e-learning
resources
Munich 2014
Sunday, 07.09.2014
Respiratory physiology and sleep disordered breathing in children
Login
Search all ERS
e-learning
resources
Disease Areas
Airways Diseases
Interstitial Lung Diseases
Respiratory Critical Care
Respiratory Infections
Paediatric Respiratory Diseases
Pulmonary Vascular Diseases
Sleep and Breathing Disorders
Thoracic Oncology
Events
International Congress
Courses
Webinars
Conferences
Research Seminars
Journal Clubs
Publications
Breathe
Monograph
ERJ
ERJ Open Research
ERR
European Lung White Book
Handbook Series
Guidelines
All ERS guidelines
e-learning
CME Online
Case reports
Short Videos
SpirXpert
Procedure Videos
CME tests
Reference Database of Respiratory Sounds
Radiology Image Challenge
Brief tobacco interventions
EU Projects
VALUE-Dx
ERN-LUNG
ECRAID
UNITE4TB
Disease Areas
Events
Publications
Guidelines
e-learning
EU Projects
Login
Search
Low catalase activity contributes to oxidative stress in patients with alpha-1 antitrypsin deficiency
A. Escribano, M. Amor, S. Castillo, F. Sanz, S. Pastor, P. Codoñer-Franch, F. Dasí (Valencia, Spain)
Source:
International Congress 2014 – Respiratory physiology and sleep disordered breathing in children
Session:
Respiratory physiology and sleep disordered breathing in children
Session type:
Thematic Poster Session
Number:
1150
Disease area:
Paediatric lung diseases, Sleep and breathing disorders
Rating:
You must
login
to grade this presentation.
Share or cite this content
Citations should be made in the following way:
A. Escribano, M. Amor, S. Castillo, F. Sanz, S. Pastor, P. Codoñer-Franch, F. Dasí (Valencia, Spain). Low catalase activity contributes to oxidative stress in patients with alpha-1 antitrypsin deficiency. Eur Respir J 2014; 44: Suppl. 58, 1150
You must
login
to share this Presentation/Article on Twitter, Facebook, LinkedIn or by email.
Member's Comments
No comment yet.
You must
Login
to comment this presentation.
Related content which might interest you:
Panel discussion: respiratory care for newborns in resource-limited settings
From pulmonary gas exchange to the interpretation of arterial blood gases
Observational cohort study of pulmonary exacerbations in alpha-1 antitrypsin deficiency
Related content which might interest you:
Circulating microRNAs as potential biomarkers in alpha-1 antitrypsin deficiency patients
Source: International Congress 2014 – Molecular pathogenesis of pulmonary inflammatory diseases (COPD, asthma and ILD)
Year: 2014
Total oxidative stress, total antioxidant status and paraoxanase activity in pediatric patients with bronchiolitis obliterans
Source: International Congress 2014 – Respiratory infections in children
Year: 2014
Serum paraoxonase activity and oxidative status in pediatric patients with pulmonary tuberculosis
Source: International Congress 2014 – Respiratory infections in children
Year: 2014
Oxidative stress in serum of patients with alpha-1 antitrypsin deficiency
Source: Annual Congress 2013 –New perspectives of lung function assessment in children
Year: 2013
Oxidative stress impairs mitochondrial function and leads to deficient antiviral responses in primary bronchial epithelial cells
Source: International Congress 2014 – Paediatric asthma: markers and molecules
Year: 2014
Lung function decline in patients with severe alpha
1
-antitrypsin deficiency
Source: International Congress 2014 – Respiratory function: quality and new technologies
Year: 2014
Specific airway resistance as a marker of early disease in alpha-1 antitrypsin deficiency
Source: International Congress 2015 – Lung function: exploring the boundaries of the respiratory system
Year: 2015
Analysis of the level of cytokines in blood serum and condensate in children with bronchopulmonary pathology
Source: International Congress 2014 – Respiratory infections in children
Year: 2014
Cluster analysis and lung function in sickle cell disease
Source: International Congress 2016 – Lung function and imaging: new findings and new approaches
Year: 2016
F alpha-1 antitrypsin polymerization activates endoplasmic reticulum overload response
Source: International Congress 2014 – Mechanisms of COPD: proteases and a whole lot more
Year: 2014
Signs of oxidative stress in bronchial asthma
Source: Annual Congress 2013 –The multiple faces of stress in lung injury
Year: 2013
Role of IL-1beta in vagal mediated control of respiration
Source: International Congress 2014 – Paediatric respiratory physiology in health and disease
Year: 2014
Evaluation of protease-antiprotease imbalance in moderate COPD without antitrypsin deficiency
Source: International Congress 2015 – Modulating the immune system in COPD and asthma
Year: 2015
Oxidative stress in TGF-β1-mediated fibrosis in Z alpha-1 antitrypsin cells
Source: International Congress 2016 – Novel insights into alveolar and bronchial epithelial cell injury and repair
Year: 2016
Interactions between antioxidant gene variants and dietary antioxidant intake for asthma outcomes in children – Food for thought?
Source: International Congress 2014 – Paediatric epidemiology: wheeze, asthma and other respiratory diseases
Year: 2014
Development of a myeloid cell-binding adenovirus for the potential gene therapy of alpha-1 anti-trypsin deficiency
Source: International Congress 2014 – Translational studies in COPD
Year: 2014
Similar adaptive immune response in COPD patients with and without alpha1 antitrypsin deficiency
Source: Annual Congress 2013 –COPD: new markers of diseases
Year: 2013
Modulation of respiration by tissue oxygen: A study in the mutant mouse with presbyterian hemoglobinopathy
Source: Annual Congress 2013 –Pulmonary circulation: basic science
Year: 2013
Lung function in Indian children with vertically acquired HIV
Source: International Congress 2014 – Respiratory infections in children
Year: 2014
Vitamin D status and lung function in children with asthma
Source: International Congress 2014 – More paediatric asthma
Year: 2014
We use cookies on our website to give you the most relevant experience by remembering your preferences and repeat visits. By clicking "Accept", you consent to the use of the cookies.
Accept