e-learning
resources
Glasgow 2004
Sunday 05.09.2004
Inflammation and tissue repair in pulmonary disease
Login
Search all ERS
e-learning
resources
Disease Areas
Airways Diseases
Interstitial Lung Diseases
Respiratory Critical Care
Respiratory Infections
Paediatric Respiratory Diseases
Pulmonary Vascular Diseases
Sleep and Breathing Disorders
Thoracic Oncology
Events
International Congress
Courses
Webinars
Conferences
Research Seminars
Journal Clubs
Publications
Breathe
Monograph
ERJ
ERJ Open Research
ERR
European Lung White Book
Handbook Series
Guidelines
All ERS guidelines
e-learning
CME Online
Case reports
Short Videos
SpirXpert
Procedure Videos
CME tests
Reference Database of Respiratory Sounds
Radiology Image Challenge
Brief tobacco interventions
EU Projects
VALUE-Dx
ERN-LUNG
ECRAID
UNITE4TB
Disease Areas
Events
Publications
Guidelines
e-learning
EU Projects
Login
Search
Glucocorticoid-induced reversal of cytokine-afforded eosinophil survival is mediated via transactivation whereas promotion of neutrophil survival occurs through transrepression
M. Janka, E. Moilanen, I. Adcock, X. Zhang, H. Kankaanranta (Tampere, Finland; London, United Kingdom)
Source:
Annual Congress 2004 - Inflammation and tissue repair in pulmonary disease
Session:
Inflammation and tissue repair in pulmonary disease
Session type:
Poster Discussion
Number:
356
Disease area:
Airway diseases
Rating:
You must
login
to grade this presentation.
Share or cite this content
Citations should be made in the following way:
M. Janka, E. Moilanen, I. Adcock, X. Zhang, H. Kankaanranta (Tampere, Finland; London, United Kingdom). Glucocorticoid-induced reversal of cytokine-afforded eosinophil survival is mediated via transactivation whereas promotion of neutrophil survival occurs through transrepression. Eur Respir J 2004; 24: Suppl. 48, 356
You must
login
to share this Presentation/Article on Twitter, Facebook, LinkedIn or by email.
Member's Comments
No comment yet.
You must
Login
to comment this presentation.
Related content which might interest you:
Late Breaking Abstract - Implications of treatable traits and treatment choices on exacerbation risk in moderate-severe asthma
Management of Severe Asthma in Pediatric Patients by an Interdisciplinary Team in a Public Hospital Setting.
Respiratory sequelae of preterm birth across the life span
Related content which might interest you:
Glucocorticoid-induced neutrophil survival is potentiated by β
2
-agonists
Source: Eur Respir J 2005; 26: Suppl. 49, 123s
Year: 2005
TNF-α inhibits human eosinophil apoptosis by activating p55 receptor and NF-κB
Source: Eur Respir J 2001; 18: Suppl. 33, 162s
Year: 2001
Amifostine reduces lipopolysaccharide-induced lung dysfunction via suppression of redox-sensitive inflammatory signaling
Source: Annual Congress 2008 - Animal models of pulmonary fibrosis: mechanisms and therapies
Year: 2008
GB001 potently inhibits PGD2 metabolite-induced DP2-mediated cell signaling and eosinophil activation
Source: Virtual Congress 2020 – Asthma science: novel targets and mechanisms
Year: 2020
Inhibition of LPS-induced neutrophil recruitment in the lungs correlates with modulation of neutrophil CD11b expression using the selective CXCR2 receptor antagonist SB-656933
Source: Eur Respir J 2004; 24: Suppl. 48, 218s
Year: 2004
Roflumilast potently inhibits CXCL1 mediated neutrophil migration in COPD patients
Source: Annual Congress 2011 - COPD: clinical studies and animal cell models
Year: 2011
Glucocorticoids promote TGF-β signalling in lung fibroblasts by upregulating ligand and receptor expression, and uncoupling negative-feedback inhibition
Source: Annual Congress 2010 - Recent developments of molecular pathology and functional genomics in non-neoplastic and neoplastic lung diseases
Year: 2010
Inhibition of osteopontin modulates tumor-stimulated immune response and suppresses mesothelioma progression
Source: Annual Congress 2012 - Biology and treatment of malignant pleural effusions
Year: 2012
Depletion of STAT6 suppresses lung cancer progression through regulating M2-macrophage polarization
Source: International Congress 2018 – Lung cancer: from the bench to the bedside
Year: 2018
Fas activation impairs the alveolar epithelial function in mice by mechanisms involving apoptosis
Source: Annual Congress 2011 - Anti-inflammatory strategies in acute lung injury
Year: 2011
A T cell-specific inhibitor of NF-kappaB and AP-1 suppresses lung fibrosis in mice
Source: Eur Respir J 2004; 24: Suppl. 48, 101s
Year: 2004
Remifentanil attenuates LPS-induced neutrophil activation
Source: Annual Congress 2012 - Infections and sepsis in the ICU
Year: 2012
G-protein mediated EGFR transactivation induces mucin production
Source: Annual Congress 2003 - Cellular and molecular mechanisms in epithelium injury and repair
Year: 2003
TGFβ overproduction in asthma reduces the epithelial repair by down-regulating the leptin/leptin receptor pathway
Source: Eur Respir J 2007; 30: Suppl. 51, 59s
Year: 2007
Modulation of neutrophil activation and survival by bronchial epithelial cells
Source: Eur Respir J 2004; 24: Suppl. 48, 437s
Year: 2004
NTHi-induced inflammation activates the PD-1 immune checkpoint in the tumor microenvironment
Source: International Congress 2018 – Lung cancer: novel molecular markers and mechanisms
Year: 2018
Bile acid receptor accelerates to the lung cancer process induced by lung fibroblast-tumor cells interaction, with high activation of phosphorylated STAT3 and alteration of cytokine secretion
Source: International Congress 2017 – Lung cancer - basic science: the latest results from the bench
Year: 2017
Decreased PTEN amplifies PI3K signaling and enhances pro-inflammatory cytokine release in COPD
Source: International Congress 2016 – Studies in airway cell biology
Year: 2016
Interleukin-12 receptor mRNA expression and corticosteroid depended eosinophil apoptosis in asthma
Source: Eur Respir J 2003; 22: Suppl. 45, 223s
Year: 2003
We use cookies on our website to give you the most relevant experience by remembering your preferences and repeat visits. By clicking "Accept", you consent to the use of the cookies.
Accept