PD-L1 expression on alveolar macrophages and in plasma of IPF patients – original research

D. Jovanovic (Belgrade, Republic of Serbia), J. Kotur-Stevuljevic (Belgrade, Republic of Serbia), J. Markovic (Belgrade, Republic of Serbia), O. Loncarevic (Belgrade, Republic of Serbia), M. Simon (Cluj Napoca, Romania), M. Roksandic-Milenkovic (Belgrade, Republic of Serbia), I. Vukanic (Belgrade, Republic of Serbia), M. Kontic (Belgrade, Republic of Serbia), N. Samardžic (Belgrade, Republic of Serbia), V. Ceriman (Belgrade, Republic of Serbia), S. Popevic (Belgrade, Republic of Serbia), S. Glumac (Belgrade, Republic of Serbia), V. Škodric-Trifunovic (Belgrade, Republic of Serbia), S. Škodric-Radojevic (Belgrade, Republic of Serbia), B. Ilic (Belgrade, Republic of Serbia)

Source: International Congress 2018 – Genetics in chronic pulmonary diseases
Session: Genetics in chronic pulmonary diseases
Session type: Thematic Poster
Number: 4232
Disease area: Interstitial lung diseases

Congress or journal article abstract

Rating: 0
You must login to grade this presentation.

Share or cite this content

Citations should be made in the following way:
D. Jovanovic (Belgrade, Republic of Serbia), J. Kotur-Stevuljevic (Belgrade, Republic of Serbia), J. Markovic (Belgrade, Republic of Serbia), O. Loncarevic (Belgrade, Republic of Serbia), M. Simon (Cluj Napoca, Romania), M. Roksandic-Milenkovic (Belgrade, Republic of Serbia), I. Vukanic (Belgrade, Republic of Serbia), M. Kontic (Belgrade, Republic of Serbia), N. Samardžic (Belgrade, Republic of Serbia), V. Ceriman (Belgrade, Republic of Serbia), S. Popevic (Belgrade, Republic of Serbia), S. Glumac (Belgrade, Republic of Serbia), V. Škodric-Trifunovic (Belgrade, Republic of Serbia), S. Škodric-Radojevic (Belgrade, Republic of Serbia), B. Ilic (Belgrade, Republic of Serbia). PD-L1 expression on alveolar macrophages and in plasma of IPF patients – original research. 4232

You must login to share this Presentation/Article on Twitter, Facebook, LinkedIn or by email.

Member's Comments

No comment yet.
You must Login to comment this presentation.


Related content which might interest you:
Expression of surface receptors on alveolar macrophages and monocytes from patients with COPD
Source: Eur Respir J 2004; 24: Suppl. 48, 453s
Year: 2004

Reduced expression of CD44 on lung macrophages from patients with COPD
Source: Annual Congress 2010 - Novel markers of lung injury
Year: 2010


Decreased TLR2 gene expression in alveolar macrophages from sarcoidosis patients with Löfgren‘s syndrome
Source: Annual Congress 2007 - COPD, sarcoidosis, idiopathic pulmonary fibrosis, cystic fibrosis, gastro-oesophageal reflux and cough variant asthma - pathogenesis and differential diagnosis
Year: 2007


Decreased reactivity of the alveolar macrophages in relation to ICAM-1 expression after IFNγ stimulation in lung cancer patients
Source: Eur Respir J 2002; 20: Suppl. 38, 276s
Year: 2002

Decreased BCL-2 expression in alveolar macrophages of patients with idiopathic pulmonary fibrosis
Source: Annual Congress 2010 - Biomarkers in diffuse parenchymal lung disease
Year: 2010


Increased SDF-1alpha production from alveolar macrophages in sarcoidosis in vitro
Source: Annual Congress 2008 - Sarcoidosis: pathophysiology and severity
Year: 2008

Pi3kδ expression is increased in peripheral lung macrophages in COPD patients
Source: Annual Congress 2009 - COPD: basic science and clinical studies
Year: 2009

Decreased apoptotic rate of alveolar macrophages of patients with idiopathic pulmonary fibrosis is not associated with difference in expression of fas or fas ligand
Source: Annual Congress 2008 - Role of bronchoalveolar lavage in studying the pathophysiology of interstitial lung disease
Year: 2008

The expression of TSLP in monocyte derived dendritic cells in the interactions between respiratory epithelium and macrophages in asthma, COPD and healthy controls
Source: International Congress 2019 – Novel insights from in vitro and in vivo models of lung disease
Year: 2019


CCR6 is a receptor for CCL18 expressed on human lung fibroblasts from IPF lungs
Source: Annual Congress 2011 - Growth factors and fibrogenesis: are they good, bad or both?
Year: 2011

Increased production of caspase-1 by alveolar macrophages in patients with pulmonary sarcoidosis
Source: Eur Respir J 2002; 20: Suppl. 38, 585s
Year: 2002

LPS-induced TLR4 expression and cytokine release is impaired in monocytes and T-lymphocytes of COPD patients
Source: Annual Congress 2008 - Novel targets in the molecular pathology of lung cancer and COPD
Year: 2008

Increased haemosiderin-laden alveolar macrophages in IPF patients
Source: Annual Congress 2010 - Usefulness of bronchoalveolar lavage in diffuse parenchymal lung disease
Year: 2010


Differential expression of IL-33 and HMGB1 in the lungs of stable cystic fibrosis patients
Source: Eur Respir J 2014; 44: 802-805
Year: 2014


Increased in vitro production of IP-10/CXCL10 and MIG/CXCL9 by alveolar macrophages in sarcoidosis and EAA but not in IPF
Source: Annual Congress 2008 - Role of bronchoalveolar lavage in studying the pathophysiology of interstitial lung disease
Year: 2008

Phenotype polarization of alveolar macrophages in COPD patients
Source: Annual Congress 2010 - Macrophages: cell biology and disease-specific characteristics
Year: 2010

Interferon-g or azathioprine plus corticosteroids do not alter the expression of apoptotic markers in alveolar macrophages of patients with IPF
Source: Annual Congress 2011 - Bronchoalveolar lavage and biomarkers in diffuse parenchymal lung disease
Year: 2011


IL-4/13-induced up-regulation of METEORIN-LIKE (ML) in alveolar macrophages promotes epithelial CCR3 expression
Source: International Congress 2017 – Asthma and allergy: from the bench to the bedside
Year: 2017

Expression of MMP-8 protein in alveolar macrophages is related to bronchial asthma severity and treatment efficacy
Source: Annual Congress 2010 - Cell biology and immunology of lung disease
Year: 2010

IL-18, a novel Th1 cytokine, is up-regulated in pulmonary tuberculosis: expression and production by alveolar epithelial cells type II
Source: Eur Respir J 2002; 20: Suppl. 38, 213s
Year: 2002