Effects of HSP70 induction on IL-6 production from murine alveolar epithelial cells pre-treated with endotoxine after subsequent endotoxin challenge Source: Eur Respir J 2005; 26: Suppl. 49, 346s Year: 2005
TLR-4 downregulation in COPD impairs cytokine release from monocytes and Th1-lymphocytes in response to bacterial endotoxin Source: Annual Congress 2009 - What is new in molecular pathology and functional genomics of neoplastic and non-neoplastic lung disease? Year: 2009
Endotoxin-induced IL-17 release by murine T-lymphocytes: interactions with airway macrophages Source: Eur Respir J 2001; 18: Suppl. 33, 408s Year: 2001
Compartmentalized neutrophil recruitment and matrix metalloprotease (MMP)-9 release after airway exposure to endotoxin in humans Source: Eur Respir J 2004; 24: Suppl. 48, 554s Year: 2004
Transgenic mice overexpressing IL-18 display increased IL-13 in the airways but no changes of the airway hyperresponsiveness in a murine model of late-phase asthma: evidence for a dual role of IL-18 on mucosal Th1/Th2 responses Source: Eur Respir J 2002; 20: Suppl. 38, 1s Year: 2002
A role of GM-CSF in the accumulation of neutrophils in the airways caused by IL-17 and TNF-α Source: Eur Respir J 2003; 21: 387-393 Year: 2003
Association of IL-17 transcription and neutrophil activation in human airways following organic dust exposure Source: Eur Respir J 2003; 22: Suppl. 45, 179s Year: 2003
Interferon-gamma (IFN-γ) alters the expression of IP-10 and IL-18 in human aveolar epithelial cells type II. Possible role in the reinforcement of Th1-dominated lung inflammation Source: Eur Respir J 2001; 18: Suppl. 33, 521s Year: 2001
CD101 eosinophil serves as an early response to suppress endotoxin-induced acute lung injury Source: International Congress 2018 – Role of microbial exposure and inflammation in lung injury Year: 2018
Upregulation of the production of TGF-β and IL-4 and downregulation of IL-6 by apoptotic human bronchial epithelial cells: possible role in regulation of epithelial repair following acute lung injury Source: Eur Respir J 2001; 18: Suppl. 33, 477s Year: 2001
Effect of muscarinic receptors inhibition on cytokine release and inflammatory cells infiltration in the airway of cat as an animal model for COPD Source: Annual Congress 2012 - Animal models of asthma and COPD and late-breaking abstracts on RCT in asthma and COPD Year: 2012
Platelet depletion abolishes neutrophil recruitment in non-allergic murine models of acute inflammation Source: Eur Respir J 2006; 28: Suppl. 50, 760s Year: 2006
Characterisation of endogenous IL-33 using a model of Alternaria -induced pulmonary inflammation Source: Annual Congress 2012 - Cellular signalling mechanisms in the airways Year: 2012
Evidence for secretion of IL-18 in asthmatic airway inflammation assessed by endobronchial allergen challenge and its relationship to accumulation of inflammatory cells Source: Eur Respir J 2001; 18: Suppl. 33, 531s Year: 2001
Free, soluble interleukin-17 protein during severe inflammation in human airways Source: Eur Respir J 2002; 19: 534-537 Year: 2002
Neutrophil airway influx by platelet-activating factor in asthma: role of adhesion molecules and LTB4 expression Source: Eur Respir J 2003; 22: 290-297 Year: 2003
Exaggerated IL-6 responses to LPS after viral airway infection in mice is mediated by endogenous TNF-a production Source: Annual Congress 2007 - Immunological function in viral and nonviral pathogens Year: 2007
Concomitant exposure to nicotine and endotoxin in vitro induces murine airway hyperreactivity possibly via nicotine-induced upregulation of toll-like receptors Source: Annual Congress 2011 - New mechanisms in airway disease Year: 2011
GM-CSF and CSF-1 co-regulate lung inflammation and ERMP12+ progenitor recruitment during immune responses to LPS in vivo Source: Eur Respir J 2006; 28: Suppl. 50, 300s Year: 2006
Different inhaled corticosteroids altered lung microbiome and regulating the expression of PAF receptor as well as the phagocytosis function of macrophage Source: International Congress 2019 – Host-microbe interactions in lung disease and exacerbations Year: 2019