e-learning
resources
Barcelona 2010
Monday, 20.09.2010
Novel insights into the pathophysiology of asthma and COPD
Login
Search all ERS
e-learning
resources
Disease Areas
Airways Diseases
Interstitial Lung Diseases
Respiratory Critical Care
Respiratory Infections
Paediatric Respiratory Diseases
Pulmonary Vascular Diseases
Sleep and Breathing Disorders
Thoracic Oncology
Events
International Congress
Courses
Webinars
Conferences
Research Seminars
Journal Clubs
Publications
Breathe
Monograph
ERJ
ERJ Open Research
ERR
European Lung White Book
Handbook Series
Guidelines
All ERS guidelines
e-learning
CME Online
Case reports
Short Videos
SpirXpert
Procedure Videos
CME tests
Reference Database of Respiratory Sounds
Radiology Image Challenge
Brief tobacco interventions
EU Projects
VALUE-Dx
ERN-LUNG
ECRAID
UNITE4TB
Disease Areas
Events
Publications
Guidelines
e-learning
EU Projects
Login
Search
Hyperoxia-induced changes of glutathione system in red blood cells and alveolar macrophages
I. Katovich, Z. Rutkovskaya, A. Tahanovich (Minsk, Belarus)
Source:
Annual Congress 2010 - Novel insights into the pathophysiology of asthma and COPD
Session:
Novel insights into the pathophysiology of asthma and COPD
Session type:
Thematic Poster Session
Number:
2477
Disease area:
Airway diseases, Paediatric lung diseases
Rating:
You must
login
to grade this presentation.
Share or cite this content
Citations should be made in the following way:
I. Katovich, Z. Rutkovskaya, A. Tahanovich (Minsk, Belarus). Hyperoxia-induced changes of glutathione system in red blood cells and alveolar macrophages. Eur Respir J 2010; 36: Suppl. 54, 2477
You must
login
to share this Presentation/Article on Twitter, Facebook, LinkedIn or by email.
Member's Comments
No comment yet.
You must
Login
to comment this presentation.
Related content which might interest you:
From pulmonary gas exchange to the interpretation of arterial blood gases
Late Breaking Abstract - Implications of treatable traits and treatment choices on exacerbation risk in moderate-severe asthma
Management of Severe Asthma in Pediatric Patients by an Interdisciplinary Team in a Public Hospital Setting.
Related content which might interest you:
Alveolar presence of red blood cells correlates with alveolar macrophage iron levels in COPD
Source: Virtual Congress 2020 – Novel immunopathological mechanisms of lung disease: knowledge from translational studies
Year: 2020
Paraquat induced damage in rat lung epithelial cells and alveolar macrophages
Source: Eur Respir J 2001; 18: Suppl. 33, 79s
Year: 2001
Compartimentalized in-vitro TNF-α production of alveolar macrophages and peripheral mononuclear cells after severe trauma
Source: Eur Respir J 2001; 18: Suppl. 33, 317s
Year: 2001
Increased cytokine production by peripheral blood mononuclear cells (PBMC) and alveolar macrophages (AM) in active sarcoidosis
Source: Eur Respir J 2003; 22: Suppl. 45, 177s
Year: 2003
Trans-basement membrane migration of eosinophils induced by LPS-stimulated neutrophils from human peripheral blood in vitro
Source: ERJ Open Res 2015; 1: 00003-2015
Year: 2015
Kaempferol-induced HO-1 expression attenuated inflammation in human pulmonary alveolar epithelial cells
Source: International Congress 2017 – Therapeutic approaches in lung disease
Year: 2017
RAGE, a marker of alveolar epithelial type I cell injury, predicts impaired alveolar fluid clearance in isolated perfused human lungs
Source: Annual Congress 2007 - New insights into clinical lung transplantation
Year: 2007
Modulation of NOS2 expression in co-cultures of human alveolar epithelial cells type II and alveolar macrophages
Source: Eur Respir J 2001; 18: Suppl. 33, 161s
Year: 2001
Development of assays to evaluate inflammasomes’ activity in the peripheral blood and in alveolar macrophages in fibrotic lung diseases
Source: International Congress 2018 – Back to basics and translational research in idiopathic interstitial pneumonias
Year: 2018
Trafficking of mononuclear phagocytes into the alveolar space of mice is associated with drastic changes in their gene expression profiles
Source: Eur Respir J 2005; 26: Suppl. 49, 730s
Year: 2005
The intracellular thioredoxin system mediates inflammatory activation of human lung microvascular endothelial cells
Source: Eur Respir J 2003; 22: Suppl. 45, 223s
Year: 2003
Expression of hyperoxidized peroxiredoxins is enhanced in peripheral blood mononuclear cells of bronchial asthma patients
Source: Annual Congress 2011 - Experimental modulation of airway inflammation
Year: 2011
Role of p53 in murine macrophages and alveolar epithelial cells response to nanoparticles
Source: Annual Congress 2011 - Environmental exposure and disease mechanisms
Year: 2011
Postconditioning with a volatile anaesthetic in alveolar epithelial cells in vitro
Source: Eur Respir J 2008; 31: 118-125
Year: 2008
Co-culturing alveolar epithelial with lung micro-vascular endothelial cells attenuates hypoxia-induced alveolar leakage and inhibition of reabsorption
Source: Annual Congress 2009 - Crossing the line: alveolo-capillary barrier control in acute lung injury
Year: 2009
Hypoxia sensing by red blood cells: inflammatory consequences in lung
Source: Annual Congress 2009 - Crossing the line: alveolo-capillary barrier control in acute lung injury
Year: 2009
Elevated percentage of hemosiderin-laden macrophages in the BAL fluid of patients with diffuse alveolar damage
Source: Annual Congress 2008 - Role of bronchoalveolar lavage in studying the pathophysiology of interstitial lung disease
Year: 2008
DNA damage of murine peripheral blood monocytes and histopathologic changes of murine lung induced by Asian dust particles
Source: Annual Congress 2008 - COPD and other environment-related lung diseases
Year: 2008
Decreased expression of haem oxygenase-1 by alveolar macrophages in idiopathic pulmonary fibrosis
Source: Eur Respir J 2008; 31: 1030-1036
Year: 2008
Overheating takes a dual effect on the viability and phagocytic activity of hypoxic alveolar macrophages
Source: Annual Congress 2007 - Inflammatory cell biology
Year: 2007
We use cookies on our website to give you the most relevant experience by remembering your preferences and repeat visits. By clicking "Accept", you consent to the use of the cookies.
Accept