Source: Annual Congress 2006 - Cellular biology of asthma and COPD
Disease area: Airway diseases



Rating:

Share or cite this content

Citations should be made in the following way:
H. Imaoka, T. Hoshino, T. Kinoshita, Y. Kitasato, T. Kawayama, T. Koga, S. Kato, T. Shirakusa, K. Watanabe, H. Aizawa (Kurume, Fukuoka, Japan). Enhanced expression of proinflammatory cytokine (IL-18) and its receptor in patients with chronic obstructive pulmonary disease (COPD). Eur Respir J 2006; 28: Suppl. 50, 3178

Member's Comments

No comment yet.

Related content which might interest you: Enhanced expression of IL-18 receptor but not soluble form of IL18R in chronic obstructive pulmonary disease (COPD) Source: Annual Congress 2011 - COPD: clinical studies and animal cell models Year: 2011 Polymorphisms of IL-4, IL-10, and TNF-α gene in chronic obstructive pulmonary disease Source: Eur Respir J 2005; 26: Suppl. 49, 442s Year: 2005 Neutrophil chemokine receptor (CXCR1 and 2) gene expression and biopsy neutrophilia in severe exacerbations of chronic obstructive pulmonary disease (COPD) Source: Eur Respir J 2001; 18: Suppl. 33, 501s Year: 2001 Levels of CC-chemokines (MCP-1, MIP-1α, MIP-1β) in induced sputum of patients with chronic obstructive pulmonary disease and patients with chronic bronchitis Source: Eur Respir J 2001; 18: Suppl. 33, 194s Year: 2001 The level of anti-inflammatory cytokine IL-10 in patients with chronic obstructive pulmonary disease of varying severity Source: Annual Congress 2013 –COPD: new markers of diseases Year: 2013 Relationships between IL6 and TNF µ and disability in chronic obstructive pulmonary disease (COPD) Source: Eur Respir J 2005; 26: Suppl. 49, 440s Year: 2005 Expression profiling of Th17 associated molecules in bronchoalveolar cells from patients with chronic obstructive pulmonary disease (COPD) Source: Annual Congress 2011 - COPD: clinical studies and animal cell models Year: 2011 Role of systemic inflammation mediated by IL-6 and TNF-alpha cytokines in chronic obstructive pulmonary disease and its progression: A three year follow up study Source: Annual Congress 2013 –Systemic and airway biomarkers in respiratory diseases Year: 2013 Release of IL-1b, IL-6 and TNF-a after pulmonary rehabilitation in patients with chronic obstructive pulmonary disease Source: Annual Congress 2008 - Physiological response to exercise performance Year: 2008 Interleukin-18 production and pulmonary function in COPD Source: Eur Respir J 2008; 31: 287-297 Year: 2008 Increased lung apoptosis and expression of ceramide in subjects with chronic obstructive pulmonary disease (COPD) Source: Annual Congress 2009 - COPD: basic science and clinical studies Year: 2009 Altered expression of TLR4 and of beta 2 defensin in chronic obstructive pulmonary disease Source: Eur Respir J 2006; 28: Suppl. 50, 549s Year: 2006 Altered expression of TLR4 and of beta defensin 2 in chronic obstructive pulmonary disease Source: Eur Respir J 2007; 30: Suppl. 51, 213s Year: 2007 Blood cytokine expression in patients with chronic obstructive airway diseases (COAD) Source: International Congress 2017 – Systemic and airway biomarkers Year: 2017 Potential association between pulmonary hypertension in COPD and monocyte chemoattractant protein (MCP)-1 Source: Annual Congress 2005 - Pulmonary hypertension: pathophysiology Year: 2005 Investigation of alarmins, interleukin (IL-)23 and IL-36 sputum levels in chronic airway obstructive diseases. Source: International Congress 2019 – Understanding the mechanisms underlying airway diseases Year: 2019 Suppressed pulmonary expression of leptin in lipopolysaccharide-induced acute and chronic lung inflammation Source: Annual Congress 2006 - Novel pathogenic aspects of acute lung injury and acute respiratory distress syndrome (ARDS) Year: 2006 IL-8 in patients with chronic obstructive pulmonary disease Source: Eur Respir J 2001; 18: Suppl. 33, 364s Year: 2001 STAT4 activation in smokers and patients with chronic obstructive pulmonary disease Source: Eur Respir J 2004; 24: 78-85 Year: 2004 TNF and LTA genes and susceptibility to chronic obstructive pulmonary disease Source: Eur Respir J 2004; 24: Suppl. 48, 305s Year: 2004