e-learning
resources
Vienna 2003
Wednesday 01.10.2003
Proteases at the cutting edge of respiratory disease
Login
Search all ERS
e-learning
resources
Disease Areas
Airways Diseases
Interstitial Lung Diseases
Respiratory Critical Care
Respiratory Infections
Paediatric Respiratory Diseases
Pulmonary Vascular Diseases
Sleep and Breathing Disorders
Thoracic Oncology
Events
International Congress
Courses
Webinars
Conferences
Research Seminars
Journal Clubs
Publications
Breathe
Monograph
ERJ
ERJ Open Research
ERR
European Lung White Book
Handbook Series
Guidelines
All ERS guidelines
e-learning
CME Online
Case reports
Short Videos
SpirXpert
Procedure Videos
CME tests
Reference Database of Respiratory Sounds
Radiology Image Challenge
Brief tobacco interventions
EU Projects
VALUE-Dx
ERN-LUNG
ECRAID
UNITE4TB
Disease Areas
Events
Publications
Guidelines
e-learning
EU Projects
Login
Search
ADAM 33 as a key susceptibility gene for asthma: the contribution of this novel protease to disease pathogenesis
S. T. Holgate (Southampton, United Kingdom)
Source:
Annual Congress 2003 - Proteases at the cutting edge of respiratory disease
Session:
Proteases at the cutting edge of respiratory disease
Session type:
Hot topics
Number:
3479
Disease area:
Airway diseases
Rating:
You must
login
to grade this presentation.
Share or cite this content
Citations should be made in the following way:
S. T. Holgate (Southampton, United Kingdom). ADAM 33 as a key susceptibility gene for asthma: the contribution of this novel protease to disease pathogenesis. Annual Congress 2003 - Proteases at the cutting edge of respiratory disease
You must
login
to share this Presentation/Article on Twitter, Facebook, LinkedIn or by email.
Member's Comments
No comment yet.
You must
Login
to comment this presentation.
Related content which might interest you:
Late Breaking Abstract - Implications of treatable traits and treatment choices on exacerbation risk in moderate-severe asthma
Management of Severe Asthma in Pediatric Patients by an Interdisciplinary Team in a Public Hospital Setting.
Respiratory sequelae of preterm birth across the life span
Related content which might interest you:
Are genetic defects of dienin a key link in the pathogenesis of COPD?
Source: Virtual Congress 2020 – Physiological and cellular mechanisms affecting pulmonary pathologies
Year: 2020
A
BTNL2
gene variant confers to sarcoidosis susceptibility by an increased risk towards the chronic form of the disease
Source: Eur Respir J 2006; 28: Suppl. 50, 148s
Year: 2006
DPP10 protein expression is associated with the pathogenesis of asthma and COPD
Source: Eur Respir J 2005; 26: Suppl. 49, 231s
Year: 2005
Investigating the role of IREB2 genetic variants in susceptibility to COPD
Source: Annual Congress 2012 - Gene-environment treatment and asthma
Year: 2012
Identification of novel pathogenic mechanisms triggered by arginase activation in patients with COPD
Source: International Congress 2016 – Airway response to infection
Year: 2016
T-cell susceptibility to glucocorticoid (GC)-induced apoptosis: the role in asthma pathogenesis and therapy
Source: Eur Respir J 2006; 28: Suppl. 50, 227s
Year: 2006
The crucial role of the expression of negative regulator of gene transcription SOCS5 in asthma pathogenesis
Source: Annual Congress 2013 –Translational research to bridge the need within clinical decision making
Year: 2013
Identification of novel susceptibility genes in ozone-induced inflammation in mice
Source: Eur Respir J 2010; 36: 428
Year: 2010
Safety profile of a novel peptide,PIN201104,with potential to act as a disease modifying,broad spectrum treatment for asthma
Source: International Congress 2018 – Understanding the immunopathology that underlies airway obstructive diseases
Year: 2018
The role of genes coding Glutation-S-Transferase M1 and T1 as risk factors for the development of bronchial asthma
Source: Eur Respir J 2001; 18: Suppl. 33, 502s
Year: 2001
The role of infections in the induction of asthma: genetic and inflammatory drivers
Source: Annual Congress 2007 - PG24 - GRACE full-day course. From cough to asthma: the role of infection
Year: 2007
MiR-320c regulates SERPINA1 expression and is associated with pulmonary disease in alpha-1-antitrypsin deficiency
Source: International Congress 2016 – How the understanding og molecular and genomic crosstalk is helping to diagnose lung disease
Year: 2016
The oncogene c-Src plays a crucial role in the pathogenesis of COPD
Source: Annual Congress 2013 –Molecular biology: state-of-the-art 2013
Year: 2013
Identification of molecular pathways in distinct asthma phenotypes identified in ADEPT
Source: International Congress 2015 – Novel strategies for the management of asthma, COPD and airway infections
Year: 2015
PAH and inflammation: its role in pathogenesis - is it of value to modulate it?
Source: Annual Congress 2005 - Challenges in the biological understanding of pulmonary arterial hypertension
Year: 2005
Latent TGF-ß binding protein 4 is an important factor that influences anti-inflammatory and cell-protective responses in the pathogenesis of pulmonary emphysema
Source: Virtual Congress 2020 – Respiratory homeostasis and structural changes in disease
Year: 2020
Microarrays reveal coordinated expression of ADAMs family of genes in an
in vitro
model of pulmonary fibrosis
Source: Annual Congress 2005 - Progresses in lung fibrosis
Year: 2005
A potential role for extracellular matrix proteins in lung ageing in COPD
Source: International Congress 2016 – How the understanding og molecular and genomic crosstalk is helping to diagnose lung disease
Year: 2016
Pathogenesis of occupational asthma, including genetic factors
Source: Annual Congress 2005 - Occupational asthma
Year: 2005
To the molecular genetic mechanisms of the COPD pathogenesis
Source: International Congress 2018 – Functional genomics and COPD: new challenges
Year: 2018
We use cookies on our website to give you the most relevant experience by remembering your preferences and repeat visits. By clicking "Accept", you consent to the use of the cookies.
Accept