Expression of MIC-A in bronchial epithelium associates with cigarette smoking: a sign of induced autoimmune mechanisms?

M. Orozco-Levi, B. Abeijón, C. Coronell, A. Ramirez-Sarmiento, J. M. Broquetas, V. Curull (Barcelona, Spain)

Source: Annual Congress 2005 - Lung structure: current concepts
Session: Lung structure: current concepts
Session type: Oral Presentation
Number: 4559
Disease area: Airway diseases

Congress or journal article abstractSlide presentation

Rating: 0
You must login to grade this presentation.

Share or cite this content

Citations should be made in the following way:
M. Orozco-Levi, B. Abeijón, C. Coronell, A. Ramirez-Sarmiento, J. M. Broquetas, V. Curull (Barcelona, Spain). Expression of MIC-A in bronchial epithelium associates with cigarette smoking: a sign of induced autoimmune mechanisms?. Eur Respir J 2005; 26: Suppl. 49, 4559

You must login to share this Presentation/Article on Twitter, Facebook, LinkedIn or by email.

Member's Comments

No comment yet.
You must Login to comment this presentation.


Related content which might interest you:
Airway epithelial cell apoptosis and inflammation in COPD, smokers and nonsmokers
Source: Eur Respir J 2013; 41: 1058-1067
Year: 2013



The impact of smoking cessation on respiratory symptoms, lung function, airway hyperresponsiveness and inflammation
Source: Eur Respir J 2004; 23: 464-476
Year: 2004



CTLA4 gene polymorphisms are associated with chronic bronchitis
Source: Eur Respir J 2009; 34: 598
Year: 2009



Inflammation in COPD: a link to systemic comorbidities
Source: Eur Respir Rev 2007; 16: 91-97
Year: 2007



Cigarette smoke-induced pulmonary inflammation, but not airway wall remodeling, is attenuated in CCR5 deficient mice
Source: Annual Congress 2007 - The best of airway epithelial cells
Year: 2007



Prognosis: what is the contribution of inflammation to the decline in lung function in airway disease?
Source: Annual Congress 2007 - Inflammatory phenotypes in airway disease
Year: 2007


Differences in the polarization of the inflammatory response of patients with chronic obstructive pulmonary disease (COPD) secondary to smoking and to biomass smoke exposure
Source: Annual Congress 2013 –COPD: new markers of diseases
Year: 2013


Epidemiology of chronic obstructive pulmonary disease
Source: Eur Respir J 2001; 17: 982-994
Year: 2001



Cigarette smoke induced pulmonary emphysema is attenuated in CCR6 deficient mice
Source: Annual Congress 2006 - Cell biology of asthma and COPD
Year: 2006


Why is it important to consider (sub)phenotypes of asthma and COPD?
Source: Annual Congress 2005 - Inflammatory phenotypes of asthma and COPD: the end of traditional diagnosis, or not?
Year: 2005

Identification of oxidative stress markers and their association with inflammation in smokers and early chronic obstructive pulmonary disease (COPD)
Source: International Congress 2016 – Hospitalisation and comorbidities in chronic lung diseases
Year: 2016

Characterisation of a sub-acute mouse model of cigarette smoke extract-induced lung inflammation
Source: Annual Congress 2007 - The best of airway epithelial cells
Year: 2007


Genetic susceptibility to chronic obstructive pulmonary disease
Source: ISSN=ISSN 1025-448x, ISBN=ISBN 1-904097-48-0, page=84
Year: 2006

Cigarette smoke induces peribronchial lymphoid follicles in the absence of TLR4 signalling
Source: Annual Congress 2006 - Cell biology of asthma and COPD
Year: 2006


Impact of cigarette smoke exposure on host–bacterial pathogen interactions
Source: Eur Respir J 2012; 39: 467-477
Year: 2012



Overview of smoking-related disease
Source: Research Seminar 2006 - Complexity of patients with multiple chronic diseases
Year: 2006



Airway mucosal inflammation in COPD is similar in smokers and ex-smokers: a pooled analysis
Source: Eur Respir J 2007; 30: 467-471
Year: 2007



The role smoking in development of dust-indusde chronic obstructive pulmonary disease (COPD)
Source: Eur Respir J 2004; 24: Suppl. 48, 608s
Year: 2004

Role of lymphotoxin-alpha in cigarette smoke-induced inflammation and lymphoid neogenesis
Source: Eur Respir J 2009; 33: 405-416
Year: 2009