Mitochondrial antiviral signaling protein is crucial for the development of pulmonary fibrosis Source: Eur Respir J, 57 (4) 2000652; 10.1183/13993003.00652-2020 Year: 2021
PGAM5 acts as a key mediator of lung fibrosis by disrupting mitochondrial homeostasis Source: International Congress 2019 – Extracellular matrix formation and remodelling in physiology and disease Year: 2019
PP208 – SRC family kinases modulate molecular pathways associated with mitochondrial dysfunction in idiopathic pulmonary fibrosis Source: ERS Lung Science Conference 2021 Year: 2021
ELMOD2, a candidate gene for idiopathic pulmonary fibrosis, regulates antiviral responses Source: Annual Congress 2010 - Lung cell biology and immunology Year: 2010
The matrix-fibroblast interplay contributes to HIF-1 alpha pathway activation: a possible therapeutic target in idiopathic pulmonary fibrosis (IPF) Source: Virtual Congress 2020 – Elucidating the mechanisms underlying lung repair and exploring therapeutic strategies in vitro Year: 2020
The pathomechanistic role of autophagy in lung fibrosis Source: International Congress 2017 – Interstitial lung diseases (ILDs): novel pathogenetic pathways Year: 2017
Regulation of alternative macrophage activation by galectin-3; potential role in lung fibrosis Source: Annual Congress 2009 - What is new in molecular pathology and functional genomics of neoplastic and non-neoplastic lung disease? Year: 2009
LSC - 2021 - SRC family kinases modulate molecular pathways associated with mitochondrial dysfunction in idiopathic pulmonary fibrosis Source: Virtual Congress 2021 – Cellular and molecular involvement in lung injury and repair Year: 2021
Cigarette-decreased NAD/SIRT1 aggravates pulmonary fibrosis by promoting senescence of alveolar type 2 cells through autophagy inhibition and mitochondrial oxidative stress induction Source: Virtual Congress 2020 – Idiopathic pulmonary fibrosis and interstitial lung disease: progression, genes and more Year: 2020
The potential role of intracellular heat shock protein 70 deficiency in aging and pulmonary fibrosis. Source: International Congress 2017 – Novel pro- and anti-fibrotic mechanisms in pulmonary fibrosis Year: 2017
Extracellular HSP90a promotes cellular senescence in pulmonary fibrosis through TGFß signaling Source: Virtual Congress 2020 – Idiopathic pulmonary fibrosis and interstitial lung disease: progression, genes and more Year: 2020
mTOR signalling is an essential pathway for TGF-β1 induced collagen synthesis Source: International Congress 2015 – Pulmonary fibrosis: mechanisms of disease Year: 2015
TRIM33 prevents pulmonary fibrosis by impairing TGF-ß1 signalling Source: Eur Respir J, 55 (6) 1901346; 10.1183/13993003.01346-2019 Year: 2020
Defective p53-mediated pro-apoptotic pathway in idiopathic pulmonary fibrosis Source: Annual Congress 2008 - New insights into the pathogenesis of idiopathic pulmonary fibrosis and sarcoidosis Year: 2008
The role of extracellular vesicles in phenotypic alteration of lung cells in pulmonary fibrosis Source: Annual Congress 2013 –The role of hormones and cytokines in lung injury Year: 2013
Oxidative DNA damage, impaired mitochondria, and activation of caspase cascade in idiopathic pulmonary fibrosis Source: Eur Respir J 2001; 18: Suppl. 33, 358s Year: 2001
The potential role of antimicrobial peptides from wasp as regulators of the fibrotic process in idiopathic pulmonary fibrosis. Source: International Congress 2019 – Assessment and management of immune-mediated interstitial lung diseases Year: 2019
ENaC inhibition in cystic fibrosis: potential role in the new era of CFTR modulator therapies Source: Eur Respir J, 56 (6) 2000946; 10.1183/13993003.00946-2020 Year: 2020
FK506-binding protein 11, a novel plasma cell specific antibody folding catalyst, is increased in idiopathic pulmonary fibrosis Source: Virtual Congress 2020 – Novel immunopathological mechanisms of lung disease: knowledge from translational studies Year: 2020
SH2 domain-containing Phosphatase-(SHP)-2 blunts fibrotic responses through regulation of fibroblast mitochondrial metabolism and autophagy Source: International Congress 2019 – Extracellular matrix formation and remodelling in physiology and disease Year: 2019