COPD pathogenesis is dependent on ferroptotic death of AT2 cells driven by pro-inflammatory Macrophages
A. Jeridi (München, Germany), T. M Conlon (München, Germany), G. Günes Günsel (München, Germany), M. Novikova (München, Germany), S. Doll (München, Germany), N. J Lang (München, Germany), S. E. Verleden (Leuven, Belgium), M. Rehberg (München, Germany), T. Stöger (München, Germany), H. B. Schiller (München, Germany), M. Conrad (München, Germany), A. Yildirim (München, Germany), A. Yildirim (Neuherberg, Germany)
Source: International Congress 2022 – In vivo and in vitro studies of airway diseases
Session: In vivo and in vitro studies of airway diseases
Session type: Thematic Poster
Number: 3174
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A. Jeridi (München, Germany), T. M Conlon (München, Germany), G. Günes Günsel (München, Germany), M. Novikova (München, Germany), S. Doll (München, Germany), N. J Lang (München, Germany), S. E. Verleden (Leuven, Belgium), M. Rehberg (München, Germany), T. Stöger (München, Germany), H. B. Schiller (München, Germany), M. Conrad (München, Germany), A. Yildirim (München, Germany), A. Yildirim (Neuherberg, Germany). COPD pathogenesis is dependent on ferroptotic death of AT2 cells driven by pro-inflammatory Macrophages. 3174
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