Source: International Congress 2018 – Disease exacerbations from a molecular point of view
Disease area: Airway diseases, Respiratory infections



Rating:

Share or cite this content

Citations should be made in the following way:
S. Kheir (Paris, France), B. Villeret (Paris, France), N. Serafini (Paris, France), R. Voulhoux (Marseille, France), I. Garcia Verdugo (Paris, France), J. Di Santo (Paris, France), J. Sallenave (Paris, France). Pseudomonas aeruginosa LasB subverts innate immunity in alveolar macrophages and epithelial cells. 4984

Member's Comments

No comment yet.

Related content which might interest you: Pseudomonas aeruginosa influences the inflammatory response of primary bronchial epithelial cells to rhinovirus infection Source: Virtual Congress 2020 – Advances in cystic fibrosis: from microbiome to MRI Year: 2020 The role of ceramide in the host defense against pseudomonas aeruginosa infection Source: Eur Respir J 2006; 28: Suppl. 50, 486s Year: 2006 IL-6 and IL-8 production induced by Pseudomonas aeruginosa flagellin in human bronchial epithelial cells. Source: International Congress 2017 – Infection and exacerbation science Year: 2017 Pseudomonas aeruginosa flagellin modulates the immune response in ex vivo lung tissue slices Source: International Congress 2019 – Mechanisms underlying respiratory infection Year: 2019 Mechanisms of induction of neutrophil apoptosis by the pseudomonas aeruginosa exotoxin pyocyanin Source: Eur Respir J 2004; 24: Suppl. 48, 107s Year: 2004 Oral presentation: Neutrophil elastase plays a dual role in host lung defense against Pseudomonas aeruginosa infection Source: Lung Science Conference 2010 - Biology of gene-environment interactions in the lung Year: 2010 ExoU-induced procoagulant activity in Pseudomonas aeruginosa-infected airway cells Source: Eur Respir J 2008; 32: 1591-1598 Year: 2008 Human airway epithelial cell response to Pseudomonas aeruginosa virulence factors preceded or not by Staphylococcus aureus Source: Eur Respir J 2006; 28: Suppl. 50, 191s Year: 2006 Azithromycin protects bronchial epithelial integrity after Pseudomonas aeruginosa infection independent of bactericidal activity Source: Annual Congress 2008 - Best of lung epithelial cells 2008 Year: 2008 Induction of innate immune response in fresh human lung tissue ex vivo following P. aeruginosa infection Source: International Congress 2018 – Cystic fibrosis in paediatric patients: current research Year: 2018 Coinfection of chlamydia pneumoniae and haemophilus influenzae enhances interleukin-8 (IL-8) production in human macrophages and alveolar epithelial cells Source: Eur Respir J 2007; 30: Suppl. 51, 405s Year: 2007 The moraxella catarrhalis induced inflammatory response of pulmonary epithelial cells is mediated by activation of EGFR Source: Annual Congress 2008 - Airway infection and host defence Year: 2008 Biofilm production and adherence to airway epithelial cells of P. aeruginosa strains: effect of N-acetyl-cysteine Source: Eur Respir J 2005; 26: Suppl. 49, 726s Year: 2005 Innate immunity in the lung: how epithelial cells fight against respiratory pathogens Source: Eur Respir J 2004; 23: 327-333 Year: 2004 Streptococcus pneumoniae inhibits Pseudomonas aeruginosa growth on nasal human epithelium in vitro Source: International Congress 2018 – New therapies for respiratory infections: experimental and translational studies Year: 2018 Inflammatory response to infectious pulmonary injury Source: Eur Respir J 2003; 22: 10S-14S Year: 2003 Impact of Pseudomonas aeruginosa infection on Na+ transport in airway epithelia Source: Annual Congress 2007 - The impact of viral and bacterial infection on the respiratory epithelium Year: 2007 Differential effect of M1 and M2 macrophages on airway epithelial cell innate immunity and wound repair Source: International Congress 2018 – Role of microbial exposure and inflammation in lung injury Year: 2018 Muramyldipeptide synergistically increases interleukin-8 secretion from bronchial epithelial cells in a cell culture model of gramnegative infection Source: Eur Respir J 2006; 28: Suppl. 50, 340s Year: 2006 Alveolar macrophages are a more important source of active IL-18 compared to dendritic or monocyte cells in the presence of the bacterial virulence factor SipB Source: Eur Respir J 2002; 20: Suppl. 38, 213s Year: 2002