Source: Annual Congress 2009 - COPD: basic science and clinical studies
Disease area: Airway diseases



Rating:

Share or cite this content

Citations should be made in the following way:
G. Caramori, P. Casolari, J. Marwick, S. Giuffrè, M. Contoli, G. Gnesini, P. Kirkham, K. F. Chung, P. J. Barnes, I. M. Adcock, A. Papi (Ferrara, Italy; London, United Kingdom). Expression and activation of the major subunit of NF-kB, protein p65, in COPD bronchial glands. Eur Respir J 2009; 34: Suppl. 53, 904

Member's Comments

No comment yet.

Related content which might interest you: siRNA against the p65 subunit of NF-κB modulates the inflammatory response in airway epithelial cells Source: Eur Respir J 2004; 24: Suppl. 48, 437s Year: 2004 Involvement of NFkB activation and c-JUN phosphorylation in bronchial epithelial cells by CD-40-CD40L pathway Source: Eur Respir J 2002; 20: Suppl. 38, 573s Year: 2002 Differential CCAAT/enhancer binding protein (C/EBP) isoforms expression in human bronchial smooth muscle cells of asthmatic and COPD patients Source: Eur Respir J 2004; 24: Suppl. 48, 695s Year: 2004 Egr-1 regulates muc5ac expression through activator protein-1 (AP-1) in chronic airway inflammation diseases Source: International Congress 2015 – Translational studies in airway cell biology Year: 2015 A T cell-specific inhibitor of NF-kappaB and AP-1 suppresses lung fibrosis in mice Source: Eur Respir J 2004; 24: Suppl. 48, 101s Year: 2004 Expression of the nuclear factor-?B inhibitor A20 is altered in the cystic fibrosis epithelium Source: Eur Respir J 2013; 41: 1315-1323 Year: 2013 Thrombin induces stomatin-like protein 2 (STOML2) expression and IL-8/CXCL8 release by ROCK, JNK, and PEA3 pathways in human lung epithelial cells Source: Virtual Congress 2020 – Asthma science: novel targets and mechanisms Year: 2020 Estimation of activity apoptosis genes based on expression of Bcl2, Bax, caspase-3 activity in bronchial epithelium in asthma Source: Annual Congress 2012 - Cell biology and inflammatory gene expression in chronic lung disease Year: 2012 The role of bromodomain-containing protein 4 in the constitutive activation of nuclear factor-kappa B in endothelial cells from patients with pulmonary arterial hypertension Source: Annual Congress 2012 - Pulmonary circulation: basic mechanisms, animal models and cell biology Year: 2012 Protein kinase C and mitochondrial signaling mediate PM10-induced ROS and TGF-β expression in the bronchial epithelial cells Source: Eur Respir J 2007; 30: Suppl. 51, 574s Year: 2007 Induced sputum differential gene expression implicates increased p38 signalling activity in severe asthma Source: Annual Congress 2011 - Molecular: pathology of infectious and inflammatory lung disease Year: 2011 The role of NRF-2 and p38 MAPK on the function of human parenchymal fibroblasts Source: Annual Congress 2013 –Cell signalling and inflammation: what's new in 2013? Year: 2013 Expression of phosphorylated p38 MAPK in the lung of smokers with COPD Source: Eur Respir J 2006; 28: Suppl. 50, 582s Year: 2006 The pan janus kinase (JAK) inhibitor KN-002 suppresses inflammatory mediator release from severe asthma bronchial epithelial cells. Source: Virtual Congress 2021 – Airway pharmacology: translational and basic science Year: 2021 Estimation of activity apoptosis genes based on expression of Bcl2, Bax, CPP32 in bronchial epithelium in asthma Source: Annual Congress 2010 - Novel cellular mechanisms in lung disease Year: 2010 MAPK inhibition modulates Hsp27 phosphorylation and glutathione in lung epithelial cells Source: Eur Respir J 2006; 28: Suppl. 50, 192s Year: 2006 The expression of muscarinic receptors is involved in the p42/44ERK and NF-kB activation of human lung fibroblasts from COPD subjects Source: Eur Respir J 2006; 28: Suppl. 50, 102s Year: 2006 Cigarette smoke inhibits bacteria-induced FABP5 expression by preventing c-Jun binding to FABP5 promoter Source: International Congress 2016 – Translational research in respiratory infections Year: 2016 Subcellular distribution of p65 NF-κB subunit in induced sputum of children with intermittent and moderate asthma Source: Eur Respir J 2005; 26: Suppl. 49, 616s Year: 2005 CRE, but not AP-1, NF-κB, or C/EBP, mediates β-agonist-induced IL-6 gene expression induced in human airway smooth muscle (HASM) cells Source: Eur Respir J 2001; 18: Suppl. 33, 407s Year: 2001