e-learning
resources
Amsterdam 2015
Tuesday, 29.09.2015
New models for treating airway diseases
Login
Search all ERS
e-learning
resources
Disease Areas
Airways Diseases
Interstitial Lung Diseases
Respiratory Critical Care
Respiratory Infections
Paediatric Respiratory Diseases
Pulmonary Vascular Diseases
Sleep and Breathing Disorders
Thoracic Oncology
Events
International Congress
Courses
Webinars
Conferences
Research Seminars
Journal Clubs
Publications
Breathe
Monograph
ERJ
ERJ Open Research
ERR
European Lung White Book
Handbook Series
Guidelines
All ERS guidelines
e-learning
CME Online
Case reports
Short Videos
SpirXpert
Procedure Videos
CME tests
Reference Database of Respiratory Sounds
Radiology Image Challenge
Brief tobacco interventions
EU Projects
VALUE-Dx
ERN-LUNG
ECRAID
UNITE4TB
Disease Areas
Events
Publications
Guidelines
e-learning
EU Projects
Login
Search
Andrographolide restores steroid sensitivity to block LPS/IFN-γ-induced IL-27 and airway hyperresponsiveness in mice
Wupeng Liao (Singapore, Singapore), Wupeng Liao, Wan Shun Daniel Tan, Wai-Shiu Fred Wong
Source:
International Congress 2015 – New models for treating airway diseases
Session:
New models for treating airway diseases
Session type:
Poster Discussion
Number:
3566
Disease area:
Airway diseases
Rating:
You must
login
to grade this presentation.
Share or cite this content
Citations should be made in the following way:
Wupeng Liao (Singapore, Singapore), Wupeng Liao, Wan Shun Daniel Tan, Wai-Shiu Fred Wong. Andrographolide restores steroid sensitivity to block LPS/IFN-γ-induced IL-27 and airway hyperresponsiveness in mice. Eur Respir J 2015; 46: Suppl. 59, 3566
You must
login
to share this Presentation/Article on Twitter, Facebook, LinkedIn or by email.
Member's Comments
No comment yet.
You must
Login
to comment this presentation.
Related content which might interest you:
Observational cohort study of pulmonary exacerbations in alpha-1 antitrypsin deficiency
Asbestosis
The impact of anti-IgE treatment on the airway microbiome in asthma.
Related content which might interest you:
The effect of trehalose for the allergen-induced airway hyperresponsiveness and airway inflamation in mice
Source: Annual Congress 2013 –Cell biology and murine models of asthma and inflammation
Year: 2013
IL-13 in a mouse model of chemical-induced airway hyperresponsiveness
Source: International Congress 2015 – Immune mechanisms in the human lung
Year: 2015
A novel inhaled PI3Kδ inhibitor, LAS194223 reduces allergen-induced airway inflammation in rat
Source: International Congress 2014 – New drugs for asthma, COPD and pulmonary fibrosis
Year: 2014
Ciclesonide and tiotropium synergistically inhibit airway eosinophilia in a guinea pig model of chronic asthma
Source: Annual Congress 2013 –Novel mechanisms for established drugs for asthma and COPD management
Year: 2013
BmTI-A: A serine protease inhibitor attenuates hyperresponsiveness in an experimental model of chronic allergic inflammation in Balb/c mice
Source: International Congress 2014 – Animal models in respiratory drug development
Year: 2014
Nicotine impairs inflammation-induced airway relaxation in a in vitro murine model of asthma
Source: Annual Congress 2013 –The role of hormones and cytokines in lung injury
Year: 2013
Repeated lipopolysaccharide stimulation causes corticosteroid insensitive inflammation in lung via phosphoinositide-3-kinase δ signaling
Source: International Congress 2014 – New treatments for cough, asthma, COPD and ILDs
Year: 2014
Influenza induced exacerbation is associated with steroid insensitive AHR and pulmonary inflammation in HDM-sensitised mice
Source: International Congress 2016 – Immune responses in the lung
Year: 2016
Characteristics of IL-25 and allergen-induced airways fibrosis in a murine model of asthma
Source: International Congress 2015 – The immunology of allergic airway disease
Year: 2015
Animal models of airway hyperresponsiveness
Source: Eur Respir J 2009; 33: 217-218
Year: 2009
Species comparison of interleukin-13 induced airway hyperreactivity as model for allergic asthma
Source: International Congress 2014 – Animal models in respiratory drug development
Year: 2014
Schistosome-induced pulmonary B cells inhibit allergic airway inflammation and display a reduced Th2-driving function
Source: International Congress 2015 – Animal studies of asthma and other inflammatory conditions
Year: 2015
CRAC channel inhibition by RP3128 ameliorates airway hyper-reactivity in antigen sensitized mice and guinea pigs
Source: International Congress 2014 – Novel targets and drugs for asthma and COPD
Year: 2014
LATE-BREAKING ABSTRACT: Inhibition of TRPA1 reduces airway inflammation and airway hyper-responsiveness in a murine model of allergic rhinitis
Source: International Congress 2016 – Novel mechanisms and therapeutic targets in acute and chronic inflammatory disease
Year: 2016
Bcl-2 antagonist ABT-737 attenuates corticosteroid-insensitive neutrophilic airway inflammation
Source: International Congress 2015 – Therapeutic approaches in inflammatory lung disease
Year: 2015
PI3Kδ inhibition confer efficacy in experimental models of allergic airway inflammation
Source: International Congress 2015 – Immune mechanisms in the human lung
Year: 2015
Effect of retinoid X receptor partial agonist on airway inflammation and hyperresponsiveness
Source: International Congress 2015 – Immune aspects of asthma and other airway diseases
Year: 2015
Effects of LTRA on T cell-induced bronchoconstriction in the mice
Source: International Congress 2015 – New models for treating airway diseases
Year: 2015
Transient receptor potential channels in a mouse model of isocyanate-induced airway hyperresponsiveness
Source: International Congress 2014 – Mechanistic murine studies of asthma and lung inflammation
Year: 2014
Acid sphingomyelinase deficiency improves lung function in allergen-induced asthma in mice
Source: Annual Congress 2013 –Cell biology and murine models of asthma and inflammation
Year: 2013
We use cookies on our website to give you the most relevant experience by remembering your preferences and repeat visits. By clicking "Accept", you consent to the use of the cookies.
Accept