DNA vaccine encoding both FasL and Der p 2 was more effective than DNA vaccine encoding only Der p 2 in suppressing airway inflammation in a mouse model of asthma Source: Eur Respir J 2006; 28: Suppl. 50, 224s Year: 2006
The vaccination with DNA encoding T cell epitope on the Der p inhibited allergic responses in mice Source: Eur Respir J 2002; 20: Suppl. 38, 28s Year: 2002
Co-administration of vaccination with DNA encoding T cell epitope on the Der p and BCG inhibited allergic responses in mice Source: Eur Respir J 2003; 22: Suppl. 45, 483s Year: 2003
Der p 2-derived peptide abrogates HDM-induced allergic asthma in mouse and humanized model Source: International Congress 2019 – Modelling and monitoring of airway diseases Year: 2019
The effect of Der f-induced airway inflammation on airway inflammation in a murine model of allergic asthma Source: Eur Respir J 2004; 24: Suppl. 48, 348s Year: 2004
Cloned Th cells confer airway obstruction upon antigen challenge in the absence of IgE antibody – a model for nonatopic bronchoconstriction Source: Annual Congress 2009 - Animal models of airways inflammation Year: 2009
Effect of plasmid DNA encoding Der p1 and Der p2 on the CD4+ CD25+ regulatory T cells in a mouse model of asthma Source: Eur Respir J 2005; 26: Suppl. 49, 24s Year: 2005
Effectiveness of Antigen85B DNA vaccine on T-helper cells cytokines release and airway inflammation in ovalbumin sensitized mice model Source: Eur Respir J 2004; 24: Suppl. 48, 6s Year: 2004
Immunomodulation of the allergic airway inflammation by allergen DNA targeted receptors for the Fc region on dendritic cells in mice sensitized/challenged by OVA allergen Source: Annual Congress 2007 - Mouse models of allergic airway inflammation Year: 2007
Total IgE and Der p 1 (D1) specific IgE in induced sputum (IS) in patients with allergic and non-allergic asthma Source: International Congress 2018 – Biomarkers for evaluating asthma Year: 2018
The non-proteolytic major house dust mite allergen Der p 2 induce proasthmatic responses in bronchial epithelial cells partly through NF-κB and MAPK pathways Source: Annual Congress 2007 - Mechanisms of inflammation in asthma and rhinitis - from cell cultures to patients Year: 2007
Characterisation of type two immune responses in a murine model of allergic airway disease Source: Virtual Congress 2020 – Scientific advances in airway pharmacology Year: 2020
House dust mite major allergens Der p 1 and Der p 5 activate human airway-derived epithelial cells by protease-dependent and protease-independent mechanisms Source: Eur Respir J 2006; 28: Suppl. 50, 756s Year: 2006
CD4 + T-helper cells engineered to produce IL-10 reverse allergen-induced airway hyperreactivity and inflammation Source: Eur Respir J 2001; 18: Suppl. 33, 432s Year: 2001
Der p 1- and Der p 2-induced experimental asthma show different histology pattern and severity Source: Eur Respir J 2005; 26: Suppl. 49, 24s Year: 2005
Deficiency of MMP-19 promotes allergen-induced eosinophil burden and airway responsiveness in mice Source: Annual Congress 2007 - Regulation of allergic airway inflammation in animal models of asthma Year: 2007
The development of an airway hyperresponsiveness (AHR) depends on mast cells following sensitization without adjuvant Source: Annual Congress 2006 - Cellular immunology in asthma pathogenesis Year: 2006
LATE-BREAKING ABSTRACT: IRF3 is required for the induction of allergic airway inflammation by lung dendritic cells in a mouse model of house dust mite-induced atopic asthma Source: Annual Congress 2009 - Models of chronic airways disease and therapy Year: 2009
Effects of IL-4Rα blockade on lung inflammation and airway hyperresponsiveness using Mu317RAXMu, a murine surrogate for AMG 317, in a treatment model of cockroach allergen-induced asthma in mice Source: Annual Congress 2008 - Novel mechanisms in the pathogenesis of pulmonary inflammation Year: 2008
The major house dust mite allergen Der p2 induces IL-8, GM-CSF and ICAM-1 expression in human bronchial epithelial cells partly by a MAPK kinase dependent pathway Source: Eur Respir J 2005; 26: Suppl. 49, 98s Year: 2005