Lipopolysaccharide (LPS) inhalation causes an increase in mucosal CD8+ve cells Source: Eur Respir J 2001; 18: Suppl. 33, 316s Year: 2001
TLR-4 downregulation in COPD impairs cytokine release from monocytes and Th1-lymphocytes in response to bacterial endotoxin Source: Annual Congress 2009 - What is new in molecular pathology and functional genomics of neoplastic and non-neoplastic lung disease? Year: 2009
Toll-like receptor mRNA levels in alveolar macrophages after inhalation of endotoxin Source: Eur Respir J 2006; 28: 622-626 Year: 2006
Oxidative burst in lipopolysaccharide-activated human alveolar macrophages is inhibited by interleukin-9 Source: Eur Respir J 2002; 20: 1198-1205 Year: 2002
IL-17, produced by lymphocytes and neutrophils, is necessary for lipopolysaccharide- but not allergen-induced airway neutrophilia Source: Eur Respir J 2002; 20: Suppl. 38, 618s Year: 2002
TLR2 and TLR4 induced tolerance in alveolar macrophages; differential effect on TNFα and IL-8 release Source: Annual Congress 2012 - Cellular signalling mechanisms in the airways Year: 2012
α1-Antitrypsin (α1-AT) instillation decreases neutrophil influx and increases cytokine levels in the BAL of LPS-treated mice Source: Annual Congress 2008 - Understanding allergic airway inflammation with the help of animal models Year: 2008
Inhaled CCR1 antagonist AZD4818 reduces leucocyte influx in the rat lipopolysaccharide (LPS) lung inflammation model Source: Annual Congress 2009 - Preclinical models in drug development Year: 2009
Effects of HSP70 induction on IL-6 production from murine alveolar epithelial cells pre-treated with endotoxine after subsequent endotoxin challenge Source: Eur Respir J 2005; 26: Suppl. 49, 346s Year: 2005
Increased expression of microRNA-146a negatively regulates both the LPS and Poly (I:C) induced inflammatory response in monocytes Source: Annual Congress 2010 - Macrophages: cell biology and disease-specific characteristics Year: 2010
A novel subpopulation of monocyte-like cells in the human lung after lipopolysaccharide inhalation Source: Eur Respir J 2012; 40: 206-214 Year: 2012
Differential properties of mucosubstance produced by allergic inflammation and lipopolysaccharide (LPS) stimulation in rat nasal epithelium Source: Eur Respir J 2002; 20: Suppl. 38, 482s Year: 2002
Effect of lipopolysaccharide (LPS) priming on Candida -induced acute lung injury (ALI) Source: Eur Respir J 2003; 22: Suppl. 45, 585s Year: 2003
Endotoxin-induced IL-17 release by murine T-lymphocytes: interactions with airway macrophages Source: Eur Respir J 2001; 18: Suppl. 33, 408s Year: 2001
Myeloperoxidase -/- mice show decreased lung neutrophilia and increased epithelial cell proliferation after lipopolysaccharide or asbestos exposure Source: Eur Respir J 2006; 28: Suppl. 50, 398s Year: 2006
Glucocorticoid sensitivity of LPS stimulated COPD alveolar macrophages Source: Annual Congress 2009 - COPD: basic science and clinical studies Year: 2009
Induced sputum, but not BALF stimulated PMN ROS production is enhanced by ionomycin during COPD Source: Annual Congress 2010 - COPD and asthma: models and human studies Year: 2010
Increased cytokine production by peripheral blood mononuclear cells (PBMC) and alveolar macrophages (AM) in active sarcoidosis Source: Eur Respir J 2003; 22: Suppl. 45, 177s Year: 2003
IgA inhibits LPS-induced IL-10 in human alveolar macrophages (HAM) Source: Eur Respir J 2005; 26: Suppl. 49, 585s Year: 2005
Trans-basement membrane migration of eosinophils induced by LPS-stimulated neutrophils from human peripheral blood in vitro Source: ERJ Open Res 2015; 1: 00003-2015 Year: 2015