e-learning
resources
Munich 2014
Tuesday, 09.09.2014
Cell biology 2014
Login
Search all ERS
e-learning
resources
Disease Areas
Airways Diseases
Interstitial Lung Diseases
Respiratory Critical Care
Respiratory Infections
Paediatric Respiratory Diseases
Pulmonary Vascular Diseases
Sleep and Breathing Disorders
Thoracic Oncology
Events
International Congress
Courses
Webinars
Conferences
Research Seminars
Journal Clubs
Publications
Breathe
Monograph
ERJ
ERJ Open Research
ERR
European Lung White Book
Handbook Series
Guidelines
All ERS guidelines
e-learning
CME Online
Case reports
Short Videos
SpirXpert
Procedure Videos
CME tests
Reference Database of Respiratory Sounds
Radiology Image Challenge
Brief tobacco interventions
EU Projects
VALUE-Dx
ERN-LUNG
ECRAID
UNITE4TB
Disease Areas
Events
Publications
Guidelines
e-learning
EU Projects
Login
Search
The role of chemokine receptors in the aberrant migration of COPD neutrophils
J. Stockley, L. Peat, G. Walton, J. Lord, E. Sapey (Birmingham, United Kingdom)
Source:
International Congress 2014 – Cell biology 2014
Session:
Cell biology 2014
Session type:
Thematic Poster Session
Number:
3859
Disease area:
Airway diseases
Rating:
You must
login
to grade this presentation.
Share or cite this content
Citations should be made in the following way:
J. Stockley, L. Peat, G. Walton, J. Lord, E. Sapey (Birmingham, United Kingdom). The role of chemokine receptors in the aberrant migration of COPD neutrophils. Eur Respir J 2014; 44: Suppl. 58, 3859
You must
login
to share this Presentation/Article on Twitter, Facebook, LinkedIn or by email.
Member's Comments
No comment yet.
You must
Login
to comment this presentation.
Related content which might interest you:
Panel discussion on ERS Statement – A core outcome set for clinical trials evaluating the management of COPD exacerbations
ERS statement: a core outcome set for clinical trials evaluating the management of COPD exacerbations
Evidence-based eHealth for COPD
Related content which might interest you:
Imbalance of immune response functional phenotype and alveolar macrophages phenotype in COPD
Source: International Congress 2014 – New insights into monocyte and macrophage biology
Year: 2014
Lymphocyte senescence in COPD is associated with loss of glucocorticoid receptor expression by cytotoxic/ pro-inflammatory lymphocytes
Source: International Congress 2014 – Effect of cigarette smoke on cell behaviour: cell biology, signalling and senescence
Year: 2014
Oxidative stress drives defective efferocytosis in COPD M2-like macrophages
Source: International Congress 2014 – New insights into monocyte and macrophage biology
Year: 2014
Expression of CX3CL1 and CX3CR1 in human asthmatic lung in relation to cell survival
Source: International Congress 2014 – New mechanisms in the pathogenesis of asthma and other lung diseases
Year: 2014
Evidence for a novel kit adhesion domain mediating human mast cell adhesion to structural airway cells
Source: International Congress 2014 – Cell biology 2014
Year: 2014
Elevated GM-CSF in COPD may drive defective macrophage phenotype
Source: International Congress 2014 – New insights into monocyte and macrophage biology
Year: 2014
Smoking alters alpha1-antitrypsin in alveolar macrophages possibly promoting misfolding
Source: International Congress 2014 – New studies into pulmonary proteases and antiproteases
Year: 2014
Comparison of inflammatory markers in induced and spontaneous sputum in COPD patients
Source: International Congress 2014 – Translational studies in COPD
Year: 2014
COPD airway epithelium secretion attracts and activates neutrophils
Source: International Congress 2014 – Novel evidence relating to respiratory treatment development
Year: 2014
The effect of budesonide on TLR expression in alveolar macrophages from smokers
Source: International Congress 2014 – New insights into monocyte and macrophage biology
Year: 2014
Interleukin-18 levels in induced sputum of stable and exacerbated COPD patients
Source: International Congress 2014 – Translational studies in COPD
Year: 2014
Macrophage migration inhibitory factor (MIF), a biomarker in COPD?
Source: International Congress 2014 – New insights into monocyte and macrophage biology
Year: 2014
The role of circulating mitochondrial DNA in modulating airway inflammation of chronic obstructive pulmonary disease
Source: International Congress 2014 – Cell biology 2014
Year: 2014
Hyperoxia impairs the inflammatory response of alveolar macrophages to multiple Toll-like receptor ligands associated with bacterial infection
Source: International Congress 2014 – New insights into monocyte and macrophage biology
Year: 2014
Crosstalk between epithelium and fibroblasts; implications for COPD
Source: International Congress 2014 – Mechanistic studies of the airway epithelium
Year: 2014
CEACAM6-high airway neutrophils and epithelial cells are a feature of severe asthma
Source: International Congress 2015 – Translational studies in airway cell biology
Year: 2015
Increased expression of receptor for advanced glycation end products in COPD
Source: International Congress 2014 – Translational studies in COPD
Year: 2014
Differential sensitivity of subsets of monocyte-derived macrophages to apoptosis; its impact on lung inflammation
Source: International Congress 2014 – New insights into monocyte and macrophage biology
Year: 2014
Functional phenotype of immune response and alveolar macrophages phenotype in pulmonary sarcoidosis
Source: International Congress 2014 – New insights into monocyte and macrophage biology
Year: 2014
LATE-BREAKING ABSTRACT: BAFF: a self-perpetuating mechanism of B cell activation in COPD
Source: International Congress 2014 – COPD physiopathology
Year: 2014
We use cookies on our website to give you the most relevant experience by remembering your preferences and repeat visits. By clicking "Accept", you consent to the use of the cookies.
Accept