Source: Annual Congress 2004 - In-vitro analysis of cellular response to lung injury



Rating:

Share or cite this content

Citations should be made in the following way:
J. D. McNeilly, L. A. Jimenez, M. F. Clay, W. MacNee, K. Donaldson (Edinburgh, United Kingdom). Welding fumes-induced lung inflammation is caused by soluble transition metals via activation of NF-κB and AP-1. Eur Respir J 2004; 24: Suppl. 48, 754

Member's Comments

No comment yet.

Related content which might interest you: Association of IL-17 transcription and neutrophil activation in human airways following organic dust exposure Source: Eur Respir J 2003; 22: Suppl. 45, 179s Year: 2003 The role of soluble transition metals in the pro-inflammatory effects of an ultra-fine occupational particulate Source: Annual Congress 2003 - Effects of outdoor air pollution in adults Year: 2003 Involvement of inflammation in cadmium induced lung cell proliferation Source: Annual Congress 2010 - Causal factors and mechanisms of outdoor air pollution and nanoparticulates Year: 2010 Inhalation of ultrafine carbon particles triggers biphasic pro-inflammatory response in the mouse lung Source: Eur Respir J 2006; 28: 275-285 Year: 2006 TACE/TGF-?/EGFR regulates CXCL8 in bronchial epithelial cells exposed to particulate matter components Source: Eur Respir J 2011; 38: 1189-1199 Year: 2011 iNOS inhibition reduces lung vascular inflammation and remodelling induced by repeated particulate matter instillation Source: Annual Congress 2010 - Regulation of airway pharmacology Year: 2010 Lung inflammation induces by exposure to particulate matter air pollution causes endothelial dysfunction Source: Annual Congress 2007 - Acute and chronic effects of air pollution Year: 2007 LSC 2010 Abstract: Acute lung inflammation in response to carbon nanoparticle exposure is augmented in the absence of NF-kB Source: Annual Congress 2010 - Emerging mechanisms in lung injury Year: 2010 Evidence for nuclear translocation of MAP kinases and the transcription factors NFκB and AP-1 in human bronchial epithelium in-vivo following exposure to diesel exhaust Source: Eur Respir J 2002; 20: Suppl. 38, 21s Year: 2002 Nrf2 is closely related to enhance bleomycin induced airway inflammatory responses caused by diesel exhaust particles in mice Source: Annual Congress 2012 - Fibrogenesis between epithelial injury and fibroblast proliferation Year: 2012 iNOS inhibition modulates lung tissue and airway remodelling induced by repeated particulate matter instillations Source: Annual Congress 2010 - Regulation of airway pharmacology Year: 2010 Magnesium and endotoxins in organic dust stimulate the activation of human plasma complement factor 3 Source: Eur Respir J 2001; 18: Suppl. 33, 113s Year: 2001 RAGE blocking ameliorates cigarette smoke extract-induced human alveolar tpye II epithelial cell injury via RAGE-mediated redox sensitive DAMP signaling through Nrf2 Source: International Congress 2018 – COPD: immune regulation and therapeutic targets Year: 2018 Glutathione redox regulates TGF-β-induced Smad activation and fibrogenic effect in human airway Source: Eur Respir J 2007; 30: Suppl. 51, 496s Year: 2007 iNOS inhibition modulates lung tissue inflammation via oxidative stress pathway activation Source: Annual Congress 2008 - Inflammatory trigger mechanisms in asthma and COPD Year: 2008 Particulate matter promotes epithelial-to-mesenchymal transition in human lung epithelial cells via ROS pathway Source: International Congress 2016 – Occupational and environmental exposures and biomarkers: human studies, animal data, and in vitro methods Year: 2016 Cigarette smoke extracts decrease basal and lipopolysaccharide-induced expression of macrophage receptor with collagenous structure via degradation of p300 Source: International Congress 2018 – Disease exacerbations from a molecular point of view Year: 2018 Inhaled cationic salts modulate macrophage function to reduce inflammation during LPS induced lung injury Source: Annual Congress 2012 - Animal models of asthma and COPD and late-breaking abstracts on RCT in asthma and COPD Year: 2012 Amifostine reduces lipopolysaccharide-induced lung dysfunction via suppression of redox-sensitive inflammatory signaling Source: Annual Congress 2008 - Animal models of pulmonary fibrosis: mechanisms and therapies Year: 2008 Expression and role of EGFR ligands induced in airway cells by PM2.5 and its components Source: Eur Respir J 2007; 30: 1064-1073 Year: 2007