Source: Annual Congress 2004 - Modulation of the allergic response in animal models
Disease area: Airway diseases



Rating:

Share or cite this content

Citations should be made in the following way:
J. N. Kline, J. V. Weinstock, K. Kitahaki, T. R. Businga (Iowa City, United States Of America). Gastrointestinal colonization by helminths prevents atopic asthma in mice: IL-10 is required. Eur Respir J 2004; 24: Suppl. 48, 165

Member's Comments

No comment yet.

Related content which might interest you: IL-37 requires IL18Rα and SIGIRR to diminish experimental allergic airway inflammation in mice Source: International Congress 2015 – Animal studies of asthma and other inflammatory conditions Year: 2015 Induced sputum levels of soluble receptor ST-2 in adult patients suffering from allergic asthma or allergic rhinitis Source: International Congress 2016 – Laboratory tests and cell biology in allergy and immunology Year: 2016 Late Breaking Abstract - Link between IL-22 and IL-10, IL-13, and severity of symptoms in patients with allergic airway diseases caused by house dust mite Source: Virtual Congress 2020 – New insight into the immunology of asthma and COPD Year: 2020 Improving of experimental allergic asthma by decreased Th2 responses in OGR-1 knockout mice Source: Annual Congress 2010 - Animal models of asthma and lung inflammation Year: 2010 Nonatopic asthma is associated with helminth infections and bronchiolitis in poor children Source: Eur Respir J 2007; 29: 1154-1160 Year: 2007 Prolonged non specific airway hyperresponsiveness in atopic versus non atopic asthmatic children after upper respiratory tract infections Source: Annual Congress 2004 - Mechanisms and treatment of virus induced asthma exacerbations Year: 2004 High-level expression of PD-L1 by DC suppresses airway inflammation in OVA-induced allergic asthma Source: Annual Congress 2010 - Animal models of asthma and lung inflammation Year: 2010 Deficiency of MMP-19 promotes allergen-induced eosinophil burden and airway responsiveness in mice Source: Annual Congress 2007 - Regulation of allergic airway inflammation in animal models of asthma Year: 2007 IL-33 mRNA in induced sputum cells from allergic rhinitis and asthma patients: a marker of Th-2 phenotype status? Source: International Congress 2017 – Monitoring asthma control Year: 2017 Local cytokine profile specifics among patients with BA phenotype, associated to allergic rhinitis: allergic rhinitis and bronchial asthma. Source: International Congress 2018 – Physiological and biological insights in asthma Year: 2018 Immunotherapy prevents the development of asthma in subjects with allergic rhinitis Source: Eur Respir J 2001; 18: Suppl. 33, 482s Year: 2001 Farm dust decreases Th2 driven allergic airway inflammation in mice: A role for airway TLR2 and TLR4? Source: Annual Congress 2011 - T-cell subsets Year: 2011 Prevention of infections in child day-care centres and later development of asthma, allergic rhinitis and atopic dermatitis Source: Annual Congress 2007 - Pets, dust and colds: early life exposures and risk of asthma Year: 2007 The inflammation and the airway hyperresponsiveness in allergic asthma is inhibited by IL-10 dependent mechanism Source: Annual Congress 2007 - Mouse models of allergic airway inflammation Year: 2007 Role of neutrophils in airway inflammation in children with severe persistent asthma Source: Eur Respir J 2002; 20: Suppl. 38, 142s Year: 2002 Interactions between antiviral and atopic inflammatory pathways sustain asthma exacerbations in children Source: Annual Congress 2009 - Infections, immunity and inflammation Year: 2009 IL-33 in patients with severe uncontrolled allergic asthma Source: International Congress 2017 – "Alarming" immunological patterns in asthma Year: 2017 Omalizumab protects against allergen-induced bronchoconstriction in patients with allergic (IgE-mediated) asthma and high baseline IgE levels Source: Annual Congress 2009 - Severe asthma and its treatment Year: 2009 Interactions between respiratory tract infections and atopy in the aetiology of asthma Source: Eur Respir J 2002; 19: 538-545 Year: 2002