Interactive inhibitory effects of formoterol and montelukast on activated human neutrophils Source: Eur Respir J 2010; 36: 1417-1424 Year: 2010
Potent anti-inflammatory effects of the highly selective iNOS inhibitor Byk402750 in a mouse model of cigarette smoke induced inflammation Source: Annual Congress 2008 - Novel experimental approaches to understanding asthma and COPD Year: 2008
Liver-X-receptor agonists, but not dexamethasone, inhibit leukotriene production from human white blood cells pretreated with GM-CSF and activated with fMLP Source: Annual Congress 2007 - New treatments for asthma and COPD - experimental evidence Year: 2007
Overexpression of CBP augments cigarette smoke-induced pro-inflammatory cytokine release in macrophages Source: Eur Respir J 2005; 26: Suppl. 49, 105s Year: 2005
TRPA1 mediates cigarette smoke-induced TNFa secretion in macrophages Source: Virtual Congress 2021 – Environmental and lifestyle impacts on chronic lung disease Year: 2021
Curcumin restores glucocorticoid function and inhibits cigarette smoke-mediated IL-8 release in oxidant stressed monocytic U937 cells Source: Eur Respir J 2005; 26: Suppl. 49, 215s Year: 2005
Ionomycin stimulates a synergistic effect of IL-8 and induced sputum on PMN chemotaxis during COPD Source: Annual Congress 2010 - COPD and asthma: models and human studies Year: 2010
A selective MMP-12 inhibitor, MV062926, reduces inflammatory responses to cigarette smoke in mice Source: Annual Congress 2010 - Pre-clinical models of airways disease Year: 2010
Effect of PDE4 inhibitors on MAPK activation and IL-8 production on A549 cells following the induction of oxidative stress Source: Eur Respir J 2005; 26: Suppl. 49, 99s Year: 2005
Cigarette smoke markedly inhibits the LPS-induced production of interleukin-8 in bronchial epithelial cells and consequent neutrophil migration in vitro Source: Eur Respir J 2004; 24: Suppl. 48, 100s Year: 2004
N-Acetylcysteine inhibits eotaxin and MCP-1 release in human airway smooth muscle cells in vitro through the inhibition of ROS production, resulting in a decrease of p38 MAP kinase activation Source: Eur Respir J 2002; 20: Suppl. 38, 170s Year: 2002
Effect of non-steroidal anti-inflammatory drugs on neutrophil recruitment, p38 map kinase phosphorylation and NF-κB activation in lung from mice exposed to cigarette smoke Source: Annual Congress 2007 - Molecular pathogenesis: focus on COPD and asthma Year: 2007
17-beclomethasone monopropionate or inhibition of MAPK p38 signalling abolish IL-8 release stimulated by cigarette smoke extract or acrolein in normal human airway smooth muscle cells Source: Annual Congress 2010 - Airway smooth muscle cells Year: 2010
Inhibition of LPS-induced cytokine/chemokine production from human lung macrophages by roflumilast is enabled by autocrine PGE2 Source: Annual Congress 2010 - Macrophages: cell biology and disease-specific characteristics Year: 2010
Phosphodiesterase 4 and 5 inhibitors decrease HO-1 and IL-8 gene expression induced by cigarette smoke Source: Annual Congress 2008 - Pathophysiology of pulmonary hypertension Year: 2008
Cigarette smoke alters the expression of the pro-inflammatory LTB4 receptor and increases the neutrophil adhesion in bronchial epithelial cells Source: Annual Congress 2011 - Epithelial cell biology Year: 2011
Effects of budesonide on p38 MAPK activation, apoptosis and IL-8 secretion, induced by TNF-alpha and Haemophilus influenzae in human bronchial epithelial cells Source: Annual Congress 2010 - Regulation of airway pharmacology Year: 2010
Extracellular Hsp70 alters cigarette smoke-induced pro-inflammatory responses in human macrophages Source: International Congress 2018 – Translational models of the immunopathology that underlies airway obstructive diseases Year: 2018
The novel phosphodiesterase 4 inhibitor CI-1044 inhibits in vitro LPS-induced TNFα production in whole blood of COPD patients Source: Eur Respir J 2001; 18: Suppl. 33, 34s Year: 2001
Protease activated receptor 2 (PAR2) antagonism reduces pro-inflammatory cytokine production in bronchial epithelial cells Source: Virtual Congress 2020 – Novel immunopathological mechanisms of lung disease: knowledge from translational studies Year: 2020