e-learning
resources
Berlin 2001
Sunday 23.09.2001
Basic mechanisms of lung injury
Login
Search all ERS
e-learning
resources
Disease Areas
Airways Diseases
Interstitial Lung Diseases
Respiratory Critical Care
Respiratory Infections
Paediatric Respiratory Diseases
Pulmonary Vascular Diseases
Sleep and Breathing Disorders
Thoracic Oncology
Events
International Congress
Courses
Webinars
Conferences
Research Seminars
Journal Clubs
Publications
Breathe
Monograph
ERJ
ERJ Open Research
ERR
European Lung White Book
Handbook Series
Guidelines
All ERS guidelines
e-learning
CME Online
Case reports
Short Videos
SpirXpert
Procedure Videos
CME tests
Reference Database of Respiratory Sounds
Radiology Image Challenge
Brief tobacco interventions
EU Projects
VALUE-Dx
ERN-LUNG
ECRAID
UNITE4TB
Disease Areas
Events
Publications
Guidelines
e-learning
EU Projects
Login
Search
Inflammatory mediator release as a consequence of hyperosmolar stimulation of the eosinophil
E. D. Moloney, C. M. Burke, L. W. Poulter, S. O'Sullivan (Dublin, Ireland; London, United Kingdom)
Source:
Annual Congress 2001 - Basic mechanisms of lung injury
Session:
Basic mechanisms of lung injury
Session type:
Thematic Poster Session
Number:
593
Disease area:
Airway diseases
Rating:
You must
login
to grade this presentation.
Share or cite this content
Citations should be made in the following way:
E. D. Moloney, C. M. Burke, L. W. Poulter, S. O'Sullivan (Dublin, Ireland; London, United Kingdom). Inflammatory mediator release as a consequence of hyperosmolar stimulation of the eosinophil. Eur Respir J 2001; 16: Suppl. 31, 593
You must
login
to share this Presentation/Article on Twitter, Facebook, LinkedIn or by email.
Member's Comments
No comment yet.
You must
Login
to comment this presentation.
Related content which might interest you:
Late Breaking Abstract - Implications of treatable traits and treatment choices on exacerbation risk in moderate-severe asthma
Management of Severe Asthma in Pediatric Patients by an Interdisciplinary Team in a Public Hospital Setting.
Respiratory sequelae of preterm birth across the life span
Related content which might interest you:
Mast cell mediator release during refractoriness to mannitol-induced bronchoconstriction
Source: Annual Congress 2009 - Aspects of uncontrolled asthma
Year: 2009
Neutrophil airway influx by platelet-activating factor in asthma: role of adhesion molecules and LTB4 expression
Source: Eur Respir J 2003; 22: 290-297
Year: 2003
Mast cell tryptase induce alarmin release and secretion of pro-inflammatory cytokines from bronchial epithelial cells
Source: International Congress 2018 – Understanding the immunopathology that underlies airway obstructive diseases
Year: 2018
A major inflammatory cytokine TNF-a inhibits the ventilatory response to hypoxia
Source: International Congress 2018 – Triggers of acute lung injury: mechanical ventilation and oxygen levels
Year: 2018
Oral presentation: Sphingosine-1-phosphate administration in vivo induces airway inflammation and hypereactivity in a IgE-dependent manner
Source: Lung Science Conference 2013 - Early origins and mechanisms of chronic lung disease
Year: 2013
Effect of muscarinic receptors inhibition on cytokine release and inflammatory cells infiltration in the airway of cat as an animal model for COPD
Source: Annual Congress 2012 - Animal models of asthma and COPD and late-breaking abstracts on RCT in asthma and COPD
Year: 2012
Thrombin stimulates the release of cytokines and histamine from basophils
Source: Annual Congress 2008 - Asthma: mechanisms and treatment
Year: 2008
Myeloperoxidase modulates lung epithelial responses to pro-inflammatory agents
Source: Eur Respir J 2008; 31: 252-260
Year: 2008
Dysregulation of the “inflammatory reflex” with abnormal neurohumoral activation may contribute to proinflammatory activity driving the progression of COPD
Source: Eur Respir J, 51 (6) 1800806; 10.1183/13993003.00806-2018
Year: 2018
Hypoxia induces IL-18 and neutrophil influx in lung parenchyma possibly mediated by MIP-2
Source: Annual Congress 2012 - The many roads to lung injury
Year: 2012
Effect of hypertonic and isotonic saline inhalation on inflammatory cells, granulocyte mediators and chemokines in induced sputum of COPD patients
Source: Eur Respir J 2006; 28: Suppl. 50, 792s
Year: 2006
Pro-inflammatory cytokines stimulate airway mucus secretion in a rat model of COPD
Source: Eur Respir J 2001; 18: Suppl. 33, 520s
Year: 2001
Regulation of angiopoietin-2 production by inflammatory stimuli
Source: Eur Respir J 2003; 22: Suppl. 45, 586s
Year: 2003
Remicade suppresses cytokine-induced up-regulation of bradykinin receptor-mediated airway smooth muscle contractions
Source: Annual Congress 2005 - Inflammation and the airway smooth muscle: cellular and pharmacological interactions
Year: 2005
Effect of airway provocation with histamine and adenosine 5’-monophosphate on sputum neutrophils and -related mediators
Source: Eur Respir J 2004; 24: Suppl. 48, 442s
Year: 2004
The role of pro-inflammatory cytokine activation in perioperative lung injury
Source: Annual Congress 2004 - Mechanisms and biomarkers of ischaemia-reperfusion-induced lung injury: lessons learnt from lung transplantation
Year: 2004
LSC Abstract – Role For IL-1alpha in viral-induced inflammatory responses in an epithelial-fibroblast co-culture model of the airway mucosa
Source: International Congress 2015 – Viral infections: basic research and clinical perspective
Year: 2015
Arginine levels regulate pro-inflammatory mediator responses by lung epithelial cells
Source: Eur Respir J 2005; 26: Suppl. 49, 554s
Year: 2005
TLR-4 downregulation in COPD impairs cytokine release from monocytes and Th1-lymphocytes in response to bacterial endotoxin
Source: Annual Congress 2009 - What is new in molecular pathology and functional genomics of neoplastic and non-neoplastic lung disease?
Year: 2009
Involvement of nitric oxide, proinflammatory cytokines and effector cells in the inflammatory process in COPD patients
Source: Eur Respir J 2005; 26: Suppl. 49, 54s
Year: 2005
We use cookies on our website to give you the most relevant experience by remembering your preferences and repeat visits. By clicking "Accept", you consent to the use of the cookies.
Accept