e-learning
resources
Barcelona 2010
Tuesday, 21.09.2010
Pre-clinical models of airways disease
Login
Search all ERS
e-learning
resources
Disease Areas
Airways Diseases
Interstitial Lung Diseases
Respiratory Critical Care
Respiratory Infections
Paediatric Respiratory Diseases
Pulmonary Vascular Diseases
Sleep and Breathing Disorders
Thoracic Oncology
Events
International Congress
Courses
Webinars
Conferences
Research Seminars
Journal Clubs
Publications
Breathe
Monograph
ERJ
ERJ Open Research
ERR
European Lung White Book
Handbook Series
Guidelines
All ERS guidelines
e-learning
CME Online
Case reports
Short Videos
SpirXpert
Procedure Videos
CME tests
Reference Database of Respiratory Sounds
Radiology Image Challenge
Brief tobacco interventions
EU Projects
VALUE-Dx
ERN-LUNG
ECRAID
UNITE4TB
Disease Areas
Events
Publications
Guidelines
e-learning
EU Projects
Login
Search
Airway epithelial cell P2Y6R-Expression is up-regulated during acute and chronic allergic inflammation and contributes via modulating of CXC8 and IL-6 secretion to development of airway remodelling
R. de Paula Vieira, T. Muller, J. Christian Virchow, Jr., V. Gernler, M. Grimm, S. Cicko, T. Durk, S. Sorichter, R. Zeiser, M. Idzko (Rostock, Germany)
Source:
Annual Congress 2010 - Pre-clinical models of airways disease
Session:
Pre-clinical models of airways disease
Session type:
Oral Presentation
Number:
5148
Disease area:
Airway diseases
Rating:
You must
login
to grade this presentation.
Share or cite this content
Citations should be made in the following way:
R. de Paula Vieira, T. Muller, J. Christian Virchow, Jr., V. Gernler, M. Grimm, S. Cicko, T. Durk, S. Sorichter, R. Zeiser, M. Idzko (Rostock, Germany). Airway epithelial cell P2Y6R-Expression is up-regulated during acute and chronic allergic inflammation and contributes via modulating of CXC8 and IL-6 secretion to development of airway remodelling. Eur Respir J 2010; 36: Suppl. 54, 5148
You must
login
to share this Presentation/Article on Twitter, Facebook, LinkedIn or by email.
Member's Comments
No comment yet.
You must
Login
to comment this presentation.
Related content which might interest you:
Late Breaking Abstract - Implications of treatable traits and treatment choices on exacerbation risk in moderate-severe asthma
Management of Severe Asthma in Pediatric Patients by an Interdisciplinary Team in a Public Hospital Setting.
Respiratory sequelae of preterm birth across the life span
Related content which might interest you:
IL-33/sST2 balance regulates neutrophilic inflammation in the human airways.
Source: International Congress 2019 – Understanding the mechanisms underlying airway diseases
Year: 2019
A dual role of IL-6 signalling and STAT-3 activation on airway inflammation versus airway hyperresponsiveness in a murine model of asthma
Source: Eur Respir J 2002; 20: Suppl. 38, 568s
Year: 2002
Evidence for secretion of IL-18 in asthmatic airway inflammation assessed by endobronchial allergen challenge and its relationship to accumulation of inflammatory cells
Source: Eur Respir J 2001; 18: Suppl. 33, 531s
Year: 2001
Increased arginase activity contributes to airway remodelling in chronic allergic asthma
Source: Eur Respir J 2011; 38: 318-328
Year: 2011
IL-8 and TSLP production as markers of bronchial epithelial cell activation in IL-17A mediated airway inflammation in COPD: Role of tiotropium
Source: Annual Congress 2013 –New insights into epigenetic mechanisms in the lung
Year: 2013
Airway G-CSF identifies neutrophilic inflammation and contributes to asthma progression
Source: Eur Respir J, 55 (2) 1900827; 10.1183/13993003.00827-2019
Year: 2020
Resveratrol impairs the release of steroid-resistant inflammatory cytokines from human airway smooth muscle cells in COPD
Source: Annual Congress 2010 - Airway cells as targets for anti-inflammatory drugs
Year: 2010
Role of cytokines and chemokines in bronchial hyperresponsiveness and airway inflammation and remodelling
Source: Annual Congress 2007 - PG1 - Asthma: from bench to bedside
Year: 2007
IL-33 blockade prevents exacerbations in a model of chronic airway inflammation by blunting persistent inflammation and remodeling
Source: International Congress 2019 – Advances in asthma pharmacology
Year: 2019
IL-25 secreted from epithelial cells has the potential to promote airway remodeling in asthma
Source: Annual Congress 2012 - Asthma and COPD: understanding through mechanisms
Year: 2012
IL-33 blockade impacts mediators of persistence and exacerbation of chronic airway inflammation.
Source: International Congress 2018 – Novel drug targets for asthma and COPD
Year: 2018
P-selectin expression in chronic allergic airway inflammation
Source: Eur Respir J 2001; 18: Suppl. 33, 271s
Year: 2001
Interleukin-17A induces glucocorticoid insensitivity in human bronchial epithelial cells
Source: Eur Respir J 2012; 39: 439-445
Year: 2012
Allergen-induced BDNF expression and neuronal remodeling contributes to airway hyperresponsiveness
Source: International Congress 2018 – Preclinical pharmacology: novel targets in airway inflammation, narrowing and neural control
Year: 2018
A role for Th1 cells in airway remodeling and hyperreactivity in mice subjected to chronic allergen exposure
Source: Eur Respir J 2007; 30: Suppl. 51, 734s
Year: 2007
Role of IL-33 signaling in pollutant-induced allergic airway inflammation
Source: International Congress 2018 – Translational models of the immunopathology that underlies airway obstructive diseases
Year: 2018
Let-7 promotes airway remodeling via regulation of IL-6 in chronic obstruction pulmonary disease
Source: Virtual Congress 2020 – Respiratory homeostasis and structural changes in disease
Year: 2020
Aerobic exercise reduces allergic airway inflammation and remodeling by deactivating airway epithelial cells and leukocytes
Source: Annual Congress 2011 - Asthma: basic science and clinical studies
Year: 2011
We use cookies on our website to give you the most relevant experience by remembering your preferences and repeat visits. By clicking "Accept", you consent to the use of the cookies.
Accept