e-learning
resources
Madrid 2019
Monday, 30.09.2019
Identification of new targets and therapeutic strategies for lung disease
Login
Search all ERS
e-learning
resources
Disease Areas
Airways Diseases
Interstitial Lung Diseases
Respiratory Critical Care
Respiratory Infections
Paediatric Respiratory Diseases
Pulmonary Vascular Diseases
Sleep and Breathing Disorders
Thoracic Oncology
Events
International Congress
Courses
Webinars
Conferences
Research Seminars
Journal Clubs
Publications
Breathe
Monograph
ERJ
ERJ Open Research
ERR
European Lung White Book
Handbook Series
Guidelines
All ERS guidelines
e-learning
CME Online
Case reports
Short Videos
SpirXpert
Procedure Videos
CME tests
Reference Database of Respiratory Sounds
Radiology Image Challenge
Brief tobacco interventions
EU Projects
VALUE-Dx
ERN-LUNG
ECRAID
UNITE4TB
Disease Areas
Events
Publications
Guidelines
e-learning
EU Projects
Login
Search
Cigarette smoke extract induces ER stress in human lung fibroblasts from COPD patients and healthy subjects
M. Ryde (Lund, Sweden), R. Hillerström (Lund, Sweden), A. Malm (Lund, Sweden), G. Westergren-Thorsson (Lund, Sweden), L. Bjermer (Lund, Sweden), E. Tufvesson (Lund, Sweden)
Source:
International Congress 2019 – Identification of new targets and therapeutic strategies for lung disease
Session:
Identification of new targets and therapeutic strategies for lung disease
Session type:
Thematic Poster
Number:
2370
Disease area:
Airway diseases
Rating:
You must
login
to grade this presentation.
Share or cite this content
Citations should be made in the following way:
M. Ryde (Lund, Sweden), R. Hillerström (Lund, Sweden), A. Malm (Lund, Sweden), G. Westergren-Thorsson (Lund, Sweden), L. Bjermer (Lund, Sweden), E. Tufvesson (Lund, Sweden). Cigarette smoke extract induces ER stress in human lung fibroblasts from COPD patients and healthy subjects. 2370
You must
login
to share this Presentation/Article on Twitter, Facebook, LinkedIn or by email.
Member's Comments
No comment yet.
You must
Login
to comment this presentation.
Related content which might interest you:
Panel discussion: Diagnostic tools for Obstructive Sleep Apnoea in adults and children
Expert interview: Physiological classification of lung function impairment
Occupational exposures to respiratory diseases: A case-based discussion
Related content which might interest you:
Effects of cigarette smoke extract on
in vitro
differentiated bronchial epithelial cells from subjects with and without COPD
Source: Eur Respir J 2006; 28: Suppl. 50, 854s
Year: 2006
Cigarette smoke extract (CSE) induces clusterin expression in human lung fibroblasts
Source: Eur Respir J 2004; 24: Suppl. 48, 105s
Year: 2004
Reduced airway expression of cytoprotective CC10 in COPD patients and mice exposed to cigarette smoke
Source: Annual Congress 2013 –COPD: experimental biology
Year: 2013
Cigarette smoke alters the EZH2/DAB2IP expression in bronchial epithelial cells. A risk factor for lung cancer in COPD patients
Source: International Congress 2016 – Studies in airway cell biology
Year: 2016
Cigarette smoke down-regulates the expression of β-catenin in primary human lung fibroblasts
Source: Annual Congress 2011 - Lung cell biology
Year: 2011
Acute exposure to second-hand smoke induces endogenous airway acidification and lung function deterioration in healthy controls
Source: Annual Congress 2010 - Asthma: breath biomarkers and asthma control
Year: 2010
COPD fibroblasts are more responsive to cigarette smoke exposure
Source: Annual Congress 2009 - Mechanisms of pulmonary inflammation and fibrosis
Year: 2009
Z-Alpha-1-antitrypsin and cigarette smoke extract impair apoptosis in human bronchial epithelial cells
Source: Eur Respir J 2006; 28: Suppl. 50, 797s
Year: 2006
Comparison of HSP mRNA relative levels in sputum of COPD patients and subjects exposed to wood smoke and/or cigarette smoke.
Source: International Congress 2017 – Novel molecular and genetic targets in COPD
Year: 2017
Effect of whole cigarette smoke exposure and CCSP on MUC5AC mucin production in primary cultures of human bronchial epithelial cells at air-liquid interface of COPD, smokers and control patients
Source: International Congress 2015 – Translational studies in airway cell biology
Year: 2015
Cigarette smoke induces overexpression of cathepsin S in active smokers with and without COPD
Source: International Congress 2018 – COPD: novel pathological mechanisms in the airway epithelium
Year: 2018
Repeated exposure to cigarette smoke extract (CSE) in primary human lung fibroblasts – effects on proliferation rate
Source: Eur Respir J 2006; 28: Suppl. 50, 635s
Year: 2006
Cigarette smoking increases apoptosis of alveolar lymphocytes: data from control group and sarcoidosis patients
Source: Annual Congress 2007 - The versatile aspects of nicotine addiction
Year: 2007
Effects of cigarette smoke extract on human airway smooth muscle cells in COPD
Source: Eur Respir J 2014; 44: 634-646
Year: 2014
Modulatory effect of cigarette smoke on the translation of
CEBPA
and
CEBPB
mRNA in human lung fibroblasts: COPD/emphysema as a translation control disease
Source: Annual Congress 2010 - Lung cell biology and immunology
Year: 2010
Cigarette smoke extract induces the release of extracellular vesicles by airway epithelial cells via cellular carbonyl stress
Source: International Congress 2015 – Immunomodulation: basic science and clinical aspects
Year: 2015
Cigarette smoke extract induces barrier changes and senescence in primary bronchial epithelial cells
Source: International Congress 2019 – Effects of environmental exposures in lung pathology and the underlying mechanisms
Year: 2019
Cigarette smoke significantly reduces proliferation and modulates the expression levels of CEBPα and CEBPβ in cultured human lung fibroblasts
Source: Annual Congress 2008 - Basic science in asthma and COPD II
Year: 2008
Cigarette smoke extract and diesel particles result in the production of pro-inflammatory molecules from human primary lung fibroblast strains and pulmonary epithelial cells
Source: Eur Respir J 2001; 18: Suppl. 33, 413s
Year: 2001
Acute cigarette smoke exposure in mice promotes airway inflammation consistent with the hallmarks of COPD
Source: Eur Respir J 2003; 22: Suppl. 45, 80s
Year: 2003
We use cookies on our website to give you the most relevant experience by remembering your preferences and repeat visits. By clicking "Accept", you consent to the use of the cookies.
Accept