The development of an allergic airway disease is influenced by a short splice variant of CYLD

S. Reuter, M. Becker, O. Arlt, N. Dehzad, H. Martin, A. Waisman, M. Stassen, R. Buhl, C. Taube (Mainz, Germany)

Source: Annual Congress 2010 - Animal models of asthma and lung inflammation
Session: Animal models of asthma and lung inflammation
Session type: Thematic Poster Session
Number: 1348
Disease area: Airway diseases

Congress or journal article abstract

Rating: 0
You must login to grade this presentation.

Share or cite this content

Citations should be made in the following way:
S. Reuter, M. Becker, O. Arlt, N. Dehzad, H. Martin, A. Waisman, M. Stassen, R. Buhl, C. Taube (Mainz, Germany). The development of an allergic airway disease is influenced by a short splice variant of CYLD. Eur Respir J 2010; 36: Suppl. 54, 1348

You must login to share this Presentation/Article on Twitter, Facebook, LinkedIn or by email.

Member's Comments

No comment yet.
You must Login to comment this presentation.


Related content which might interest you:
T cell specific expression of a short splice variant of the tumor suppressor gene CYLD amplifies the development of allergic airway disease
Source: Annual Congress 2011 - T-cell subsets
Year: 2011

A short splice variant of the tumor suppressor gene CYLD induces allergic airway disease in mice and is also expressed in humans
Source: International Congress 2015 – Advances in asthma and allergy in animal models
Year: 2015

Enhanced allergen capture and processing by dendritic cells and other airway APC precedes the onset of allergic airways disease
Source: Annual Congress 2007 - Regulation of allergic airway inflammation in animal models of asthma
Year: 2007


ST2 gene knockout attenuates allergic airways disease
Source: Annual Congress 2006 - Immunological mechanisms in cell recruitment to the lungs
Year: 2006


Osteopontin blockade prevents fibrosis in a murine chronic model of allergic airway disease
Source: Eur Respir J 2007; 30: Suppl. 51, 60s
Year: 2007

Overexpression of eNOS suppresses asthmatic features in a mouse model of allergic asthma
Source: Eur Respir J 2002; 20: Suppl. 38, 28s
Year: 2002

HDAC6 inhibition and deletion reduce COPD symptoms in mice and human airway epithelia
Source: Virtual Congress 2020 – Elucidating the mechanisms underlying lung repair and exploring therapeutic strategies in vitro
Year: 2020




Distinct epithelial phenotypes in child allergic respiratory disease are revealed by gene expression profiling
Source: Annual Congress 2009 - What is new in molecular pathology and functional genomics of neoplastic and non-neoplastic lung disease?
Year: 2009

Characterisation of type two immune responses in a murine model of allergic airway disease
Source: Virtual Congress 2020 – Scientific advances in airway pharmacology
Year: 2020


PDE-4-inhibitor rolipram supports treg-mediated suppression in a murine model of allergic airway disease
Source: Annual Congress 2008 - Risk factors for asthma versus asthma control
Year: 2008

Disruption of CD44 variant 6 abolished bronchial hyperresponsiveness, inflammation and mast cell myositis in a murine model of asthma
Source: Eur Respir J 2006; 28: Suppl. 50, 217s
Year: 2006

Genetic deletion of SLPI promotes inflammatory cell recruitment in a model of chronic lung disease
Source: Virtual Congress 2021 – Emerging new mechanisms of chronic lung disease
Year: 2021


CARD9S12N allele expression imbalance favors development of allergic bronchopulmonary aspergillosis
Source: Virtual Congress 2020 – Inflammatory pathways in chronic airway diseases
Year: 2020

A decreased expression of leptin receptor may contribute to the pathogenesis of the allergic rhinitis
Source: Annual Congress 2010 - Asthma: clinical aspects and treatment
Year: 2010

DPP10 protein expression is associated with the pathogenesis of asthma and COPD
Source: Eur Respir J 2005; 26: Suppl. 49, 231s
Year: 2005

Glutaredoxins in allergic airway disease and regulation by H2O2 in vivo
Source: Eur Respir J 2005; 26: Suppl. 49, 482s
Year: 2005

Chronic persistence of human ?-herpesviruses and regulatory cells phenotypes in severe bronchial asthma.
Source: International Congress 2017 – "Alarming" immunological patterns in asthma
Year: 2017

CpG oligodeoxynucleotide treatment prevents the development of airway remodelling in a murine model of chronic asthma
Source: Eur Respir J 2002; 20: Suppl. 38, 2s
Year: 2002

T-cell co-stimulatory molecules: novel targets for the treatment of allergic airway disease
Source: Eur Respir J 2007; 30: 383-390
Year: 2007



Functional single nucleotide polymorphisms of the CCL5 gene and nonemphysematous phenotype in COPD patients
Source: Eur Respir J 2008; 32: 372-378
Year: 2008