Reactive oxygen species regulate autophagy in COPD myotubes in vitro

Pascal Pomiès (Montpellier, France), Pascal Pomiès, Marine Blaquière, Fares Gouzi, Jonathan Maury, Jacques Mercier, Maurice Hayot

Source: International Congress 2015 – New insights into exercise and muscle performance
Session: New insights into exercise and muscle performance
Session type: Poster Discussion
Number: 4604
Disease area: Airway diseases

Congress or journal article abstract

Rating: 0
You must login to grade this presentation.

Share or cite this content

Citations should be made in the following way:
Pascal Pomiès (Montpellier, France), Pascal Pomiès, Marine Blaquière, Fares Gouzi, Jonathan Maury, Jacques Mercier, Maurice Hayot. Reactive oxygen species regulate autophagy in COPD myotubes in vitro. Eur Respir J 2015; 46: Suppl. 59, 4604

You must login to share this Presentation/Article on Twitter, Facebook, LinkedIn or by email.

Member's Comments

No comment yet.
You must Login to comment this presentation.


Related content which might interest you:
Mitochondrial reactive oxygen species and glycolysis in airway smooth muscle cell proliferation in COPD
Source: International Congress 2015 – Advances in the future treatment of COPD
Year: 2015

Mitochondrial reactive oxygen species (ROS) mediate proliferation and cytokine release in airway smooth muscle cells of patients with COPD
Source: International Congress 2014 – Cell biology 2014
Year: 2014

Role of exosomal microRNA in driving skeletal muscle wasting in COPD
Source: International Congress 2015 – Latest news on miRNAs in pulmonary research
Year: 2015


Effect of oxidative stress on mitochondrial function in airway smooth muscle (ASM) cells from COPD patients
Source: Annual Congress 2013 –Airway smooth muscle and cell biology
Year: 2013

Peripheral muscle mitophagy is enhanced in patients with COPD
Source: International Congress 2016 – How the understanding og molecular and genomic crosstalk is helping to diagnose lung disease
Year: 2016

Altered mitochondrial reactive oxygen species (ROS) production in airway smooth muscle cells of severe asthma.
Source: International Congress 2019 – Novel mechanisms elucidated by translational studies of asthma
Year: 2019

Site of ROS production by mitochondria of skeletal muscle of patients with COPD
Source: Annual Congress 2011 - Physiological basis of respiratory disease
Year: 2011


Impaired coordination of skeletal muscle protein turnover signalling in sarcopenic COPD patients
Source: International Congress 2016 – How the understanding og molecular and genomic crosstalk is helping to diagnose lung disease
Year: 2016

Cultured satellite cells as a model for the study of muscle dysfunction in COPD
Source: Annual Congress 2013 –The best posters in exercise testing and physical inactivity
Year: 2013

TNF-α-induced NF-κB activation stimulates skeletal muscle glycolytic metabolism through activation of HIF-1α
Source: International Congress 2015 – New insights into lung disease pathogenesis
Year: 2015

p53 dysfunction increased mitochondrial biogenesis and bronchial smooth muscle cell proliferation in asthma
Source: International Congress 2015 – Airway remodelling: recent developments
Year: 2015


Role of complex I and III in the production of reactive oxygen species by mitochondria of peripheral muscle in COPD subjects
Source: Annual Congress 2009 - Gas exchange and exercise in respiratory diseases
Year: 2009

Targeting ASK1 in preventing airway smooth muscle growth: Implications for airway remodeling in COPD
Source: International Congress 2016 – New findings in mucosal immunology
Year: 2016


Activation of skeletal muscle mitophagy in a murine model of pulmonary and systemic inflammation
Source: International Congress 2016 – How the understanding og molecular and genomic crosstalk is helping to diagnose lung disease
Year: 2016

Investigating mitochondrial dysfunction in asthma
Source: International Congress 2015 – Advances in the future treatment of severe asthma
Year: 2015


DP2/CRTh2 is expressed by ASM cells in asthma and its inhibition suppresses ASM migration
Source: International Congress 2016 – Mechanisms of disease
Year: 2016

The key role of microRNA-34a in the reduction of sirtuin-1 in COPD
Source: International Congress 2016 – Emerging concepts in lung disease pathogenesis
Year: 2016

Targeting the mTOR signaling pathway to inhibit lung cell senescence in COPD
Source: International Congress 2016 – New signalling pathways in COPD
Year: 2016

Mitochondrial membrane potential in the airway and skeletal muscle compartments of smokers with and without COPD
Source: International Congress 2014 – Translational studies in COPD
Year: 2014