e-learning
resources
Paris 2018
Monday, 17.09.2018
Lung cancer: from the bench to the bedside
Login
Search all ERS
e-learning
resources
Disease Areas
Airways Diseases
Interstitial Lung Diseases
Respiratory Critical Care
Respiratory Infections
Paediatric Respiratory Diseases
Pulmonary Vascular Diseases
Sleep and Breathing Disorders
Thoracic Oncology
Events
International Congress
Courses
Webinars
Conferences
Research Seminars
Journal Clubs
Publications
Breathe
Monograph
ERJ
ERJ Open Research
ERR
European Lung White Book
Handbook Series
Guidelines
All ERS guidelines
e-learning
CME Online
Case reports
Short Videos
SpirXpert
Procedure Videos
CME tests
Reference Database of Respiratory Sounds
Radiology Image Challenge
Brief tobacco interventions
EU Projects
VALUE-Dx
ERN-LUNG
ECRAID
UNITE4TB
Disease Areas
Events
Publications
Guidelines
e-learning
EU Projects
Login
Search
Analyzing the role of proteasome activator 200 (PA200) in lung cancer
V. Welk (Munich, Germany), T. Meul (Munich, Germany), C. Lukas (Munich, Germany), D. Bölükbas (Lund, Sweden), I. Koch (Gauting, Germany), M. Lindner (Gauting, Germany), S. Meiners (Munich, Germany)
Source:
International Congress 2018 – Lung cancer: from the bench to the bedside
Session:
Lung cancer: from the bench to the bedside
Session type:
Thematic Poster
Number:
2849
Disease area:
Thoracic oncology
Rating:
You must
login
to grade this presentation.
Share or cite this content
Citations should be made in the following way:
V. Welk (Munich, Germany), T. Meul (Munich, Germany), C. Lukas (Munich, Germany), D. Bölükbas (Lund, Sweden), I. Koch (Gauting, Germany), M. Lindner (Gauting, Germany), S. Meiners (Munich, Germany). Analyzing the role of proteasome activator 200 (PA200) in lung cancer. 2849
You must
login
to share this Presentation/Article on Twitter, Facebook, LinkedIn or by email.
Member's Comments
No comment yet.
You must
Login
to comment this presentation.
Related content which might interest you:
Panel discussion: ICU admission of lung cancer patients – Is COVID-19 a game-changer?
Surgical vs non-surgical treatment of early stage non-small cell lung cancer: here's the fight
N3 hilar sampling decision in the staging of mediastinal lung cancer
Related content which might interest you:
PP138 – Regulation of PA200 in lung cancer
Source: ERS Lung Science Conference 2021
Year: 2021
Regulation of PA200 in lung cancer
Source: Virtual Congress 2021 – Biology of lung cancer: preclinical and translational research
Year: 2021
PAK interacting exchange factor (BPIX-a) overexpression in human lung cancer
Source: Annual Congress 2009 - Biological and clinical markers in lung cancer
Year: 2009
Proapoptotic and anti-proliferative effect of the stable plasminogen activator inhibitor type 1 (PAI-1) mutant on the lung cancer and endothelial cells
Source: Annual Congress 2009 - What is new in molecular pathology and functional genomics of neoplastic and non-neoplastic lung disease?
Year: 2009
Plasminogen activator inhibitor type 1 (PAI-1) is a negative regulator of lung cancer cells growth due to its anti-proteinase activity
Source: Annual Congress 2008 - Novel targets in the molecular pathology of lung cancer and COPD
Year: 2008
Proteinase inhibition regulates the anti-tumor activity of PAI-1 towards lung and prostate cancer cells
Source: Annual Congress 2011 - Lung cancer: molecular pathology and functional genomics
Year: 2011
TGF-beta-dependent inhibitor of DNA protein (Id) expression in lung fibrosis
Source: Eur Respir J 2006; 28: Suppl. 50, 521s
Year: 2006
Caspase recruitment domain containing protein 9 suppresses non-small cell lung cancer proliferation and invasion via inhibiting MAPK/p38 pathway
Source: Virtual Congress 2020 – Molecular alterations and immunology in lung cancer
Year: 2020
Nuclear expression of Y-Box binding protein (YB-1) as a negative prognostic marker in non-small cell lung cancer
Source: Annual Congress 2003 - Lung cancer biology
Year: 2003
LSC 2012 abstract – Effects of the Hedgehog (Hh) pathway inhibitor GDC-0449 on lung cancer cells and cisplatin resistant lung cancer cells are mediated by lung cancer stem cells (SPs)
Source: Annual Congress 2012 - New insights in the pathology of lung cancer
Year: 2012
ONX0912, a novel proteasome inhibitor for the treatment of lung fibrosis?
Source: Annual Congress 2012 - New drug targets and pre-clinical models for respiratory diseases
Year: 2012
Overexpression of inhibitor of DNA-binding proteins and angiogenic markers have higher impact on survival of non small cell lung cancer patients
Source: Annual Congress 2012 - New insights in the pathology of lung cancer
Year: 2012
Expression of the ILK-PINCH-PARVIN (IPP) complex and its binding partner Rsu-1 in human non-small cell lung cancer
Source: International Congress 2016 – Prognostic variables in lung cancer II
Year: 2016
Increased vascular-endothelial growth factor a (VEGF-A) expression and response to angiogenesis inhibitor erlotinib in non-small-cell lung cancer (NSCLC) – hint for a new predictive marker?
Source: Annual Congress 2007 - Therapeutic approaches in thoracic oncology
Year: 2007
CDCA promotes non-small-cell lung cancer (NSCLC) migration by regulating Akt/Erk1/2 signaling pathways
Source: International Congress 2019 – Thoracic oncology: Treatment, diagnostic options and biology
Year: 2019
Long-term proteasome inhibition promotes chemical lung carcinogenesis
Source: Annual Congress 2010 - Pathology of lung cancer
Year: 2010
Inhibition of the effect of epidermal growth factor (EGF) on lung cancer cells. The use of plasmids encoding specific siRNA molecules
Source: International Congress 2015 – Biomarkers, immunology and molecular pathology of lung cancer
Year: 2015
Specific plasmids encoding shRNA molecules directed against growth factor receptors in cell proliferation inhibition and induction phenotype changes relevant to enhancement of anti-tumor immunity. Model of human non-small (NSCLC) and small (SCLC) cell lung cancer lines
Source: Annual Congress 2010 - Pathology of lung cancer
Year: 2010
The mechanism of plasminogen activator inhibitor type-1 (PAI-1) Cys mutants inhibitory activity towards lung capillaries endothelial cells (HMVEC-L)
Source: Eur Respir J 2005; 26: Suppl. 49, 93s
Year: 2005
We use cookies on our website to give you the most relevant experience by remembering your preferences and repeat visits. By clicking "Accept", you consent to the use of the cookies.
Accept