e-learning
resources
Barcelona 2010
Monday, 20.09.2010
Cystic fibrosis: clinical and laboratory studies
Login
Search all ERS
e-learning
resources
Disease Areas
Airways Diseases
Interstitial Lung Diseases
Respiratory Critical Care
Respiratory Infections
Paediatric Respiratory Diseases
Pulmonary Vascular Diseases
Sleep and Breathing Disorders
Thoracic Oncology
Events
International Congress
Courses
Webinars
Conferences
Research Seminars
Journal Clubs
Publications
Breathe
Monograph
ERJ
ERJ Open Research
ERR
European Lung White Book
Handbook Series
Guidelines
All ERS guidelines
e-learning
CME Online
Case reports
Short Videos
SpirXpert
Procedure Videos
CME tests
Reference Database of Respiratory Sounds
Radiology Image Challenge
Brief tobacco interventions
EU Projects
VALUE-Dx
ERN-LUNG
ECRAID
UNITE4TB
Disease Areas
Events
Publications
Guidelines
e-learning
EU Projects
Login
Search
Alpha-1 antitrypsin (AAT) in cystic fibrosis-associated endoplasmic reticulum (ER) stress
A. Hector, M. Kappler, M. Griese, D. Hartl (Munich, Germany)
Source:
Annual Congress 2010 - Cystic fibrosis: clinical and laboratory studies
Session:
Cystic fibrosis: clinical and laboratory studies
Session type:
Thematic Poster Session
Number:
2663
Disease area:
Airway diseases
Rating:
You must
login
to grade this presentation.
Share or cite this content
Citations should be made in the following way:
A. Hector, M. Kappler, M. Griese, D. Hartl (Munich, Germany). Alpha-1 antitrypsin (AAT) in cystic fibrosis-associated endoplasmic reticulum (ER) stress. Eur Respir J 2010; 36: Suppl. 54, 2663
You must
login
to share this Presentation/Article on Twitter, Facebook, LinkedIn or by email.
Member's Comments
No comment yet.
You must
Login
to comment this presentation.
Related content which might interest you:
Late Breaking Abstract - Implications of treatable traits and treatment choices on exacerbation risk in moderate-severe asthma
Impact of Dexamethasone on pathogen profile of COVID-19 patients requiring intensive care: a multicentre retrospective study
Muscle energy techniques for COPD patients: Effects on pulmonary function and activities of daily living
Related content which might interest you:
Alpha-1 antitrypsin deficiency (AATD) and airway imflammation: Methods to study endoplasmatic reticulum stress (ERS)
Source: Annual Congress 2010 - Proteases and the matrix
Year: 2010
Endoplasmic reticulum stress in circulating cystic fibrosis neutrophils
Source: Annual Congress 2012 - Cystic fibrosis (adults and children): new basic and clinical physiology research
Year: 2012
Endoplasmic reticulum stress in COPD: implications for alpha1-antitrypsin deficiency
Source: Annual Congress 2011 - The unfolding story of endoplasmic reticulum stress signalling in lung disease
Year: 2011
Epithelial endoplasmic reticulum stress and apoptosis in sporadic idiopathic pulmonary fibrosis
Source: Annual Congress 2008 - Management of idiopathic pulmonary fibrosis: what can we learn from the pathophysiology?
Year: 2008
F alpha-1 antitrypsin polymerization activates endoplasmic reticulum overload response
Source: International Congress 2014 – Mechanisms of COPD: proteases and a whole lot more
Year: 2014
Intra-epithelial polymerization, accumulation and secretion of alpha
1
antitrypsin (AAT) Z polymers in the lung of patients with alpha-1 antitrypsin deficiency (AATD)
Source: Annual Congress 2010 - Epithelial cell biology
Year: 2010
Endoplasmic reticulum stress in lung disease
Source: Eur Respir Rev, 26 (144) 170018; 10.1183/16000617.0018-2017
Year: 2017
Alpha 1 antitrypsin (A1AT) deficiency among patients with cystic fibrosis
Source: Eur Respir J 2003; 22: Suppl. 45, 501s
Year: 2003
LSC Abstract – Alpha1-antitrypsin reduces inflammation and endoplasmic reticulum stress in lung epithelial cells in vitro
Source: International Congress 2015 – Cellular and molecular mechanisms of COPD and emphysema
Year: 2015
Alpha-1 antitrypsin (AAT) polymerization in alveolar macrophages of AAT deficient individuals and in smokers.
Source: International Congress 2017 – New Mechanistic Findings in COPD
Year: 2017
Association between alpha1 antitrypsin (AAT) circulating polymers (CP) and lung and liver disease in patients with AAT deficiency (AATD).
Source: Virtual Congress 2021 – Molecular pathology and cell biology of pulmonary diseases
Year: 2021
Effects of double dose alpha 1 antitrypsin (AAT) therapy on cytokine pathways in AAT Deficiency (AATD)
Source: International Congress 2018 – COPD phenotypes: a1 antitrypsin deficiency and beyond
Year: 2018
Regional deposition of
99m
Tc-labelled alpha-1 antitrypsin (AAT) in patients with alpha-1 antitrypsin deficiency and cystic fibrosis (CF) compared with healthy volunteers
Source: Annual Congress 2007 - Cystic fibrosis: the balance between treatment and side-effects
Year: 2007
A quantitative method for the detection of spliced X-box binding protein-1 (XBP1) mRNA in primary bronchial epithelial cells (PBEC) as a measure of endoplasmatic reticulum (ER) stress
Source: Annual Congress 2011 - Lung cell biology
Year: 2011
A high purity alpha-1 antitrypsin (AAT) from human plasma, TAL6004
Source: Annual Congress 2009 - Treatment of airways disease
Year: 2009
Surfactant protein C G100S mutation causes familial pulmonary fibrosis in Japanese kindred
Source: Eur Respir J 2011; 38: 861-869
Year: 2011
Inflammation in cystic fibrosis airways: relationship to increased bacterial adherence
Source: Eur Respir J 2001; 17: 27-35
Year: 2001
Pseudomonas aeruginosa
causes endoplasmic reticulum (ER) stress in bronchial epithelial cells via its virulence factors pyocyanin (PCN) and alkaline protease (AP)
Source: Annual Congress 2013 –Stress responses and T-cell behaviour in the lung
Year: 2013
Endoplasmic reticulum stress implication in senescence of small airway fibroblasts from COPD patients.
Source: Virtual Congress 2020 – Elucidating the mechanisms underlying lung repair and exploring therapeutic strategies in vitro
Year: 2020
We use cookies on our website to give you the most relevant experience by remembering your preferences and repeat visits. By clicking "Accept", you consent to the use of the cookies.
Accept