e-learning
resources
Munich 2014
Monday, 08.09.2014
Mechanisms of COPD: proteases and a whole lot more
Login
Search all ERS
e-learning
resources
Disease Areas
Airways Diseases
Interstitial Lung Diseases
Respiratory Critical Care
Respiratory Infections
Paediatric Respiratory Diseases
Pulmonary Vascular Diseases
Sleep and Breathing Disorders
Thoracic Oncology
Events
International Congress
Courses
Webinars
Conferences
Research Seminars
Journal Clubs
Publications
Breathe
Monograph
ERJ
ERJ Open Research
ERR
European Lung White Book
Handbook Series
Guidelines
All ERS guidelines
e-learning
CME Online
Case reports
Short Videos
SpirXpert
Procedure Videos
CME tests
Reference Database of Respiratory Sounds
Radiology Image Challenge
Brief tobacco interventions
EU Projects
VALUE-Dx
ERN-LUNG
ECRAID
UNITE4TB
Disease Areas
Events
Publications
Guidelines
e-learning
EU Projects
Login
Search
Pathogenic potential of an autoprocessed form of neutrophil elastase
T. Dau, R. Sarker, A. O. Yildirim, O. Eickelberg, D. Jenne (Munich, Germany)
Source:
International Congress 2014 – Mechanisms of COPD: proteases and a whole lot more
Session:
Mechanisms of COPD: proteases and a whole lot more
Session type:
Poster Discussion
Number:
1839
Disease area:
Airway diseases
Rating:
You must
login
to grade this presentation.
Share or cite this content
Citations should be made in the following way:
T. Dau, R. Sarker, A. O. Yildirim, O. Eickelberg, D. Jenne (Munich, Germany). Pathogenic potential of an autoprocessed form of neutrophil elastase. Eur Respir J 2014; 44: Suppl. 58, 1839
You must
login
to share this Presentation/Article on Twitter, Facebook, LinkedIn or by email.
Member's Comments
No comment yet.
You must
Login
to comment this presentation.
Related content which might interest you:
Panel discussion on ERS Statement – A core outcome set for clinical trials evaluating the management of COPD exacerbations
ERS statement: a core outcome set for clinical trials evaluating the management of COPD exacerbations
Evidence-based eHealth for COPD
Related content which might interest you:
NSP4 is an elastase-related protease with arginine specificity that is released by activated human neutrophils as an active endoprotease
Source: International Congress 2014 – Mechanisms of COPD: proteases and a whole lot more
Year: 2014
Neutrophil elastase inhibitor sivelestat ameliorates gefitinib-induced acute pneumonitis in mice
Source: International Congress 2014 – Experimental models in acute lung injury
Year: 2014
Rapid activation of pulmonary endothelial cells during high stretch ventilation
in vivo
Source: International Congress 2014 – Acute lung injury: hitting the barriers
Year: 2014
Effects of extracellular vesicles from mesenchymal stromal cells in experimental silicosis
Source: International Congress 2014 – Pulmonary fibrosis: receptors, signalling pathways and mesenchymal cells
Year: 2014
Injury and repair responses of parenchymal fibroblasts in advanced chronic obstructive pulmonary disease
Source: International Congress 2014 – Mechanisms of COPD: proteases and a whole lot more
Year: 2014
Neutrophil elastase degraded elastin is elevated in serum of idiopathic pulmonary fibrosis
Source: International Congress 2014 – New and old players in fibrotic lung disease
Year: 2014
Essential oils reduced lung inflammation in a model of acute lung injury
Source: International Congress 2014 – Experimental models in acute lung injury
Year: 2014
Wnt-heparan sulfate proteoglycan interaction in fibrotic lung disease
Source: International Congress 2014 – New and old players in fibrotic lung disease
Year: 2014
Impact of Wnt ligand secretion on cellular phenotypes in pulmonary fibrosis
Source: International Congress 2014 – New and old players in fibrotic lung disease
Year: 2014
LSC 2014 abstract - Influenza infection may cause exacerbation of lung fibrosis through collagen deposition
Source: International Congress 2014 – Pulmonary fibrosis: receptors, signalling pathways and mesenchymal cells
Year: 2014
Matriptase triggers pleiotropic functional responses in normal human lung fibroblasts (NHLF): Potential relevance in pathophysiology
Source: International Congress 2014 – Lung cell biology in injury and repair
Year: 2014
CD39 deficiency results in increased inflammation in an animal model of pulmonary fibrosis
Source: International Congress 2014 – New and old players in fibrotic lung disease
Year: 2014
The role of CD74 in experimental and clinical acute lung injury
Source: International Congress 2014 – Experimental models in acute lung injury
Year: 2014
Polarization of alveolar macrophages is regulated by the immunoproteasome
Source: International Congress 2014 – Lung cell biology in injury and repair
Year: 2014
Ventilator-Induced Lung Injury Increases Systemic Inflammation in Murine Pseudomonas Aeruginosa Pneumonia
Source: International Congress 2019 – Severe pneumonia: epidemiology, mechanisms and treatment
Year: 2019
Inflammatory Pathways and Mediators in the Pathogenesis of Severe Asthma
Source: Research Seminar 2007 - Severe Asthma
Year: 2007
Feasibility of measuring sputum rheology in COPD
Source: International Congress 2014 – Mechanisms of COPD and inhalation injury
Year: 2014
Proteasome function in pulmonary fibrosis
Source: International Congress 2014 – Pulmonary fibrosis: receptors, signalling pathways and mesenchymal cells
Year: 2014
LATE-BREAKING ABSTRACT: Trasplantation of alveolar type II cells in experimental Acute Lung Injury
Source: International Congress 2014 – Lung cell biology in injury and repair
Year: 2014
Regulation of WISP1 by profibrotic cytokines in pulmonary fibrosis
Source: International Congress 2014 – Pulmonary fibrosis: receptors, signalling pathways and mesenchymal cells
Year: 2014
We use cookies on our website to give you the most relevant experience by remembering your preferences and repeat visits. By clicking "Accept", you consent to the use of the cookies.
Accept