Electronic cigarette aerosol condensate is cytotoxic and increases ACE2 expression in human airway epithelial cells: implications for COVID-19
K. McAlinden (Launceston, Australia), W. Lu (Launceston, Australia), P. Vahidi Ferdowsi (Launceston, Australia), S. Myers (Launceston, Australia), J. Markos (Launceston, Australia), C. Chia (Launceston, Australia), H. Weber (Burnie, Australia), G. Haug (Launceston, Australia), M. Eapen (Launceston, Australia), S. Singh Sohal (Launceston, Australia)
Source: Virtual Congress 2021 – Pathogenic progress in airway diseases
Session: Pathogenic progress in airway diseases
Session type: E-poster
Number: 2394
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K. McAlinden (Launceston, Australia), W. Lu (Launceston, Australia), P. Vahidi Ferdowsi (Launceston, Australia), S. Myers (Launceston, Australia), J. Markos (Launceston, Australia), C. Chia (Launceston, Australia), H. Weber (Burnie, Australia), G. Haug (Launceston, Australia), M. Eapen (Launceston, Australia), S. Singh Sohal (Launceston, Australia). Electronic cigarette aerosol condensate is cytotoxic and increases ACE2 expression in human airway epithelial cells: implications for COVID-19. 2394
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