e-learning
resources
Amsterdam 2015
Sunday, 27.09.2015
Translational studies in airway cell biology
Login
Search all ERS
e-learning
resources
Disease Areas
Airways Diseases
Interstitial Lung Diseases
Respiratory Critical Care
Respiratory Infections
Paediatric Respiratory Diseases
Pulmonary Vascular Diseases
Sleep and Breathing Disorders
Thoracic Oncology
Events
International Congress
Courses
Webinars
Conferences
Research Seminars
Journal Clubs
Publications
Breathe
Monograph
ERJ
ERJ Open Research
ERR
European Lung White Book
Handbook Series
Guidelines
All ERS guidelines
e-learning
CME Online
Case reports
Short Videos
SpirXpert
Procedure Videos
CME tests
Reference Database of Respiratory Sounds
Radiology Image Challenge
Brief tobacco interventions
EU Projects
VALUE-Dx
ERN-LUNG
ECRAID
UNITE4TB
Disease Areas
Events
Publications
Guidelines
e-learning
EU Projects
Login
Search
Neutrophil elastase contributes to activated human neutrophil-induced rat small airway dysfunction
Natasha Madge (Kent, United Kingdom), Natasha Madge, Louise Donnelly, Duncan Rogers
Source:
International Congress 2015 – Translational studies in airway cell biology
Session:
Translational studies in airway cell biology
Session type:
Thematic Poster Session
Number:
913
Disease area:
Airway diseases
Rating:
You must
login
to grade this presentation.
Share or cite this content
Citations should be made in the following way:
Natasha Madge (Kent, United Kingdom), Natasha Madge, Louise Donnelly, Duncan Rogers. Neutrophil elastase contributes to activated human neutrophil-induced rat small airway dysfunction. Eur Respir J 2015; 46: Suppl. 59, 913
You must
login
to share this Presentation/Article on Twitter, Facebook, LinkedIn or by email.
Member's Comments
No comment yet.
You must
Login
to comment this presentation.
Related content which might interest you:
From pulmonary gas exchange to the interpretation of arterial blood gases
The physiology of breathlessness
Panel discussion on ERS Statement – A core outcome set for clinical trials evaluating the management of COPD exacerbations
Related content which might interest you:
Glucagon inhibits airway hyperreactivity and lung inflammation in a murine model of acute lung injury
Source: International Congress 2015 – Therapeutic approaches in inflammatory lung disease
Year: 2015
Plasminogen-stimulated inflammatory cytokine production by airway smooth muscle cells is regulated by annexin A2
Source: Annual Congress 2013 –Airway smooth muscle and cell biology
Year: 2013
Contribution of bronchial smooth muscle to inflammation
Source: Annual Congress 2005 - Role of the bronchial smooth muscle in regulating airway disease
Year: 2005
Responses of bronchial smooth muscle to inflammation and disease
Source: Annual Congress 2005 - Role of the bronchial smooth muscle in regulating airway disease
Year: 2005
IL-13 induces glucocorticoid-insensitive hyperreactivity of human small airways
Source: International Congress 2015 – Modulating the immune system in COPD and asthma
Year: 2015
CCL2 release by airway smooth muscle is increased in asthma and promotes fibrocyte migration
Source: International Congress 2014 – Cell biology 2014
Year: 2014
Targeting ASK1 in preventing airway smooth muscle growth: Implications for airway remodeling in COPD
Source: International Congress 2016 – New findings in mucosal immunology
Year: 2016
Link between airway inflammation and remodeling: TNFα induces airway smooth muscle cell proliferation via an ET1/GMCSF/IL6 network
Source: International Congress 2015 – Airway remodelling: recent developments
Year: 2015
Endothelin receptor B protects GM-CSF mRNA from degradation in human airway smooth muscle cells
Source: International Congress 2015 – Immunomodulation: basic science and clinical aspects
Year: 2015
Effect of oxidative stress on mitochondrial function in airway smooth muscle (ASM) cells from COPD patients
Source: Annual Congress 2013 –Airway smooth muscle and cell biology
Year: 2013
Mitochondrial reactive oxygen species (ROS) mediate proliferation and cytokine release in airway smooth muscle cells of patients with COPD
Source: International Congress 2014 – Cell biology 2014
Year: 2014
Eosinophil-induced airway smooth muscle cells remodeling is attenuated by blocking of RGD-binding integrins in asthma
Source: International Congress 2016 – New findings in mucosal immunology
Year: 2016
MNK-1 inhibition reduces proliferation and CXCL10 in airway smooth muscle cells
Source: Annual Congress 2013 –Airway smooth muscle and cell biology
Year: 2013
Integrins regulate eosinophil adhesion to the airway smooth muscle cells in asthma patients
Source: International Congress 2015 – Asthma mechanisms and management
Year: 2015
Rhinovirus-induced IL-33 expression in asthmatic airway smooth muscle cells is TLR3-dependent and involves activation of TAK1
Source: International Congress 2016 – Emerging concepts in lung disease pathogenesis
Year: 2016
Mevastatin ameliorates sphingosine 1-phosphate-induced inflammation in human tracheal smooth muscle cells
Source: International Congress 2016 – Preclinical pharmacology: drug targets and mechanisms of action
Year: 2016
Role of apoptosis in airway smooth muscle remodeling in experimental asthma
Source: Eur Respir J 2007; 30: Suppl. 51, 369s
Year: 2007
Mechanisms of bronchial hyperresponsiveness in a clinical setting – putative role of smooth muscle
Source: Annual Congress 2005 - Role of the bronchial smooth muscle in regulating airway disease
Year: 2005
Syk mediates airway contractility independently of leukocyte function
Source: Annual Congress 2013 –Murine models of lung immunology
Year: 2013
Impaired inhibitory action of corticosteroids on chemokine expression induced by TNFalpha in airway smooth muscle (ASM) cells from patients with severe asthma
Source: Annual Congress 2013 –Airway smooth muscle and cell biology
Year: 2013
We use cookies on our website to give you the most relevant experience by remembering your preferences and repeat visits. By clicking "Accept", you consent to the use of the cookies.
Accept