e-learning
resources
Stockholm 2002
Sunday 15.09.2002
Cellular reactions in allergy in humans and animals
Login
Search all ERS
e-learning
resources
Disease Areas
Airways Diseases
Interstitial Lung Diseases
Respiratory Critical Care
Respiratory Infections
Paediatric Respiratory Diseases
Pulmonary Vascular Diseases
Sleep and Breathing Disorders
Thoracic Oncology
Events
International Congress
Courses
Webinars
Conferences
Research Seminars
Journal Clubs
Publications
Breathe
Monograph
ERJ
ERJ Open Research
ERR
European Lung White Book
Handbook Series
Guidelines
All ERS guidelines
e-learning
CME Online
Case reports
Short Videos
SpirXpert
Procedure Videos
CME tests
Reference Database of Respiratory Sounds
Radiology Image Challenge
Brief tobacco interventions
EU Projects
VALUE-Dx
ERN-LUNG
ECRAID
UNITE4TB
Disease Areas
Events
Publications
Guidelines
e-learning
EU Projects
Login
Search
Overexpression of eNOS suppresses asthmatic features in a mouse model of allergic asthma
R. Ten Broeke, G. Folkerts, R. De Crom, R. Van Haperen, V. Verwey, T. Leusink-Muis, I. Van Ark, F. De Clerck, F. Nijkamp (Utrecht, Rotterdam, The Netherlands)
Source:
Annual Congress 2002 - Cellular reactions in allergy in humans and animals
Session:
Cellular reactions in allergy in humans and animals
Session type:
Poster Discussion
Number:
300
Disease area:
Airway diseases
Rating:
You must
login
to grade this presentation.
Share or cite this content
Citations should be made in the following way:
R. Ten Broeke, G. Folkerts, R. De Crom, R. Van Haperen, V. Verwey, T. Leusink-Muis, I. Van Ark, F. De Clerck, F. Nijkamp (Utrecht, Rotterdam, The Netherlands). Overexpression of eNOS suppresses asthmatic features in a mouse model of allergic asthma. Eur Respir J 2002; 20: Suppl. 38, 300
You must
login
to share this Presentation/Article on Twitter, Facebook, LinkedIn or by email.
Member's Comments
No comment yet.
You must
Login
to comment this presentation.
Related content which might interest you:
From pulmonary gas exchange to the interpretation of arterial blood gases
Late Breaking Abstract - Implications of treatable traits and treatment choices on exacerbation risk in moderate-severe asthma
Management of Severe Asthma in Pediatric Patients by an Interdisciplinary Team in a Public Hospital Setting.
Related content which might interest you:
MMP-9 deficiency reduces airway inflammation and hyperresponsiveness in a mouse model of asthma
Source: Eur Respir J 2001; 18: Suppl. 33, 20s
Year: 2001
Decreased Smad7 expression in the airways of a mouse model of chronic asthma
Source: Annual Congress 2004 - Animal models of allergic airway disease
Year: 2004
Activated protein C inhibits bronchial inflammation and hyperresponsiveness in a mouse model of asthma
Source: Eur Respir J 2002; 20: Suppl. 38, 618s
Year: 2002
Narirutin inhibits airway inflammation in an allergic mouse model
Source: Annual Congress 2010 - Animal models of asthma and lung inflammation
Year: 2010
Estrogen prevents airway hyperresponsiveness in a murine model of allergic asthma
Source: Eur Respir J 2006; 28: Suppl. 50, 433s
Year: 2006
Analysis of mucus production in mouse models of allergic asthma
Source: Annual Congress 2010 - Asthma: basic science and clinical studies
Year: 2010
ST2 gene knockout attenuates allergic airways disease
Source: Annual Congress 2006 - Immunological mechanisms in cell recruitment to the lungs
Year: 2006
Effects of OVA-induced allergic airway inflammation and remodeling in mice in three distinct asthma models
Source: Annual Congress 2010 - Animal models of asthma and lung inflammation
Year: 2010
Obesity enhances allergen-induced airway inflammation in a murine model of asthma
Source: International Congress 2019 – Modelling and monitoring of airway diseases
Year: 2019
Deficiency of MMP-19 promotes allergen-induced eosinophil burden and airway responsiveness in mice
Source: Annual Congress 2007 - Regulation of allergic airway inflammation in animal models of asthma
Year: 2007
Estrogen prevents airway inflammation and hyperresponsiveness in a murine model of acute allergic asthma
Source: Eur Respir J 2007; 30: Suppl. 51, 360s
Year: 2007
Experimental specific immunotherapy with timpol decreases allergic sensitization and airway inflammation in murine model of bronchial asthma
Source: Annual Congress 2010 - Animal models of asthma and lung inflammation
Year: 2010
Disruption of CD44 variant 6 abolished bronchial hyperresponsiveness, inflammation and mast cell myositis in a murine model of asthma
Source: Eur Respir J 2006; 28: Suppl. 50, 217s
Year: 2006
Suppression of allergic airway inflammation in a mouse model of asthma by exogenous mesenchymal stem cells
Source: Annual Congress 2010 - Animal models of asthma and lung inflammation
Year: 2010
PDE-4-inhibitor rolipram supports treg-mediated suppression in a murine model of allergic airway disease
Source: Annual Congress 2008 - Risk factors for asthma versus asthma control
Year: 2008
IL-33 mRNA in induced sputum cells from allergic rhinitis and asthma patients: a marker of Th-2 phenotype status?
Source: International Congress 2017 – Monitoring asthma control
Year: 2017
A role of fungal extracts on allergic airway inflammation in the ovalbumin-induced asthma model
Source: Annual Congress 2010 - Animal models of asthma and lung inflammation
Year: 2010
Effects of IL-4Rα blockade on lung inflammation and airway hyperresponsiveness using Mu317RAXMu, a murine surrogate for AMG 317, in a treatment model of cockroach allergen-induced asthma in mice
Source: Annual Congress 2008 - Novel mechanisms in the pathogenesis of pulmonary inflammation
Year: 2008
High-level expression of PD-L1 by DC suppresses airway inflammation in OVA-induced allergic asthma
Source: Annual Congress 2010 - Animal models of asthma and lung inflammation
Year: 2010
Identifying eosinophil subsets in a mouse model of pollutant-aggravated allergic asthma
Source: Virtual Congress 2021 – New insight into the immunology of allergies, asthma and COPD: from mouse to man
Year: 2021
We use cookies on our website to give you the most relevant experience by remembering your preferences and repeat visits. By clicking "Accept", you consent to the use of the cookies.
Accept