Cigarette smoke condensate inhibits apoptosis and induces cell necrosis Source: Eur Respir J 2002; 20: Suppl. 38, 97s Year: 2002
Cigarette smoke extract inhibits the NLRP3 inflammasome in human macrophages and impairs cell metabolism via NLRP3-independent activation of Caspase-1 Source: International Congress 2019 – Effects of environmental exposures in lung pathology and the underlying mechanisms Year: 2019
Cigarette smoke switches epithelial cell apoptosis into necrosis by inhibition of ATP production through a blockade of the mitochondrial respiratory chain Source: Eur Respir J 2006; 28: Suppl. 50, 605s Year: 2006
Cigarette smoke suppresses mast cell maturation and cytokine release independent of TLR4 signaling Source: Annual Congress 2012 - Basic and translational studies in COPD Year: 2012
Cigarette smoke exposure decreases CFLAR expression in bronchial epithelium, augmenting susceptibility for cell death and DAMP release Source: International Congress 2018 – Regenerative and anti-fibrotic approaches Year: 2018
Cigarette smoke exposure directly induces increased ubiquination and impairs hypoxic-angiogenic signalling in skeletal muscles Source: Annual Congress 2011 - Molecular: pathology of infectious and inflammatory lung disease Year: 2011
Cigarette smoke induce Epithelial-Derived Inflammation through activating autophagy via RAGE signaling in bronchial epithelial cells. Source: International Congress 2019 – Effects of environmental exposures in lung pathology and the underlying mechanisms Year: 2019
RAGE blocking ameliorates cigarette smoke extract-induced human alveolar tpye II epithelial cell injury via RAGE-mediated redox sensitive DAMP signaling through Nrf2 Source: International Congress 2018 – COPD: immune regulation and therapeutic targets Year: 2018
Cigarette smoke induces mitochondrial dysfunction via PKCd-p66Shc signaling pathway in human bronchial epithelial cells Source: International Congress 2018 – COPD: novel pathological mechanisms in the airway epithelium Year: 2018
Cigarette smoke extracts decrease basal and lipopolysaccharide-induced expression of macrophage receptor with collagenous structure via degradation of p300 Source: International Congress 2018 – Disease exacerbations from a molecular point of view Year: 2018
Caspase-1 activation by cigarette smoke occurs via TLR4/TRIF/caspase-8 axis leading to metabolic alterations in human macrophages Source: ERS Lung Science Conference 2020 Year: 2020
Cigarette smoke induces the release of CXCL-8 from human bronchial epithelial cells via TLRs and the induction of inflammasome Source: Annual Congress 2011 - Epithelial cell biology Year: 2011
Cigarette smoke impairs Sirt1 activity and promotes pro-inflammatory responses in bronchial epithelial cells Source: International Congress 2015 – Immunomodulation: basic science and clinical aspects Year: 2015
Cigarette smoke markedly inhibits the LPS-induced production of interleukin-8 in bronchial epithelial cells and consequent neutrophil migration in vitro Source: Eur Respir J 2004; 24: Suppl. 48, 100s Year: 2004
Overexpression of CBP augments cigarette smoke-induced pro-inflammatory cytokine release in macrophages Source: Eur Respir J 2005; 26: Suppl. 49, 105s Year: 2005
Lack of RTP801, a suppressor of the mTOR pathway, attenuates cigarette smoke-induced pulmonary injuries in mice Source: Eur Respir J 2007; 30: Suppl. 51, 140s Year: 2007
Curcumin restores glucocorticoid function and inhibits cigarette smoke-mediated IL-8 release in oxidant stressed monocytic U937 cells Source: Eur Respir J 2005; 26: Suppl. 49, 215s Year: 2005
Cigarette smoke extract induces a multi-phase cell cycle arrest in normal human lung fibroblasts. Ivolvement of MAPK pathway Source: Eur Respir J 2004; 24: Suppl. 48, 106s Year: 2004
Propolis reverts cigarette smoke-induced emphysema through macrophage alternative activation Source: International Congress 2017 – From the bench: therapeutic approaches in COPD and emphysema Year: 2017
Cigarette smoke enhances beta-defensin 2 expression in rat airways via nuclear factor-kappa-B activation Source: Eur Respir J 2010; 36: 638-645 Year: 2010