e-learning
resources
Amsterdam 2015
Wednesday, 30.09.2015
Asthma mechanisms and management
Login
Search all ERS
e-learning
resources
Disease Areas
Airways Diseases
Interstitial Lung Diseases
Respiratory Critical Care
Respiratory Infections
Paediatric Respiratory Diseases
Pulmonary Vascular Diseases
Sleep and Breathing Disorders
Thoracic Oncology
Events
International Congress
Courses
Webinars
Conferences
Research Seminars
Journal Clubs
Publications
Breathe
Monograph
ERJ
ERJ Open Research
ERR
European Lung White Book
Handbook Series
Guidelines
All ERS guidelines
e-learning
CME Online
Case reports
Short Videos
SpirXpert
Procedure Videos
CME tests
Reference Database of Respiratory Sounds
Radiology Image Challenge
Brief tobacco interventions
EU Projects
VALUE-Dx
ERN-LUNG
ECRAID
UNITE4TB
Disease Areas
Events
Publications
Guidelines
e-learning
EU Projects
Login
Search
The capacity of human airway epithelium to regulate T cell proliferation is reduced in asthma
Irene Heijink (Groningen, Netherlands), Irene Heijink, Paul Fitch, Janneke Heimweg, Claire Wilson, Reina Matsuda, Maarten Van den Berge, Antoon Van Oosterhout, Jurgen Schwarze
Source:
International Congress 2015 – Asthma mechanisms and management
Session:
Asthma mechanisms and management
Session type:
Poster Discussion
Number:
5094
Disease area:
Airway diseases
Rating:
You must
login
to grade this presentation.
Share or cite this content
Citations should be made in the following way:
Irene Heijink (Groningen, Netherlands), Irene Heijink, Paul Fitch, Janneke Heimweg, Claire Wilson, Reina Matsuda, Maarten Van den Berge, Antoon Van Oosterhout, Jurgen Schwarze. The capacity of human airway epithelium to regulate T cell proliferation is reduced in asthma. Eur Respir J 2015; 46: Suppl. 59, 5094
You must
login
to share this Presentation/Article on Twitter, Facebook, LinkedIn or by email.
Member's Comments
No comment yet.
You must
Login
to comment this presentation.
Related content which might interest you:
Late Breaking Abstract - Implications of treatable traits and treatment choices on exacerbation risk in moderate-severe asthma
Management of Severe Asthma in Pediatric Patients by an Interdisciplinary Team in a Public Hospital Setting.
Respiratory sequelae of preterm birth across the life span
Related content which might interest you:
Human lung mast cells alter asthmatic airway epithelial cell function
Source: Annual Congress 2013 –The pulmonary epithelium: recent developments
Year: 2013
Regulation of GROα production in human bronchial epithelial cells
Source: International Congress 2016 – Laboratory tests and cell biology in allergy and immunology
Year: 2016
Differential expression of uPAR in cultured bronchial epithelial cells from asthma patients
Source: International Congress 2015 – Translational studies in airway cell biology
Year: 2015
Inhibition of asthma-related immunological responses in mast cells and Th2 cell lines by cultured epithelial cell lines
Source: International Congress 2016 – Mechanisms of disease
Year: 2016
The role of WNT5a in Th17 asthma
Source: International Congress 2016 – How the understanding og molecular and genomic crosstalk is helping to diagnose lung disease
Year: 2016
Human mast cells and type 2 innate lymphoid cells activate gene expression and mucus composition in human bronchial epithelial cells via IL-13
Source: International Congress 2014 – Mechanistic studies of the airway epithelium
Year: 2014
Altered airway epithelial cell specification in COPD
Source: International Congress 2015 – Translational studies in airway cell biology
Year: 2015
Periostin: A key contributor to changes in the epithelial phenotype in asthma?
Source: International Congress 2015 – Clincal aspects of asthma: monitoring and mechanisms
Year: 2015
SIRT6-induced activation of autophagy inhibits CSE-induced bronchial epithelial cell senescence
Source: Annual Congress 2013 –New insights into epigenetic mechanisms in the lung
Year: 2013
IL-4 induced changes in the Wnt signalling of bronchial epithelial cells
Source: International Congress 2015 – Asthma mechanisms and management
Year: 2015
Mitochondrial function in three different types of airway epithelial cells
Source: International Congress 2015 – Translational studies in airway cell biology
Year: 2015
Airway epithelial cells modulate dendritic cell differentiation and maturation
Source: Annual Congress 2007 - The best of airway epithelial cells
Year: 2007
DP2/CRTh2 is expressed by ASM cells in asthma and its inhibition suppresses ASM migration
Source: International Congress 2016 – Mechanisms of disease
Year: 2016
Leukotriene E4 affects expression of genes involved in inflammatory, remodelling and mitogenic responses in human bronchial epithelial cells
Source: International Congress 2016 – Novel insights into alveolar and bronchial epithelial cell injury and repair
Year: 2016
Inhibition of IRAK4 suppresses chemokine release from human bronchial epithelial cells
Source: Annual Congress 2013 –Molecular biology: state-of-the-art 2013
Year: 2013
Regulation and role of notch signaling in epithelial progenitor cell differentiation and proliferation in the normal and the fibrotic lung
Source: Annual Congress 2013 –Idiopathic pulmonary fibrosis: pathogenesis
Year: 2013
TGFβ-induced epithelial to mesenchymal transition (EMT) as a model of COPD
Source: International Congress 2016 – Novel insights into alveolar and bronchial epithelial cell injury and repair
Year: 2016
CEACAM6-high airway neutrophils and epithelial cells are a feature of severe asthma
Source: International Congress 2015 – Translational studies in airway cell biology
Year: 2015
Human bronchial epithelium orchestrates dendritic cell activation in severe asthma
Source: Eur Respir J , 49 (3) 1602399; DOI: 10.1183/13993003.02399-2016
Year: 2017
Association between T-regulatory cells blood content and Th2 cells activity in asthma
Source: Annual Congress 2013 –Asthma: new mechanisms and markers
Year: 2013
We use cookies on our website to give you the most relevant experience by remembering your preferences and repeat visits. By clicking "Accept", you consent to the use of the cookies.
Accept