e-learning
resources
Barcelona 2013
Tuesday, 10.09.2013
Cell signalling and inflammation: what's new in 2013?
Login
Search all ERS
e-learning
resources
Disease Areas
Airways Diseases
Interstitial Lung Diseases
Respiratory Critical Care
Respiratory Infections
Paediatric Respiratory Diseases
Pulmonary Vascular Diseases
Sleep and Breathing Disorders
Thoracic Oncology
Events
International Congress
Courses
Webinars
Conferences
Research Seminars
Journal Clubs
Publications
Breathe
Monograph
ERJ
ERJ Open Research
ERR
European Lung White Book
Handbook Series
Guidelines
All ERS guidelines
e-learning
CME Online
Case reports
Short Videos
SpirXpert
Procedure Videos
CME tests
Reference Database of Respiratory Sounds
Radiology Image Challenge
Brief tobacco interventions
EU Projects
VALUE-Dx
ERN-LUNG
ECRAID
UNITE4TB
Disease Areas
Events
Publications
Guidelines
e-learning
EU Projects
Login
Search
Silica-induced inflammasome activation
in vitro
and in rat lungs
P. Peeters, N. Reynaert, I. Eurlings, T. Perkins, B. Mossman, R. Schins, E. Wouters, C. Albrecht (Maastricht, Netherlands; Burlington, United States Of America; Düsseldorf, Germany)
Source:
Annual Congress 2013 –Cell signalling and inflammation: what's new in 2013?
Session:
Cell signalling and inflammation: what's new in 2013?
Session type:
Poster Discussion
Number:
4743
Disease area:
Interstitial lung diseases
Rating:
You must
login
to grade this presentation.
Share or cite this content
Citations should be made in the following way:
P. Peeters, N. Reynaert, I. Eurlings, T. Perkins, B. Mossman, R. Schins, E. Wouters, C. Albrecht (Maastricht, Netherlands; Burlington, United States Of America; Düsseldorf, Germany). Silica-induced inflammasome activation
in vitro
and in rat lungs. Eur Respir J 2013; 42: Suppl. 57, 4743
You must
login
to share this Presentation/Article on Twitter, Facebook, LinkedIn or by email.
Member's Comments
No comment yet.
You must
Login
to comment this presentation.
Related content which might interest you:
Panel discussion: ANCA associated vasculitis and lung disease
Panel discussion on genetics in pulmonary fibrosis
Late Breaking Abstract - Implications of treatable traits and treatment choices on exacerbation risk in moderate-severe asthma
Related content which might interest you:
KCa3.1 channel inhibition prevents fibrotic responses in a human lung explant model
Source: International Congress 2015 – Pulmonary fibrosis: mechanisms of disease
Year: 2015
A role of Foxc2 gene in mice with bleomycin-induced pulmonary fibrosis
Source: International Congress 2015 – Pulmonary fibrosis: mechanisms of disease
Year: 2015
Aquaporin levels in murine lung injury
Source: International Congress 2016 – Novel insights into alveolar and bronchial epithelial cell injury and repair
Year: 2016
PAR-2 agonists induce TGF-beta and EMT leading to lung fibrosis
Source: Annual Congress 2013 –Growth factors at the crossroads of acute and chronic inflammation
Year: 2013
Activation of NLRP3 inflammasome is regulated by mitochondrial ROS via PI3K-HIF-VEGF pathway in acute lung injury
Source: International Congress 2015 – Novel insights into the pathobiology of acute lung injury
Year: 2015
Hypoxia induces inflammasome activation and influx of neutrophils and T-cells in the lungs
Source: Annual Congress 2013 –Novel mechanisms in lung injury
Year: 2013
Role of the inflammasome in the pathophysiology of antisynthetase-associated interstitial lung disease.
Source: Virtual Congress 2021 – Clinical evolution, management and outcomes of patients with rare diffuse parenchymal lung diseases
Year: 2021
Lung ageing in C57/Bl6 and BALB/c mice is characterized by a defective surfactant metabolism, increased ER stress and inflammasome induction
Source: International Congress 2016 – Novel insights into alveolar and bronchial epithelial cell injury and repair
Year: 2016
Targeting the hedgehog/GLI pathway decreases bleomycin-induced lung fibrosis
Source: Annual Congress 2013 –Translational studies in chronic repair and remodelling in the lung
Year: 2013
Collagen V activation and matrix extracellular remodeling mediated pulmonary fibrosis in experimental models of bleomycin and 3-5-di-tert-4-hidroxitoluene
Source: Annual Congress 2013 –Growth factors at the crossroads of acute and chronic inflammation
Year: 2013
Role of TNF-a in ventilator-induced lung injury in the developing murine lung
Source: Annual Congress 2013 –The role of hormones and cytokines in lung injury
Year: 2013
Mucus production, lung inflammation and functional decay in IPF
Source: International Congress 2014 – ILDs: pathogenesis and clinical features
Year: 2014
Matriptase triggers pleiotropic functional responses in normal human lung fibroblasts (NHLF): Potential relevance in pathophysiology
Source: International Congress 2014 – Lung cell biology in injury and repair
Year: 2014
FGF-9 overexpression prevents pleural fibrosis induced by intra-pleural adenovirus injection in mice
Source: International Congress 2015 – IPF: from bench to bedside
Year: 2015
SIRT1 contributes to LPS-induced lung injury through interaction with mitochondrial ROS generation
Source: International Congress 2015 – Novel insights into the pathobiology of acute lung injury
Year: 2015
Apoptosis in lung injury and fibrosis
Source: Eur Respir J 2008; 32: 1631-1638
Year: 2008
The enhancement of Stanniocalcin-1 (STC1) secretion increases the ability of mesenchymal stem cells (MSCs) to reduce bleomycin-induced lung fibrosis in a mouse model through inhibition of endoplasmic reticulum stress (ER-stress)/TGFbeta1 pathway
Source: International Congress 2014 – ILDs 1
Year: 2014
Nintedanib prevents IL-1beta-stimulated proliferation of primary human lung fibroblasts from patients with idiopathic pulmonary fibrosis or from control donors
Source: International Congress 2014 – ILDs: pathogenesis and clinical features
Year: 2014
Dexamethasone inhibits secretory and cytosolic phospholipases A2 in alveolar macrophages in acute lung injury
Source: Annual Congress 2013 –Macrophages, mononuclear cells and viral infection
Year: 2013
Inhibition of profibrotic signaling in fibroblasts from patients with idiopathic pulmonary fibrosis (IPF) by histone deacetylase-inhibitors (HDACi) or the IPF drug pirfenidone
Source: International Congress 2014 – ILDs 1
Year: 2014
We use cookies on our website to give you the most relevant experience by remembering your preferences and repeat visits. By clicking "Accept", you consent to the use of the cookies.
Accept