e-learning
resources
Vienna 2003
Sunday 28.09.2003
Models and markers of COPD
Login
Search all ERS
e-learning
resources
Disease Areas
Airways Diseases
Interstitial Lung Diseases
Respiratory Critical Care
Respiratory Infections
Paediatric Respiratory Diseases
Pulmonary Vascular Diseases
Sleep and Breathing Disorders
Thoracic Oncology
Events
International Congress
Courses
Webinars
Conferences
Research Seminars
Journal Clubs
Publications
Breathe
Monograph
ERJ
ERJ Open Research
ERR
European Lung White Book
Handbook Series
Guidelines
All ERS guidelines
e-learning
CME Online
Case reports
Short Videos
SpirXpert
Procedure Videos
CME tests
Reference Database of Respiratory Sounds
Radiology Image Challenge
Brief tobacco interventions
EU Projects
VALUE-Dx
ERN-LUNG
ECRAID
UNITE4TB
Disease Areas
Events
Publications
Guidelines
e-learning
EU Projects
Login
Search
Induction of pro-inflammatory mediator release by cigarette smoke condensate: differential effects on alveolar epithelial cells and macrophages
H. Jardine, K. Donaldson, W. MacNee (Edinburgh, United Kingdom)
Source:
Annual Congress 2003 - Models and markers of COPD
Session:
Models and markers of COPD
Session type:
Thematic Poster Session
Number:
601
Disease area:
Airway diseases
Rating:
You must
login
to grade this presentation.
Share or cite this content
Citations should be made in the following way:
H. Jardine, K. Donaldson, W. MacNee (Edinburgh, United Kingdom). Induction of pro-inflammatory mediator release by cigarette smoke condensate: differential effects on alveolar epithelial cells and macrophages. Eur Respir J 2003; 22: Suppl. 45, 601
You must
login
to share this Presentation/Article on Twitter, Facebook, LinkedIn or by email.
Member's Comments
No comment yet.
You must
Login
to comment this presentation.
Related content which might interest you:
Panel discussion on Basic translational and clinical research – building a career in paediatric pulmonology- experiences from clinicians in lower middle income countries
Panel discussion on ERS Statement – A core outcome set for clinical trials evaluating the management of COPD exacerbations
ERS statement: a core outcome set for clinical trials evaluating the management of COPD exacerbations
Related content which might interest you:
Overexpression of CBP augments cigarette smoke-induced pro-inflammatory cytokine release in macrophages
Source: Eur Respir J 2005; 26: Suppl. 49, 105s
Year: 2005
Cigarette smoke induces the release of CXCL-8 from human bronchial epithelial cells via TLRs and the induction of inflammasome
Source: Annual Congress 2011 - Epithelial cell biology
Year: 2011
Cigarette smoke alters the expression of the pro-inflammatory LTB4 receptor and increases the neutrophil adhesion in bronchial epithelial cells
Source: Annual Congress 2011 - Epithelial cell biology
Year: 2011
Effect of cigarette smoke extract on inflammatory cytokine and chemokine production in human airway smooth muscle cells
Source: Virtual Congress 2021 – Environmental and lifestyle impacts on chronic lung disease
Year: 2021
Extracellular Hsp70 alters cigarette smoke-induced pro-inflammatory responses in human macrophages
Source: International Congress 2018 – Translational models of the immunopathology that underlies airway obstructive diseases
Year: 2018
Cigarette smoke markedly inhibits the LPS-induced production of interleukin-8 in bronchial epithelial cells and consequent neutrophil migration in vitro
Source: Eur Respir J 2004; 24: Suppl. 48, 100s
Year: 2004
Effect of cigarette smoke extract and Th17 cytokine on production of inflammatory cytokines and chemokines in human airway smooth muscle cells
Source: Virtual Congress 2021 – Airway pharmacology: translational and basic science
Year: 2021
Cigarette smoke increases TLR4 expression and modifies LPS mediated responses in airway epithelial cells
Source: Annual Congress 2007 - Epithelial networks in the airways
Year: 2007
Cigarette smoke extract and diesel particles result in the production of pro-inflammatory molecules from human primary lung fibroblast strains and pulmonary epithelial cells
Source: Eur Respir J 2001; 18: Suppl. 33, 413s
Year: 2001
Cigarette smoke upregulating the expression of miR 21 increases IL-8 in airway epithelial cells
Source: International Congress 2018 – New kids on the block: novel molecular pathways and therapeutic targets in chronic lung diseases
Year: 2018
Effect of cigarette smoke on proteasome function in alveolar epithelial cells
Source: Annual Congress 2011 - Epithelial cell biology
Year: 2011
Cigarette smoke modulates rhinovirus-induced airway epithelial cell chemokine production
Source: Eur Respir J 2010; 35: 1256-1263
Year: 2010
α,β-unsaturated aldehydes contained in cigarette smoke elicit release of inflammatory mediators from human macrophages
Source: Annual Congress 2007 - Recent advances in the understanding of lung injury and repair
Year: 2007
Cigarette smoke can increase the neutrophil recruitment in the airways of COPD patients by increasing PGE2 release in alveolar macrophages (AM)
Source: Eur Respir J 2003; 22: Suppl. 45, 85s
Year: 2003
Cigarette smoke exposure disrupts the molecular regulation of mitochondrial metabolism in human bronchial and alveolar epithelial cells
Source: ERS Lung Science Conference 2020
Year: 2020
TRPA1 mediates cigarette smoke-induced TNFa secretion in macrophages
Source: Virtual Congress 2021 – Environmental and lifestyle impacts on chronic lung disease
Year: 2021
Ghrelin inhibits interleukin-6 production induced by cigarette smoke extract in the bronchial epithelial cells via NF-κB pathway
Source: Annual Congress 2013 –Molecular biology: state-of-the-art 2013
Year: 2013
Cigarette smoke exposure decreases CFLAR expression in bronchial epithelium, augmenting susceptibility for cell death and DAMP release
Source: International Congress 2018 – Regenerative and anti-fibrotic approaches
Year: 2018
Cigarette smoke induces a distinct fibrotic signature in bronchial epithelial cells
Source: Annual Congress 2012 - The smoking gun in COPD biology
Year: 2012
Quercetin attenuates inflammation in cigarette smoke stimulated airway epithelial cells: Possible involvement of autophagy
Source: Annual Congress 2013 –New drugs in respiratory medicine
Year: 2013
We use cookies on our website to give you the most relevant experience by remembering your preferences and repeat visits. By clicking "Accept", you consent to the use of the cookies.
Accept