e-learning
resources
Barcelona 2010
Sunday, 19.09.2010
Causal factors and mechanisms of outdoor air pollution and nanoparticulates
Login
Search all ERS
e-learning
resources
Disease Areas
Airways Diseases
Interstitial Lung Diseases
Respiratory Critical Care
Respiratory Infections
Paediatric Respiratory Diseases
Pulmonary Vascular Diseases
Sleep and Breathing Disorders
Thoracic Oncology
Events
International Congress
Courses
Webinars
Conferences
Research Seminars
Journal Clubs
Publications
Breathe
Monograph
ERJ
ERJ Open Research
ERR
European Lung White Book
Handbook Series
Guidelines
All ERS guidelines
e-learning
CME Online
Case reports
Short Videos
SpirXpert
Procedure Videos
CME tests
Reference Database of Respiratory Sounds
Radiology Image Challenge
Brief tobacco interventions
EU Projects
VALUE-Dx
ERN-LUNG
ECRAID
UNITE4TB
Disease Areas
Events
Publications
Guidelines
e-learning
EU Projects
Login
Search
Diesel exhaust inhalation causes elevated oxidative burst in airway macrophages and neutrophils that is mitigated by anti-oxidant supplementation
C. Carlsten, M. Pui, J. Lay, N. Alexis (Vancouver, Canada; Chapel Hill, United States Of America)
Source:
Annual Congress 2010 - Causal factors and mechanisms of outdoor air pollution and nanoparticulates
Session:
Causal factors and mechanisms of outdoor air pollution and nanoparticulates
Session type:
E-Communication Session
Number:
431
Disease area:
Airway diseases
Rating:
You must
login
to grade this presentation.
Share or cite this content
Citations should be made in the following way:
C. Carlsten, M. Pui, J. Lay, N. Alexis (Vancouver, Canada; Chapel Hill, United States Of America). Diesel exhaust inhalation causes elevated oxidative burst in airway macrophages and neutrophils that is mitigated by anti-oxidant supplementation. Eur Respir J 2010; 36: Suppl. 54, 431
You must
login
to share this Presentation/Article on Twitter, Facebook, LinkedIn or by email.
Member's Comments
No comment yet.
You must
Login
to comment this presentation.
Related content which might interest you:
Late Breaking Abstract - Implications of treatable traits and treatment choices on exacerbation risk in moderate-severe asthma
Observational cohort study of pulmonary exacerbations in alpha-1 antitrypsin deficiency
Management of Severe Asthma in Pediatric Patients by an Interdisciplinary Team in a Public Hospital Setting.
Related content which might interest you:
N-acetylcysteine prevents pulmonary decrement and neutrophils recruitment in the airways but does not reduce the products of oxidative stress due to ozone exposure in asthmatics
Source: Annual Congress 2008 - Occupational asthma - agents and management
Year: 2008
Airway antioxidant and inflammatory responses to diesel exhaust exposure in healthy humans
Source: Eur Respir J 2006; 27: 359-365
Year: 2006
PP123 – Dimethyl fumarate attenuates lung inflammation and oxidative stress induced by chronic exposure to diesel exhaust particles in mice
Source: ERS Lung Science Conference 2021
Year: 2021
Diesel exhaust exposure enhances the ozone-induced airway inflammation in healthy humans
Source: Eur Respir J 2008; 31: 1234-1240
Year: 2008
Nrf2 is closely related to enhance bleomycin induced airway inflammatory responses caused by diesel exhaust particles in mice
Source: Annual Congress 2012 - Fibrogenesis between epithelial injury and fibroblast proliferation
Year: 2012
Aerobic exercise inhibit pulmonary damage induced by exposition to diesel exhaust particles
Source: Annual Congress 2010 - Causal factors and mechanisms of outdoor air pollution and nanoparticulates
Year: 2010
Diesel exhaust enhances airway responsiveness in asthmatic subjects
Source: Eur Respir J 2001; 17: 909-915
Year: 2001
Effects of swimming on lung inflammation and oxidative stress in diesel exposed mice
Source: Annual Congress 2012 - Indoor and outdoor air pollution
Year: 2012
Oxidative stress by cigarette smoke exposure mediates decrease in ventilation competence and airway neutrophil infiltration in mice
Source: Eur Respir J 2005; 26: Suppl. 49, 578s
Year: 2005
Differential effects of diesel exhaust particles and endotoxin on phagocytosis and cytokine release by monocyte-derived macrophages
Source: Annual Congress 2010 - Macrophages: cell biology and disease-specific characteristics
Year: 2010
Nitric oxide and N-acetylcysteine reduced oxidative stress in alveolar macrophages exposed to cigarette smoke extract
Source: Annual Congress 2010 - Macrophages: cell biology and disease-specific characteristics
Year: 2010
Different airway inflammatory responses in asthmatic and healthy humans exposed to diesel
Source: Eur Respir J 2004; 23: 82-86
Year: 2004
The role of glutaredoxin-1 in pulmonary inflammation induced by diesel exhaust particles
Source: Annual Congress 2013 –Murine models of lung immunology
Year: 2013
Diesel exhaust exposure suppresses alveolar macrophage Cu,Zn superoxide dismutase and catalase activities in mild asthmatic subjects
Source: Eur Respir J 2007; 30: Suppl. 51, 484s
Year: 2007
Inhalation of ultrafine carbon particles triggers biphasic pro-inflammatory response in the mouse lung
Source: Eur Respir J 2006; 28: 275-285
Year: 2006
Protective effects of nootkatone against particles-induced lung injury caused by diesel exhaust is mediated via the NF-?B pathway
Source: International Congress 2018 – Occupational and environmental lung diseases: inhaled air quality and health effects
Year: 2018
Junctional modulation by lipopolysaccharide on epithelial damage caused by diesel exhaust particles
Source: International Congress 2018 – COPD: novel pathological mechanisms in the airway epithelium
Year: 2018
Protective effect of curcumin on pulmonary and cardiovascular effects induced by repeated exposure to diesel exhaust particles
Source: Annual Congress 2012 - Indoor and outdoor air pollution
Year: 2012
Allergen inhalation generates pro-inflammatory oxidised phosphatidylcholine associated with airway dysfunction
Source: Eur Respir J, 57 (2) 2000839; 10.1183/13993003.00839-2020
Year: 2021
The effects of diesel particulates on normal and asthmatic airways
Source: ISSN=1025-448x, ISBN=1-904097-24-3, page=193
Year: 2002
We use cookies on our website to give you the most relevant experience by remembering your preferences and repeat visits. By clicking "Accept", you consent to the use of the cookies.
Accept