2 responses Source: Eur Respir J 2002; 20: Suppl. 38, 1s Year: 2002
Interferon-γ-pretreated dendritic cells induced dual Th1 and Th2 responses and failed to prevent development of allergic airway inflammation Source: Eur Respir J 2006; 28: Suppl. 50, 230s Year: 2006
Smooth-muscle-derived WNT-5A drives allergen-induced remodelling and Th2 type inflammation Source: International Congress 2018 – Preclinical pharmacology: novel targets in airway inflammation, narrowing and neural control Year: 2018
High-level expression of PD-L1 by DC suppresses airway inflammation in OVA-induced allergic asthma Source: Annual Congress 2010 - Animal models of asthma and lung inflammation Year: 2010
B cells in different stages of development increase in the lung in a murine model of airway allergic inflammation Source: Annual Congress 2009 - Animal models of airways inflammation Year: 2009
Aerobic exercise reduces allergic airway inflammation and remodeling by deactivating airway epithelial cells and leukocytes Source: Annual Congress 2011 - Asthma: basic science and clinical studies Year: 2011
Interferon-gamma (IFN-γ) alters the expression of IP-10 and IL-18 in human aveolar epithelial cells type II. Possible role in the reinforcement of Th1-dominated lung inflammation Source: Eur Respir J 2001; 18: Suppl. 33, 521s Year: 2001
LSC Abstract – Role For IL-1alpha in viral-induced inflammatory responses in an epithelial-fibroblast co-culture model of the airway mucosa Source: International Congress 2015 – Viral infections: basic research and clinical perspective Year: 2015
Airway epithelial cell P2Y6R-Expression is up-regulated during acute and chronic allergic inflammation and contributes via modulating of CXC8 and IL-6 secretion to development of airway remodelling Source: Annual Congress 2010 - Pre-clinical models of airways disease Year: 2010
IL-8 and TSLP production as markers of bronchial epithelial cell activation in IL-17A mediated airway inflammation in COPD: Role of tiotropium Source: Annual Congress 2013 –New insights into epigenetic mechanisms in the lung Year: 2013
Role of CCL21 and CCL19 in allergic inflammation examined by murine asthmatic model Source: Annual Congress 2006 - Immunology of the airways Year: 2006
Chemokines induced in human lung epithelial cells by inflammatory cytokines Source: Eur Respir J 2006; 28: Suppl. 50, 748s Year: 2006
Regulatory role of antigen-induced IL-10, produced by Tr1 cells, in airway neutrophilia in a murine model for asthma Source: Annual Congress 2012 - Asthma: mechanisms of airway inflammation Year: 2012
Airways inflammation and remodeling is inhibited by aerobic exercise via decreases of IL-4 and IL-5 release by inflammatory cells Source: Annual Congress 2007 - Towards an understanding of asthma pathogenesis - novel in vitro assays and results from animal models Year: 2007
CD40 ligation modulates inflammatory cytokines production by human bronchial epithelial cells Source: Eur Respir J 2001; 18: Suppl. 33, 246s Year: 2001