e-learning
resources
Munich 2006
Monday 04.09.2006
Cellular immunology in asthma pathogenesis
Login
Search all ERS
e-learning
resources
Disease Areas
Airways Diseases
Interstitial Lung Diseases
Respiratory Critical Care
Respiratory Infections
Paediatric Respiratory Diseases
Pulmonary Vascular Diseases
Sleep and Breathing Disorders
Thoracic Oncology
Events
International Congress
Courses
Webinars
Conferences
Research Seminars
Journal Clubs
Publications
Breathe
Monograph
ERJ
ERJ Open Research
ERR
European Lung White Book
Handbook Series
Guidelines
All ERS guidelines
e-learning
CME Online
Case reports
Short Videos
SpirXpert
Procedure Videos
CME tests
Reference Database of Respiratory Sounds
Radiology Image Challenge
Brief tobacco interventions
EU Projects
VALUE-Dx
ERN-LUNG
ECRAID
UNITE4TB
Disease Areas
Events
Publications
Guidelines
e-learning
EU Projects
Login
Search
The development of an airway hyperresponsiveness (AHR) depends on mast cells following sensitization without adjuvant
S. Reuter, A. Heinz, S. Züfle, R. Wiewrodt, M. Stassen, C. Taube, R. Buhl (Mainz, Germany)
Source:
Annual Congress 2006 - Cellular immunology in asthma pathogenesis
Session:
Cellular immunology in asthma pathogenesis
Session type:
Oral Presentation
Number:
2156
Disease area:
Airway diseases
Rating:
You must
login
to grade this presentation.
Share or cite this content
Citations should be made in the following way:
S. Reuter, A. Heinz, S. Züfle, R. Wiewrodt, M. Stassen, C. Taube, R. Buhl (Mainz, Germany). The development of an airway hyperresponsiveness (AHR) depends on mast cells following sensitization without adjuvant. Eur Respir J 2006; 28: Suppl. 50, 2156
You must
login
to share this Presentation/Article on Twitter, Facebook, LinkedIn or by email.
Member's Comments
No comment yet.
You must
Login
to comment this presentation.
Related content which might interest you:
Late Breaking Abstract - Implications of treatable traits and treatment choices on exacerbation risk in moderate-severe asthma
Management of Severe Asthma in Pediatric Patients by an Interdisciplinary Team in a Public Hospital Setting.
Respiratory sequelae of preterm birth across the life span
Related content which might interest you:
Local application of FTY720 to the lung abrogates experimental asthma by altering myeloid dendritic cell function
Source: Annual Congress 2006 - Towards understanding the pathogenesis of asthma
Year: 2006
Budesonide prevents but does not reverse sustained airway hyperresponsiveness in mice
Source: Eur Respir J 2008; 32: 970-978
Year: 2008
Oophorectomy decreases airway responsiveness but not eosinophilia in an ovalbumin sensitization murine model of allergy
Source: Annual Congress 2006 - Cell biology of asthma and COPD
Year: 2006
Effects of IL-4Rα blockade on lung inflammation and airway hyperresponsiveness using Mu317RAXMu, a murine surrogate for AMG 317, in a treatment model of cockroach allergen-induced asthma in mice
Source: Annual Congress 2008 - Novel mechanisms in the pathogenesis of pulmonary inflammation
Year: 2008
Increased arginase activity contributes to airway remodelling in chronic allergic asthma
Source: Eur Respir J 2011; 38: 318-328
Year: 2011
TNF-α negatively modulates allergen-induced airway hyperresponsiveness through γδ T cells
Source: Eur Respir J 2002; 20: Suppl. 38, 27s
Year: 2002
Inhaled doxycycline reduces allergen-induced airway inflammation, hyperresponsiveness and remodelling in mice
Source: Annual Congress 2005 - Inflammation and remodelling in allergy and asthma
Year: 2005
Deficiency of MMP-19 promotes allergen-induced eosinophil burden and airway responsiveness in mice
Source: Annual Congress 2007 - Regulation of allergic airway inflammation in animal models of asthma
Year: 2007
The inflammation and the airway hyperresponsiveness in allergic asthma is inhibited by IL-10 dependent mechanism
Source: Annual Congress 2007 - Mouse models of allergic airway inflammation
Year: 2007
The effect of Der f-induced airway inflammation on airway inflammation in a murine model of allergic asthma
Source: Eur Respir J 2004; 24: Suppl. 48, 348s
Year: 2004
Dissociation of the effects of a steroid on inflammation compared to airway hyperresponsiveness (AHR) in a model of asthma in conscious mice
Source: Eur Respir J 2001; 18: Suppl. 33, 30s
Year: 2001
Role of airway responsivenes in asthma monitoring: new horizons
Source: Annual Congress 2006 - PG14 - Noninvasive monitoring of asthma inflammation and treatment
Year: 2006
HIF-1α inhibitor, 2ME2, attenuates airway inflammation and hyperresponsiveness in allergic airway disease
Source: Annual Congress 2008 - Novel therapeutic approaches and immunological mechanisms in airway cell pathobiology
Year: 2008
Experimental specific immunotherapy with timpol decreases allergic sensitization and airway inflammation in murine model of bronchial asthma
Source: Annual Congress 2010 - Animal models of asthma and lung inflammation
Year: 2010
Role of macrophage migration inhibitory factor in ovalbumin-induced airway inflammation in rats
Source: Eur Respir J 2006; 27: 726-734
Year: 2006
Dissociation of airway inflammation and hyperresponsiveness by cyclooxygenase inhibition in allergen challenged mice
Source: Eur Respir J 2009; 34: 200-208
Year: 2009
How innate immunity regulates adaptive immunity
Source: Annual Congress 2006 - Targeting innate immunity in respiratory diseases
Year: 2006
The role of CCR5 in allergen-induced airway hyperresponsiveness and airway inflammation
Source: Annual Congress 2009 - Animal models of airways inflammation
Year: 2009
The inhaled PDE4 inhibitor UK-500,001 does not significantly inhibit airway responses to allergen and histamine
Source: Annual Congress 2007 - New drugs for asthma
Year: 2007
We use cookies on our website to give you the most relevant experience by remembering your preferences and repeat visits. By clicking "Accept", you consent to the use of the cookies.
Accept